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MAOA is one of two neighboring gene family members that encode mitochondrial enzymes which catalyze the oxidative deamination of amines, such as dopamine, norepinephrine, and serotonin. Additionally we are shipping Monoamine Oxidase A Kits (48) and Monoamine Oxidase A Proteins (12) and many more products for this protein.
Showing 10 out of 91 products:
Human Polyclonal Monoamine Oxidase A Primary Antibody for WB - ABIN517731
Xiao, Li, Jones-Brando, Yolken: Abnormalities of neurotransmitter and neuropeptide systems in human neuroepithelioma cells infected by three Toxoplasma strains. in Journal of neural transmission (Vienna, Austria : 1996) 2013
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Human Polyclonal Monoamine Oxidase A Primary Antibody for IHC (p), IHC - ABIN446982
Maeda, Morikawa, Takadate, Suzuki, Minowa, Hanagata, Onogawa, Motoi, Nishimura, Unno: Mass spectrometry-based proteomic analysis of formalin-fixed paraffin-embedded extrahepatic cholangiocarcinoma. in Journal of hepato-biliary-pancreatic sciences 2015
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Human Polyclonal Monoamine Oxidase A Primary Antibody for ELISA, WB - ABIN250814
Domschke, Sheehan, Lowe, Kirley, Mullins, Osullivan, Freitag, Becker, Conroy, Fitzgerald, Gill, Hawi et al.: Association analysis of the monoamine oxidase A and B genes with attention deficit hyperactivity disorder (ADHD) in an Irish sample: preferential transmission of the MAO-A 941G allele to affected ... in American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics 2005
Human Polyclonal Monoamine Oxidase A Primary Antibody for WB - ABIN4890309
Matsumoto, Watanabe, Taguchi, Kobayashi: Mechanisms underlying increased serotonin-induced contraction in carotid arteries from chronic type 2 diabetic Goto-Kakizaki rats. in Pharmacological research 2014
The present study extends previous work in the field by demonstrating that MAOA and harsh parenting assessed in early childhood interact to not only predict antisocial behavior in early adulthood, but also predict social information processing, a well-established social-cognitive correlate of antisocial behavior.
Study demonstrated a gene-hormone interaction both on a behavioral and neural level on risk-taking in young men. MAOA-S carriers showed attenuated automatic avoidance tendencies as reflected via response times on cash-outs. While under placebo MAOA-S and MAOA-L carriers did not differ concerning their riskiness during a risk task, testosterone administration promoted risk-taking in MAOA-S carriers.
This study demonstrated that the MAOA-uVNTR genotypes did not show a significant association with ESS scores neither on women nor on men in patient with excessive daytime sleepiness.
Depressive symptoms in schizophrenic patients may be influenced by functional gene polymorphisms, especially those implicated in serotonergic neurotransmission, sert (show SLC6A4 Antibodies), mao-a, and comt (show COMT Antibodies).
This study extends earlier findings demonstrating an interaction between MAOA genotype and prenatal nicotine exposure on aggressive behavior into young adulthood.
Anxiety and Mood were not related to the MAO-A gene polymorphisms in healthy, late reproductive-stage women.
By examining criminal proceedings in which MAOA-L genotype evidence was introduced, we explored the forensic uses of behavioral genetic science. Westlaw and LexisNexis legal databases were electronically searched for cases from 1995 to 2016 to identify court documents from cases involving the MAOA-L genotype. Evidence of the MAOA-L genotype was included in records from 11 criminal cases (9 U.S. and 2 Italian).
MAOA is a risk gene for psychiatric disorders.
High MAOA expression is associated with glioma progression.
The relationship between personality traits of postmenopausal women and the presence of the 44-bp VNTR polymorphism in the serotonin transporter (show SLC6A4 Antibodies) (5-HTT) (SLC6A4 (show SLC6A4 Antibodies)) promoter region and the 30-bp VNTR polymorphism in the MAO-A promoter region was determined.
Study provides experimental evidence showing that sleep deprivation induces an increase in monoamine oxidase A levels in certain brain regions, such as the amygdala and hippocampus of mice that might mediate depressive-like behaviors.
This study unravels a new link between MAO (show MAO Antibodies)-dependent H2O2 production and lysosomal dysfunction. Altogether, our findings demonstrate that the MAO-A/H2O2 axis has a negative impact on the elimination and recycling of mitochondria through the autophagy-lysosome pathway, which participates in cardiomyocyte death and heart failure
Results demonstrate that C17H11IOSe, a selective inhibitor of cortical MAO-A, elicited an antidepressant-like action in mice by interacting with the serotonergic system
data lead us to conclude that elevation of AP-1 (show JUN Antibodies) or NF-kB indirectly decreases MAO-A protein levels which, in turn, diminishes MAO-A ability in the VTA of the mesolimbic dopaminergic pathway
Huntington disease (show HTT Antibodies) neural cells exhibit increased Monoamine oxidase-A and Monoamine oxidases-B expression and activity
acute stress induces anxiety-like responses by affecting rapid dendritic remodeling in the pyramidal cells of orbitofrontal cortex and basolateral amygdala; furthermore, our data show that MAO-A and monoamine metabolism are required for these phenomena.
These findings demonstrate that regulation of monoamine levels by Mao (show MAO Antibodies) activity in beta cells is pivotal for physiological insulin (show INS Antibodies) secretion and that loss of MaoB (show MAOB Antibodies) expression may contribute to the beta cell dysfunction in type 2 diabetes.
Results suggest a role for KLF11 (show KLF11 Antibodies) in upregulating MAO-A in depressive disorder and chronic social stress, suggesting that inhibition of the pathways regulated by this transcription factor may aid in the therapeutics of neuropsychiatric illnesses
The results of this study suggest that heightened dACC and amygdala activation and their connectivity are neuroaffective mechanisms underlying anger control in participants with the low-functioning allele of the MAOA gene.
These results suggest that early developmental enhancements in 5-HT (show DDC Antibodies) levels have long-term effects on the modulation of behavioral flexibility associated with MAO-A deficiency.
Two pools of ten bovine embryos, comprised of the 4-, 8- to 16-cell, morula, blastocyst, and expanded blastocyst stages, were collected. Total RNA was isolated, and the RT-PCR-RFLP technique was used to observe expression of the MAOA gene.
MAOA was seriously demethylated or showed aberrant methylation patterns in four aborted clones.
cloning and characterization of monoamine oxidase A and B genes in pig
Social behavior was facilitated by chronic fluoxetine treatment in juvenile rhesus monkeys interacting with a familiar peer. The type of social behavior affected depended on the MAOA genotype of the monkey and its partner.
Results provide novel evidence that blood MAOA expression predicts alcohol consumption and that heavy alcohol use is linked to low MAOA expression in both the blood and NAc (show NLRP1 Antibodies) core.
study found the ability to consolidate sleep during the dark cycle was disrupted by prenatal iron deprivation, specifically in monkeys with the low-MAOA genotype
The effects of iron deficiency are dependent on MAOA genotype in terms of both direction and size of the effect.
rhMAOA-LPR (show FAS Antibodies) allele modifies the effects of maternal temperament on offspring's personality development.
Zebrafish MAO (zMAO (show MAOB Antibodies)) exhibits functional properties similar to human hMAO A as well as exhibits its own unique behavior.
expression, purification & characterization of zebrafish monoamine oxidase (zMAO (show MAOB Antibodies)) using Pichia pastoris expression system is described; zMAO (show MAOB Antibodies) possesses little differential sensitivity to acetylenic inhibitors than exhibited by either human MAO A & MAO B (show MAOB Antibodies)
this study confirm the presence of functionally active MAO (show MAOB Antibodies) in zebrafish brain and other tissues and characterize the neural systems that express MAO (show MAOB Antibodies) and areas of intense activity in the brain.
This gene is one of two neighboring gene family members that encode mitochondrial enzymes which catalyze the oxidative deamination of amines, such as dopamine, norepinephrine, and serotonin. Mutation of this gene results in Brunner syndrome. This gene has also been associated with a variety of other psychiatric disorders, including antisocial behavior. Alternatively spliced transcript variants encoding multiple isoforms have been observed.
amine oxidase [flavin-containing] A
, monoamine oxidase type A
, monoamine oxidase A
, amine oxidase [flavin-containing] A-like
, amine oxidase
, amine oxidase [flavin-containing]
, putative monoamine oxidase A