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IL32 encodes a member of the cytokine family. Additionally we are shipping IL32 Antibodies (165) and IL32 Proteins (11) and many more products for this protein.
Showing 10 out of 42 products:
Human IL32 ELISA Kit for Sandwich ELISA - ABIN415100
Inoue, Shoda, Seri, Kubo, Kanda, Fujio, Yamamoto: Three cases of lupus nephritis patients with serum interleukin-32? detection. in Lupus 2014
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Human IL32 ELISA Kit for Sandwich ELISA - ABIN366546
Huang, Qi, Ma, He, Ji, Sun, Ruan: The expression of interleukin-32 is activated by human cytomegalovirus infection and down regulated by hcmv-miR-UL112-1. in Virology journal 2013
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Strong expression of IL-32 was detected in cardiomyocytes from heart failure patients.
The increase in serum levels of IL-32 in accordance with additive effect of the presence of C allele in Multiple sclerosis patients might introduce IL-32 as a key player in MS pathogenesis or immunedysregulation
Collectively, these results demonstrated that IL-32alpha upregulates the atheroprotective genes Timp3 (show TIMP3 ELISA Kits) and Reck (show RECK ELISA Kits) by downregulating microRNA-205 through regulation of the Rprd2-Dgcr8/Ddx5 (show DDX5 ELISA Kits)-Dicer1 (show DICER1 ELISA Kits) biogenesis pathway.
high expression of both IL17A (show IL17A ELISA Kits) and IL32 leads to enhancement of T cell responses in breast tumors
High IL32 expression is associated with lung metastasis in melanoma.
In Kaposi sarcoma (KS) splicing ratio of the IL-32 isoforms showed IL-32gamma as the highest expressed isoform, followed by IL-32beta, in HIV-related KS cases compared with controls. Our data suggest a possible survival mechanism by the splicing of IL-32gamma to IL-32beta and also IL-6 (show IL6 ELISA Kits), IL-8 (show IL8 ELISA Kits), and CXCR1 (show CXCR1 ELISA Kits) signaling pathways to reverse the proapoptotic effect of the IL-32gamma isoform, leading to tumor cell survival and th...
Three single nucleotide polymorphisms (SNPs) (rs28372698, rs12934561, rs4786370) of the IL32 gene have been proposed as modifiers for different diseases. The present study found no significant differences in the genotypic frequencies between the patients and healthy controls and no relation to survival for any of the SNPs.
This study showed that the induction level of IL-32 was increased in chronic rhinosinusitis with nasal polyps compared to normal nasal mucosa and that LPS-induced IL-32 expression in nasal polyp-derived fibroblasts was regulated via the TLR4 (show TLR4 ELISA Kits)/JNK (show MAPK8 ELISA Kits)/AKT (show AKT1 ELISA Kits)/CREB (show CREB1 ELISA Kits) signaling pathway.
findings show that IL-32alpha acts on NK cells to inhibit IL-15 (show IL15 ELISA Kits)-mediated STAT5 (show STAT5A ELISA Kits) phosphorylation and to suppress their IL-15 (show IL15 ELISA Kits)-induced effector molecule expression and cytolytic capacity. IL-32alpha also acted on DCs by downregulating IL-15 (show IL15 ELISA Kits)-induced IL-18 (show IL18 ELISA Kits) production, an important cytokine in NK cell activity.
Thus, endogenous IL-32 is a crucial cytokine involved in the host defense against Leishmania parasites.
This gene encodes a member of the cytokine family. The protein contains a tyrosine sulfation site, 3 potential N-myristoylation sites, multiple putative phosphorylation sites, and an RGD cell-attachment sequence. Expression of this protein is increased after the activation of T-cells by mitogens or the activation of NK cells by IL-2. This protein induces the production of TNFalpha from macrophage cells. Alternate transcriptional splice variants, encoding different isoforms, have been characterized.
, interleukin-32 eta
, interleukin-32 small
, interleukin-32 theta
, natural killer cell transcript 4
, natural killer cells protein 4
, tumor necrosis factor alpha-inducing factor