Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
ENG encodes a homodimeric transmembrane protein which is a major glycoprotein of the vascular endothelium. Additionally we are shipping Endoglin Antibodies (765) and Endoglin Proteins (53) and many more products for this protein.
Showing 7 out of 74 products:
Human ENG ELISA Kit for Sandwich ELISA - ABIN414754
Silva, Teixeira, Oliveira, Almeida, Almeida, Águas, Vasconcelos: Endothelial Dysfunction and Nailfold Videocapillaroscopy Pattern as Predictors of Digital Ulcers in Systemic Sclerosis: a Cohort Study and Review of the Literature. in Clinical reviews in allergy & immunology 2015
Show all 4 Pubmed References
Human ENG ELISA Kit for Sandwich ELISA - ABIN624967
Levy, Finestone: Renal response to four hours of biliary obstruction in the dog. in The American journal of physiology 1983
Show all 2 Pubmed References
Human ENG ELISA Kit for Sandwich ELISA - ABIN2684119
Grisanti, Canbek, Kaiserling, Adam, Lafaut, Gelisken, Szurman, Henke-Fahle, Oficjalska-Mlynczak, Bartz-Schmidt: Expression of endoglin in choroidal neovascularization. in Experimental eye research 2004
Show all 3 Pubmed References
ENG mutation carriers were more likely than ACVRL1 (show ACVRL1 ELISA Kits) mutation carriers to have pAVMs (P < 0.001) or multiple lesions (P = 0.03), and to undergo procedural intervention (P = 0.02). The HHT (show ACVRL1 ELISA Kits) severity score was significantly higher in ENG than in ACVRL1 (show ACVRL1 ELISA Kits) (P = 0.02).
levels of the angiogenesis mediators endoglin, HB-EGF (show HBEGF ELISA Kits), BMP-9 (show GDF2 ELISA Kits) and FGF-2 (show FGF2 ELISA Kits) in patients with severe sepsis and septic shock; endoglin and HB-EGF (show HBEGF ELISA Kits) could be involved in the host response of sepsis; additional studies are warrant to investigate their role as biomarker or therapeutic targets in sepsis
A heterozygous genetic variant, c.704dupC; p.Val236Glyfs*98, was identified in the ENG gene. The variant c.704dupC was not previously described in the HHT (show ACVRL1 ELISA Kits) Mutations Database
Endoglin levels are changed after stroke.
we found significant correlation with CD105 and survival rate of the patient. Similar correlation was found between histological grades and TNM staging in CD105. High expression was associated with low survival; whereas no significant correlation was found with expression of VEGF with survival, also with TNM staging and histological grading.
CD105 expression was associated with more aggressive tumor behavior, more advanced disease, and worse prognosis in clear cell renal cell carcinoma
Data indicate that in pancreatic cancer cells, the expression of ENG may be controlled by a pathway mediated by SMAD4 (show SMAD4 ELISA Kits). In addition, ENG was found to be related to the spheroid-forming ability of cells and to be involved in the invasive capacity of pancreatic cancer cells.
The increase in TGF-beta3 (show TGFB3 ELISA Kits) found in inflammatory wound healing (WF) highlights its negative effect on wound healing, while the increased levels of sEng in granulating WF affects the leukocyte adhesion/transmigration through the endothelium, reducing the inflammatory response and favoring the wound healing.
Soluble endoglin level could serve as a determinant of walking abilities change after supervised treadmill training program in patients with peripheral artery disease.
In primary hip OA, angiogenesis may be induced by a combined mechanism: hypoxia-related VEGF (show VEGFA ELISA Kits)-dependent vasculogenesis and endothelial differentiation of the activated pluripotent cells, which are released from the hyperplastic synovial cells layer. An endothelial mesenchymal transition is assumed to be involved in the fibrotic process
Transplantation of mesenchymal stem cells, when combined with endothelial progenitor cells, accelerate muscle recovery in a mouse model of hind limb ischemia, through an endoglin-dependent mechanism.
we cannot rule out a possibility that a long-term chronic exposure (9 months and more) to soluble endoglin alone or combined with other cardiovascular risk factors may contribute to alterations of heart function and structure in Sol (show KALRN ELISA Kits)-Eng+ mice, which is the topic in our lab in ongoing experiments
Data show that concomitant activin receptor-like kinase 1 (Acvrl1 (show ACVRL1 ELISA Kits)) and endoglin (Eng) deficiency synergistically decreases pancreatic neuroendocrine tumor volume.
Endoglin is necessary for the pro-angiogenic properties of the cardiosphere-derived cells secretome.
systemic DPP4 (show DPP4 ELISA Kits) inhibition restores the impaired mononuclear cell homing in Eng+/- animals post-myocardial infarction, and enhances cardiac repair, which might be explained by restoring the balance between the inflammatory and regenerative macrophages present in the heart
results suggest that soluble endoglin modulates inflammatory-related diseases and open new perspectives leading to the development of novel and targeted approaches for the prevention and treatment of cardiovascular diseases
Endoglin controls blood vessel diameter through endothelial cell shape changes in response to haemodynamic cues.
Endoglin prevents vascular malformation by regulating flow-induced cell migration and specification through VEGFR2 (show KDR ELISA Kits) signalling.
Identification of novel regulators of developmental hematopoiesis using Endoglin regulatory elements as molecular probes
Endoglin has an important role in VSMC recruitment and blood vessel maturation during angiogenesis.
This gene encodes a homodimeric transmembrane protein which is a major glycoprotein of the vascular endothelium. This protein is a component of the transforming growth factor beta receptor complex and it binds TGFB1 and TGFB3 with high affinity. Mutations in this gene cause hereditary hemorrhagic telangiectasia, also known as Osler-Rendu-Weber syndrome 1, an autosomal dominant multisystemic vascular dysplasia. Alternatively spliced transcript variants encoding different isoforms have been found for this gene.
endoglin (Osler-Rendu-Weber syndrome 1)
, CD105 antigen
, cell surface MJ7/18 antigen
, transmembrane glycoprotein