More product categories related to IL25 ELISA Kit
Top referenced IL25 ELISA Kits
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Human IL25 ELISA Kit for Sandwich ELISA - ABIN414794 : Javan, Seyfizadeh, Aslani, Farhoodi, Babaloo: Molecular analysis of interleukin-25 exons 1 and 2 and its serum levels in Iranian patients with multiple sclerosis. in American journal of clinical and experimental immunology 2014 (PubMed)
Show all 4 Pubmed References -
Human IL25 ELISA Kit for Sandwich ELISA - ABIN365233 : Hams, Armstrong, Barlow, Saunders, Schwartz, Cooke, Fahy, Crotty, Hirani, Flynn, Voehringer, McKenzie, Donnelly, Fallon: IL-25 and type 2 innate lymphoid cells induce pulmonary fibrosis. in Proceedings of the National Academy of Sciences of the United States of America 2014 (PubMed)
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Human IL25 ELISA Kit for Sandwich ELISA - ABIN2859300 : Meng, Shen, Shen, Zhou, Shen, Das: Effect of n-3 and n-6 unsaturated fatty acids on prostate cancer (PC-3) and prostate epithelial (RWPE-1) cells in vitro. in Lipids in health and disease 2014 (PubMed)
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Human IL25 ELISA Kit for Sandwich ELISA - ABIN2703183 : Wang, Fan, Lv, Yao, Wang, Huang, Meng, Fang, Lee, Corrigan, An, Ying: Interleukin-25 promotes basic fibroblast growth factor expression by human endothelial cells through interaction with IL-17RB, but not IL-17RA. in Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology 2013 (PubMed)
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Mouse (Murine) IL25 ELISA Kit for Sandwich ELISA - ABIN425028 : Cai, Zhang, Zhang, Chen, Hu: Effect of Th9/IL-9 on the growth of gastric cancer in nude mice. in OncoTargets and therapy 2019 (PubMed)
More ELISA Kits for IL25 Interaction Partners
Human Interleukin 25 (IL25) interaction partners
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Nasal IL-25 predicts the response to oral corticosteroids.
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IL-25 (also known as IL-17E) is a member of the IL-17 cytokine family and plays a variety of roles in different inflammatory murine models, such as asthma, atopic dermatitis, and pulmonary fibrosis.
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expression of IL-17A, E, and F and their receptors in non-small-cell lung cancer; data suggest that IL-17A, E, F and their receptors IL-17RA, RB, RC may be involved in the pathogenesis of non-small cell lung cancer
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MiR-31a-5p suppression decreased IL-25 expression, inhibited IL-1beta, IL-6, IL-8, and TNF-alpha secretions, and decreased proliferation in CC-2511 cells under hyperthermic stress.
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study provides a comprehensive characterization of the thymic stroma and identifies a new tuft-like thymic epithelial cell population, which is critical for shaping the immune niche in the thymus
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The data demonstrate that an IL-17-induced autoregulatory circuit in keratinocytes is mediated by IL-25 and suggest that this circuit could be targeted in the treatment of psoriasis patients.
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this review details the structural characteristics, expression patterns, responder cells, biological properties, and role of IL-25 in disease pathogenesis
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this study shows that IL-25 reduces Th17 cells and inflammatory responses in human peripheral blood mononuclear cells
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Endometrial stromal cells differentiate into decidual stromal cells in the presence of ovarian hormones, resulting in the upregulation of IL25/IL17RB expression in endometrial stromal cells.
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In conclusion, the findings of the present study suggest that IL-25 but not IL-33 might contribute to the pathogenesis of chronic eosinophilic inflammation of the lung in patients with CEP.
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Nasal Il-25 is overexpressed in patients with chronic rhinosinusitis with nasal polyps and airway hypersensitiveness.
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IL-4 signaling up-regulates the IL-25 axis in human monocytic cells, and IL-25 may provide autocrine signals in monocytes and macrophages to sustain IL-17Rb expression and predispose to alternative activation.
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IL-25-induced activation of nasal fibroblast and its association with the remodeling of chronic rhinosinusitis with nasal polyposis
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The authors concluded that IL-25 provides protection from amebiasis, which is dependent upon intestinal eosinophils and suppression of TNF-alpha.
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Inhibition of IL-25 resulted in decreased type 2 T cells and macrophages in the primary tumor microenvironments, both reported to enhance breast tumor invasion and subsequent metastasis to the lung.
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Our findings suggest that the IL-25/IL-25R axis may play an important role in promoting the recruitment and proinflammatory function of eosinophils in allergic asthma.
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Chronic rhinosinusitis with nasal polyps (CRSwNP) epithelial cells release TSLP and IL-25 when stimulated by poly(I:C) but not by DP or AF, suggesting that viral infection may contribute to maintain and amplify the T2 immune response seen in CRSwNP.
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Increased induction and expression of TSLP, IL-25, and IL-33 from nasal epithelial cells contribute to the pathophysiology of eosinophilic chronic rhinosinusitis.
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Women with endometriosis showed significantly higher levels of IL-25 in their PF (p=0.019) compared to controls. IL-25 levels did not correlate with the stage of endometriosis.
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IL-8 and IL-25 are targets of miR-20b.
Mouse (Murine) Interleukin 25 (IL25) interaction partners
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Our results identify a novel IL-25 inflammatory pathway involved in induction of TH17 cell-mediated, but not TH2 cell-mediated, contact hypersensitivity (CHS). IL-25 neutralization can be a potential approach for treatment of CHS.
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Prevention of food allergy development and suppression of established food allergy by neutralization of thymic stromal lymphopoietin, IL-25, and IL-33.
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endogenous IL-25 has an atheroprotective role in the murine aortic arch has potential implications for atherosclerosis development and management in humans
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study provides a comprehensive characterization of the thymic stroma and identifies a new tuft-like thymic epithelial cell population, which is critical for shaping the immune niche in the thymus
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these data suggest that IL-25 plays an inhibitory role in the growth and development of colonic tumors.
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this study shows that the signaling axis of microRNA-31/interleukin-25 regulates Th1/Th17-mediated inflammation response in colitis
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IL-25 is a component of the immune response that is regulated by a healthy microbiota and reduces pathology associated with Clostridium difficile Infection.
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study concludes that IL-33 and TSLP are required for epithelial cell IL-25 expression, mucous metaplasia, and ILC2 expansion following early-life rhinovirus infection
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IL-17E (IL-25) and IL-17RB promote respiratory syncytial virus-induced pulmonary disease
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data suggested that deletion of Shp2 impaired IL-25 production in bronchial epithelial cells in vitro, but might yet have minor influence on OVA-induced allergic reaction in vivo
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study showed that IL-25 promoted the accumulation of ICOS and T1/ST2 on nuocytes, further induced the pro-inflammatory Th2 cells, and promoted Th2 cytokine responses in OVA-induced airway inflammation
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Inhibition of IL-25 resulted in decreased type 2 T cells and macrophages in the primary tumor microenvironments, both reported to enhance breast tumor invasion and subsequent metastasis to the lung.
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this study shows that activation of primed TH2 cells occurs independently of group 2 innate lymphoid cells or their cytokines but that the effector function of both cell types was dependent on combinatorial exposure to IL-33, IL-25 and TSLP
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These data demonstrate that IL-25 is critical for host protective immunity against H. polygyrus bakeri infection, highlighting its potential application as a therapeutic agent against parasitic nematode infection worldwide.
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Findings indicate that the release of interleukin 25 (IL-25) from tumour-associated fibroblasts (TAFs) may serve as a check point for control of mammary tumour metastasis.
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IL-25 and CD4(+) TH2 cells enhance type 2 innate lymphoid cell-derived IL-13 production, which promotes IgE-mediated experimental food allergy.
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IL-22 restrains tapeworm-mediated protection against colitis via regulation of IL-25 expression
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IL-25 promotes the function of CD4+CD25+ T regulatory cells and prolongs skin-graft survival in murine models
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IL-25 and IL-33 are critical for induction of Th2-type cytokine-mediated allergic airway eosinophilia, but not Th17-type cytokine-mediated airway neutrophilia, at the local sites of lungs in the challenge phase of mice sensitized EC with OVA
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Suggest that chronic exposure of murine airways to IL-25 alone is able to reproduce a local angiogenic milieu in asthmatic mice.
IL25 Antigen Profile
Antigen Summary
The protein encoded by this gene is a cytokine that shares sequence similarity with interleukin 17. This cytokine can induce NF-kappaB activation, and stimulate the production of interleukin 8. Both this cytokine and interleukin 17B are ligands for the cytokine receptor IL17BR. Studies of a similar gene in mice suggest that this cytokine may be a pro-inflammatory cytokine favoring the Th2-type immune response. Alternative splicing results in multiple transcript variants.
Gene names and symbols associated with Interleukin 25 (IL25) ELISA Kits
- interleukin 25 (IL25) antibody
- interleukin 25 (Il25) antibody
- IL-17e antibody
- Il17e antibody
- IL25 antibody
- RGD1561632 antibody
Protein level used designations for Interleukin 25 (IL25) ELISA Kits
interleukin-17E , interleukin-25 , interleukin 17E , interleukin 25 , interleukin 25-like
GENE ID | SPECIES |
---|---|
64806 | Homo sapiens |
140806 | Mus musculus |
467404 | Pan troglodytes |
480252 | Canis lupus familiaris |
501996 | Rattus norvegicus |
526816 | Bos taurus |
100154041 | Sus scrofa |
100499506 | Ovis aries |
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