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anti-Mouse (Murine) IL1R1 Antibodies:
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Results demonstrated the essential role of IL-1R1 in kindling-induced sleep disturbance by using transgenic IL-1R1 KO mice. Epilepsy-induced sleep disturbances were absent in the IL-1R1 KO mice, indicating the importance of IL-1 (show IL1A Antibodies) signals. The knockout of IL-1R1 did not change the seizure thresholds, suggesting that IL-1R1 signaling is not in involved in the kindling-induced epileptogenesis.
findings show elements of the IL-1 (show IL1A Antibodies) network, including IL-1alpha, IL-1beta (show IL1B Antibodies), and IL-1R1, are essential for the optimal host response to VACV cutaneous infection
These data show that the normal distribution of surface glycosylation requires IL-1R, but not MyD88 (show MYD88 Antibodies), and is not sufficient to prevent bacterial binding.
IL-1R1 signaling via MyD88 (show MYD88 Antibodies) is critical for the first step of inflammatory response to papain.
The work identifies a pathogenic role of postnatal IL1b (show IL1B Antibodies)/IL-1R1 pathway and subsequent prolonged prominent increase of endocannabinoid signaling in adult seizure susceptibility following prolonged febrile seizures.
SER140 treatment postpones the onset of diabetes in female NOD mice by interfering with IL-1beta (show IL1B Antibodies)/Il1 (show IL1A Antibodies) receptor signaling.
IL-1 (show IL1A Antibodies) receptor (IL-1R1) deficiency or blockade limits blood pressure elevation in this model by mitigating sodium reabsorption via the NKCC2 (show SLC12A1 Antibodies) co-transporter in the nephron.
data identify a previously unappreciated Trim24 (show TRIM24 Antibodies)-dependent requirement for IL-1R expression on TH2 cells and an important nonredundant role for T-cell-intrinsic Trim24 (show TRIM24 Antibodies) in TH2-mediated allergy and antihelminth immunity
The results present identification of critical regions within the TIR domain of IL-1 (show IL1A Antibodies) receptor type in humans and mice.
IL-1R1/MyD88 (show MYD88 Antibodies) signaling negatively regulates bone regeneration via impairment of mesenchymal stem cell proliferation, migration and differentiation by inhibiting the Akt (show AKT1 Antibodies)/GSK-3beta (show GSK3b Antibodies)/beta-catenin (show CTNNB1 Antibodies) pathway.
Since mumps virus SH coimmunoprecipitated with tumor necrosis factor receptor 1 (TNFR1 (show TNFRSF1A Antibodies)), RIP1 (show UQCRFS1 Antibodies), and IRAK1 (show IRAK1 Antibodies), we hypothesize that SH exerts its NF-kappaB (show NFKB1 Antibodies) activation inhibitory function by interacting with TNFR1 (show TNFRSF1A Antibodies), interleukin-1 receptor type 1 (IL-1R1), and TLR3 (show TLR3 Antibodies) complexes in the plasma membrane of infected cells.
ligand-activation of GPER (show GPER Antibodies) generates a feedforward loop coupling IL1beta (show IL1B Antibodies) induction by CAFs (show TBX1 Antibodies) to IL1R1 (show IL1RN Antibodies) expression by cancer cells, promoting the up-regulation of IL1beta (show IL1B Antibodies)/IL1R1 (show IL1RN Antibodies) target genes such as PTGES (show PTGES Antibodies), COX2 (show COX2 Antibodies), RAGE (show AGER Antibodies) and ABCG2 (show ABCG2 Antibodies)
this study shows that IL-1a (show IL1A Antibodies) gene variants are not associated with susceptibility to juvenile idiopathic arthritis in Iranian population
The work identifies a pathogenic role of postnatal IL1b (show IL1B Antibodies)/IL-1R1 pathway and subsequent prolonged prominent increase of endocannabinoid signaling in adult seizure susceptibility following prolonged febrile seizures, and highlights IL-1R1 as a potential therapeutic target for preventing the development of epilepsy after infantile febrile seizures.
IL1R1 (show IL1RN Antibodies) SNP rs949963 is associated with the susceptibility to asthma in children from Central China and may increase the serum expression of IL1R1 (show IL1RN Antibodies).
Altered expression of EP2 (show SPAG11B Antibodies) in patients with aspirin-exacerbated respiratory disease contributes to deficient induction of IL-1RI, reducing the capacity of IL-1beta (show IL1B Antibodies) to increase COX-2 (show COX2 Antibodies) and mPGES-1 (show PTGES Antibodies) expression, which results in low PGE2 production
investigated the association between four inflammatory cytokines (CD121a, interleukin [IL]-1beta, IL-8 (show IL8 Antibodies), and IL-11 (show IL11 Antibodies)) and coronary heart disease
This study found that the mRNA expression of IL-1R1, TNFR1 (show TNFRSF1A Antibodies), and TNFR2 (show TNFRSF1B Antibodies) was significantly higher in schizophreina
IL-1R1 expression appears to define a tissue regulatory T cell phenotype together with the expression of CD25 (show IL2RA Antibodies), glucocorticoid-induced tumor necrosis factor (show TNF Antibodies) receptor family-related gene and CTLA-4 (show CTLA4 Antibodies).
The protein encoded by this gene is a cytokine receptor that belongs to the interleukin 1 receptor family. This protein is a receptor for interleukin alpha (IL1A), interleukin beta (IL1B), and interleukin 1 receptor, type I(IL1R1/IL1RA). It is an important mediator involved in many cytokine induced immune and inflammatory responses. This gene along with interleukin 1 receptor, type II (IL1R2), interleukin 1 receptor-like 2 (IL1RL2), and interleukin 1 receptor-like 1 (IL1RL1) form a cytokine receptor gene cluster in a region mapped to chromosome 2q12.
CD121 antigen-like family member A
, IL-1 receptor alpha chain
, interleukin-1 receptor alpha
, interleukin-1 receptor type 1
, interleukin-1 receptor type I
, antigen CD121a
, interleukin 1 receptor alpha, type I
, interleukin receptor 1
, interleukin 1 beta receptor type 1 short transmembrane form
, interleukin 1 beta receptor type 1 soluble form
, interleukin 1 bete receptor type 1 long transmembrane form