Dimethylarginine Dimethylaminohydrolase 1 (DDAH1) ELISA Kits

DDAH1 belongs to the dimethylarginine dimethylaminohydrolase (DDAH) gene family. Additionally we are shipping DDAH1 Antibodies (87) and DDAH1 Proteins (13) and many more products for this protein.

list all ELISA KIts Gene Name GeneID UniProt
DDAH1 23576 O94760
DDAH1 64157 O08557
DDAH1 69219 Q9CWS0
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Top DDAH1 ELISA Kits at antibodies-online.com

Showing 5 out of 17 products:

Catalog No. Reactivity Sensitivity Range Images Quantity Supplier Delivery Price Details
Human 0.056 ng/mL 0.15 ng/mL - 10 ng/mL 96 Tests Log in to see 13 to 16 Days
$736.84
Details
Rat 9.75 pg/mL 39-2500 pg/mL Typical standard curve 96 Tests Log in to see 15 to 18 Days
$910.56
Details
Human < 0.065 ng/mL 0.156 ng/mL - 10 ng/mL   96 Tests Log in to see 11 to 18 Days
$810.17
Details
Mouse < 0.086 ng/mL 0.156 ng/mL - 10 ng/mL   96 Tests Log in to see 11 to 18 Days
$589.86
Details
Cow
  96 Tests Log in to see 15 to 18 Days
$1,029.60
Details

Top referenced DDAH1 ELISA Kits

  1. Rat (Rattus) DDAH1 ELISA Kit for Sandwich ELISA - ABIN858752 : Ferrigno, Rizzo, Bianchi, Di Pasqua, Berardo, Richelmi, Vairetti: Changes in ADMA/DDAH pathway after hepatic ischemia/reperfusion injury in rats: the role of bile. in BioMed research international 2014 (PubMed)

  2. Human DDAH1 ELISA Kit for Sandwich ELISA - ABIN419628 : El Assar, Angulo, Santos-Ruiz, Ruiz de Adana, Pindado, Sánchez-Ferrer, Hernández, Rodríguez-Mañas: Asymmetric dimethylarginine (ADMA) elevation and arginase up-regulation contribute to endothelial dysfunction related to insulin resistance in rats and morbidly obese humans. in The Journal of physiology 2016 (PubMed)

More ELISA Kits for DDAH1 Interaction Partners

Human Dimethylarginine Dimethylaminohydrolase 1 (DDAH1) interaction partners

  1. MiR (show MLXIP ELISA Kits)-21-mediated DDAH1/ADMA/NO signal pathway.

  2. DDAH-1 expression is associated with promotion of angiogenesis stimulating factor VEGF (show VEGFA ELISA Kits).

  3. These findings suggest that DDAH1 functions as a tumor suppressor in gastric cancer (GC) and may be exploited as a diagnostic and prognostic biomarker for GC.

  4. Data demonstrate that DDAH1 deficiency promotes the epithelial to mesenchymal transition in renal proximal tubular epithelial cells and causes fibrosis, and oxidative stress in aging and diabetic kidneys. The study provides the first direct evidence that the DDAH1 has a marked effect on kidney fibrosis and oxidative stress induced (show SQSTM1 ELISA Kits) by aging or diabetes.

  5. Results confirmed DDAH1 3'-UTR (show UTS2R ELISA Kits) as a target for miR (show MLXIP ELISA Kits)-21, and endogenous miR (show MLXIP ELISA Kits)-21 showed increased inhibitory effect on DDAH1-V3 transcript.

  6. Inflammatory factors expressed in response to myocardial ischemia contributed to up-regulation of DDAH1.

  7. DDAH1 plays dual roles in a particular matter-induced cell death in alveolar epithelial cells.

  8. rs3087894 in DDAH1 was significantly associated with hypertension and showed conflicting results in different ethnic groups. This is therefore a candidate for further studies with the aim of helping to ascertain the mechanisms of hypertension in different populations.

  9. results suggest that miR (show MLXIP ELISA Kits)-21 may regulate renal fibrosis by the Wnt (show WNT2 ELISA Kits) pathway via directly targeting DDAH1

  10. The most significant associations were detected for PECAM1 (show PECAM1 ELISA Kits)*V/V + DDAH1*C (OR = 4.17 CI 1.56-11.15 Pperm = 0.005)

Mouse (Murine) Dimethylarginine Dimethylaminohydrolase 1 (DDAH1) interaction partners

  1. Cardiomyocyte DDAH1 activity is dispensable for cardiac function under basal conditions, but plays an important role in attenuating cardiac hypertrophy and ventricular remodeling under stress conditions, possibly through locally confined regulation of subcellular ADMA and NO signaling.

  2. the ADMA/DDAH1 pathway has a marked effect on hepatic lipogenesis and steatosis induced by HFD feeding. Our findings suggest that strategies to increase DDAH1 activity in hepatocytes may provide a novel approach to attenuate NAFLD (show TSC2 ELISA Kits) development.

  3. A time-dependent decrease in serum and tissue ADMA and increase in mRNA expression of DDAH-1 and PRMT-1 as well as higher rates of mRNA expression of CAT-1 and lower rates of CAT-2A and CAT-2B were found after 8-week MCD diet.

  4. our results suggest that DDAH1 not only acts as an enzyme degrading ADMA but also controls cellular oxidative stress and apoptosis via a miR (show MLXIP ELISA Kits)-21-dependent pathway.

  5. In mild CKD, dysregulation of the ADMA/DDAH (show DDAH2 ELISA Kits) pathway in adipose tissue triggers lipodystrophy-like phenotype changes, including ectopic fat depositions.

  6. Endothelial deletion of DDAH1 profoundly impairs the angiogenic capacity of endothelial cells.

  7. Our findings suggest that decreased expression of DDAH1 and DDAH2 (show DDAH2 ELISA Kits) in the lungs may contribute to allergic asthma and overexpression of DDAH1 attenuates allergen-induced airway inflammation through modulation of Th2 responses.

  8. Overexpression of the ADMA degrading enzyme, DDAH1, did not ameliorate atherosclerosis in ApoE (show APOE ELISA Kits)-deficient subtotally nephrectomized mice.

  9. DDAH1 deficiency attenuates endothelial cell cycle progression and angiogenesis.

  10. DDAH1 overexpression selectively decreased the sustained phase of hypoxic pulmonary vasoconstriction, partly via activation of the NO-cGMP pathway.

Cow (Bovine) Dimethylarginine Dimethylaminohydrolase 1 (DDAH1) interaction partners

  1. dimethylargininase-1 inhibited upon specific Cys (show DNAJC5 ELISA Kits)-S-nitrosylation.

  2. High-resolution crystal structures of DDAH (show DDAH2 ELISA Kits) isoform 1 was presented.

  3. Demonstrate a critical role for DDAH-1 and endogenous methylarginines in the pathogenesis of endothelial dysfunction.

  4. DDAH-1 and DDAH-2 (show DDAH2 ELISA Kits) manifest their effects through different mechanisms, the former of which is largely ADMA-dependent and the latter ADMA-independent.

DDAH1 Antigen Profile

Antigen Summary

This gene belongs to the dimethylarginine dimethylaminohydrolase (DDAH) gene family. The encoded enzyme plays a role in nitric oxide generation by regulating cellular concentrations of methylarginines, which in turn inhibit nitric oxide synthase activity.

Gene names and symbols associated with DDAH1

  • dimethylarginine dimethylaminohydrolase 1 (DDAH1) antibody
  • dimethylarginine dimethylaminohydrolase 1 (Ddah1) antibody
  • dimethylarginine dimethylaminohydrolase 1 L homeolog (ddah1.L) antibody
  • dimethylarginine dimethylaminohydrolase 1 (ddah1) antibody
  • 2410006N07Rik antibody
  • 2510015N06Rik antibody
  • AI987801 antibody
  • AW050362 antibody
  • DDAH antibody
  • DDAH1 antibody
  • wu:fc30c11 antibody
  • zgc:85829 antibody

Protein level used designations for DDAH1

DDAH-1 , DDAHI , N(G),N(G)-dimethylarginine dimethylaminohydrolase 1 , NG, NG-dimethylarginine dimethylaminohydrolase , dimethylargininase-1 , NG,NG dimethylarginine dimethylaminohydrolase , dimethylarginine dimethylaminohydrolase 1 , fc30c11

GENE ID SPECIES
23576 Homo sapiens
64157 Rattus norvegicus
69219 Mus musculus
378898 Gallus gallus
380577 Xenopus laevis
537391 Bos taurus
711395 Macaca mulatta
736613 Pan troglodytes
100145661 Xenopus (Silurana) tropicalis
100339913 Oryctolagus cuniculus
490182 Canis lupus familiaris
100153814 Sus scrofa
406561 Danio rerio
100729503 Cavia porcellus
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