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Gamma-aminobutyric acid (GABA) is the main inhibitory neurotransmitter in the mammalian central nervous system. Additionally we are shipping GABBR1 Proteins (10) and GABBR1 Kits (4) and many more products for this protein.
Showing 10 out of 164 products:
Human Monoclonal GABBR1 Primary Antibody for IF, WB - ABIN395531
Oblak, Gibbs, Blatt: Decreased GABA(B) receptors in the cingulate cortex and fusiform gyrus in autism. in Journal of neurochemistry 2010
Show all 5 references for ABIN395531
Human Monoclonal GABBR1 Primary Antibody for ICC, IF - ABIN1027717
Duthey, Caudron, Perroy, Bettler, Fagni, Pin, Prézeau: A single subunit (GB2) is required for G-protein activation by the heterodimeric GABA(B) receptor. in The Journal of biological chemistry 2002
Show all 2 references for ABIN1027717
Human Monoclonal GABBR1 Primary Antibody for ICC, IF - ABIN2484100
Kaupmann, Huggel, Heid, Flor, Bischoff, Mickel, McMaster, Angst, Bittiger, Froestl, Bettler: Expression cloning of GABA(B) receptors uncovers similarity to metabotropic glutamate receptors. in Nature 1997
Human Polyclonal GABBR1 Primary Antibody for IF, ELISA - ABIN1535688
White, Wise, Main, Green, Fraser, Disney, Barnes, Emson, Foord, Marshall: Heterodimerization is required for the formation of a functional GABA(B) receptor. in Nature 1999
Results show that GABBR1 minor genotypes/alleles were protective against risk for alcoholism in 3 ethnically diverse cohorts.
Cell surface ubiquitination precedes endocytosis, after which USP14 acts as an ubiquitin-binding protein that targets the ubiquitinated GABA B receptor to lysosomal degradation and promotes its deubiquitination.
GABA(B)R stimulation promotes chemotaxis in RBL cells which is dependent on signaling via PI3-K/Akt, Src kinases and on rearrangement of both microtubules and actin cytoskeleton.
GABBR1 receptors are expressed in aortic smooth muscle cells and regulate the [Ca(2 (show CA2 Antibodies)+)]i via a Gi/o-coupled receptor pathway and a phospholipase C (show PLC Antibodies) activation pathway.
GABBR1 protein involved in phenylthiourea bitter taste detection.
Authors suggest GABBR1, GABA receptor B1, is implicated in schizophrenia based on a Human endogenous retrovirus long terminal repeat (HERV-W LTR) in the regulatory region of GABBR1.
GABA may inhibit the growth of cholangiocarcinoma QBC939 cells through the GABAB receptor, and the anti-cancer effects may be partly mediated via the JAK (show JAK3 Antibodies)/STAT3 (show STAT3 Antibodies) pathway
Activation of GABA(B) receptors significantly inhibits Akt/GSK-3 signaling in a beta-arrestin-dependent pathway.
We have discovered main triplets of amino acid residues in the GABAB1 receptor and several other neural receptors which seem to come from immunoglobulin chains and appear also as homologies in receptor heteromers.
Long interval intracortical inhibition is significantly reduced and the cortical silent period is significantly shortened in patients with succinic semialdehyde dehydrogenase (SSADH (show ALDH5A1 Antibodies)) deficiency compared to heterozygous parents and control groups.
These results suggest a role for GABAB receptor in Fmrp (show FMR1 Antibodies) regulation and a potential interest of GABAB receptor signaling in Fragile X (show FMR1 Antibodies) syndrome improvement.
Activation of GABA(B) or mGlu2 (show GRM2 Antibodies)/3 receptors inhibited both evoked presynaptic Ca(2 (show CA2 Antibodies)+) transients and striatal field potentials.
Immunoblotting and RT-PCR results showed that NIHL increased the expression of PKCgamma (show PRKCG Antibodies) but decreased that of GABABR1 and GABABR2 (show GABBR2 Antibodies) at both protein and mRNA levels in the CNC
GABAB(1) receptor subunit isoforms differentially regulate the deleterious effects of stress and, thus, may be important therapeutic targets for the treatment of depression
Results establish an endogenous GABA/GABAB1 feedback mechanism that keeps TRPV1 (show TRPV1 Antibodies)-mediated pain hypersensitivity in check.
The Gabbr1 has an important role in mediating the anxiety-related effects induced by nicotine.
Sex differences in reproductive genes Gnrh1 (show GNRH1 Antibodies), Gad1 (show GAD1 Antibodies), and Kiss1 (show KISS1 Antibodies) are critically disturbed in GABAB1-deficient mice, probably altering the organization and development of neural circuits governing the reproductive axis.
GABAB signaling may normally directly or indirectly inhibit Kiss1 (show KISS1 Antibodies) expression, particularly in the bed nucleus of the stria terminalis and medial amygdala
Pharmacological inhibition of GABAB receptors stimulated neural stem cell proliferation and genetic deletion of GABAB1 receptor subunits increased neural stem cell proliferation and differentiation of neuroblasts in vivo.
Enhanced GABA B receptor activity reduces expression of Arg3.1 (show Arc Antibodies), which is critical for long-term memory.
Gamma-aminobutyric acid (GABA) is the main inhibitory neurotransmitter in the mammalian central nervous system. GABA exerts its effects through ionotropic
gamma-aminobutyric acid (GABA) B receptor, 1
, gamma-aminobutyric acid type B receptor subunit 1
, GABA-B receptor 1
, GABAB, subunit 1c
, seven transmembrane helix receptor
, GABA-BR1b receptor
, bM573K1.1.1 (gamma-aminobutyric acid (GABA) B receptor, 1a)
, gamma-aminobutyric acid (GABA-B) receptor, 1
, gamma-aminobutyric acid (GABA) B receptor 1
, gamma-aminobutyric acid B receptor 1