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MAOA is one of two neighboring gene family members that encode mitochondrial enzymes which catalyze the oxidative deamination of amines, such as dopamine, norepinephrine, and serotonin. Additionally we are shipping Monoamine Oxidase A Antibodies (91) and Monoamine Oxidase A Proteins (12) and many more products for this protein.
Showing 10 out of 46 products:
Rat (Rattus) Monoamine Oxidase A ELISA Kit for Sandwich ELISA - ABIN432431
Baluchnejadmojarad, Rabiee, Zabihnejad, Roghani: Ellagic acid exerts protective effect in intrastriatal 6-hydroxydopamine rat model of Parkinson's disease: Possible involvement of ERβ/Nrf2/HO-1 signaling. in Brain research 2017
This study demonstrated that the MAOA-uVNTR genotypes did not show a significant association with ESS scores neither on women nor on men in patient with excessive daytime sleepiness.
Depressive symptoms in schizophrenic patients may be influenced by functional gene polymorphisms, especially those implicated in serotonergic neurotransmission, sert (show SLC6A4 ELISA Kits), mao-a, and comt (show COMT ELISA Kits).
This study extends earlier findings demonstrating an interaction between MAOA genotype and prenatal nicotine exposure on aggressive behavior into young adulthood.
Anxiety and Mood were not related to the MAO-A gene polymorphisms in healthy, late reproductive-stage women.
By examining criminal proceedings in which MAOA-L genotype evidence was introduced, we explored the forensic uses of behavioral genetic science. Westlaw and LexisNexis legal databases were electronically searched for cases from 1995 to 2016 to identify court documents from cases involving the MAOA-L genotype. Evidence of the MAOA-L genotype was included in records from 11 criminal cases (9 U.S. and 2 Italian).
MAOA is a risk gene for psychiatric disorders.
High MAOA expression is associated with glioma progression.
The relationship between personality traits of postmenopausal women and the presence of the 44-bp VNTR polymorphism in the serotonin transporter (show SLC6A4 ELISA Kits) (5-HTT) (SLC6A4 (show SLC6A4 ELISA Kits)) promoter region and the 30-bp VNTR polymorphism in the MAO-A promoter region was determined.
Post-stroke fatigue may be associated with a genetic polymorphism involving MAO-A, at least in female stroke patients.
Childhood trauma interacts with haplotypes in COMT (show COMT ELISA Kits), MAOA and MAOB (show MAOB ELISA Kits), increasing risk for OCD.
Study provides experimental evidence showing that sleep deprivation induces an increase in monoamine oxidase A levels in certain brain regions, such as the amygdala and hippocampus of mice that might mediate depressive-like behaviors.
This study unravels a new link between MAO (show MAO ELISA Kits)-dependent H2O2 production and lysosomal dysfunction. Altogether, our findings demonstrate that the MAO-A/H2O2 axis has a negative impact on the elimination and recycling of mitochondria through the autophagy-lysosome pathway, which participates in cardiomyocyte death and heart failure
Results demonstrate that C17H11IOSe, a selective inhibitor of cortical MAO-A, elicited an antidepressant-like action in mice by interacting with the serotonergic system
data lead us to conclude that elevation of AP-1 (show JUN ELISA Kits) or NF-kB indirectly decreases MAO-A protein levels which, in turn, diminishes MAO-A ability in the VTA of the mesolimbic dopaminergic pathway
Huntington disease (show HTT ELISA Kits) neural cells exhibit increased Monoamine oxidase-A and Monoamine oxidases-B expression and activity
acute stress induces anxiety-like responses by affecting rapid dendritic remodeling in the pyramidal cells of orbitofrontal cortex and basolateral amygdala; furthermore, our data show that MAO-A and monoamine metabolism are required for these phenomena.
These findings demonstrate that regulation of monoamine levels by Mao (show MAO ELISA Kits) activity in beta cells is pivotal for physiological insulin (show INS ELISA Kits) secretion and that loss of MaoB (show MAOB ELISA Kits) expression may contribute to the beta cell dysfunction in type 2 diabetes.
Results suggest a role for KLF11 (show KLF11 ELISA Kits) in upregulating MAO-A in depressive disorder and chronic social stress, suggesting that inhibition of the pathways regulated by this transcription factor may aid in the therapeutics of neuropsychiatric illnesses
The results of this study suggest that heightened dACC and amygdala activation and their connectivity are neuroaffective mechanisms underlying anger control in participants with the low-functioning allele of the MAOA gene.
These results suggest that early developmental enhancements in 5-HT (show DDC ELISA Kits) levels have long-term effects on the modulation of behavioral flexibility associated with MAO-A deficiency.
Two pools of ten bovine embryos, comprised of the 4-, 8- to 16-cell, morula, blastocyst, and expanded blastocyst stages, were collected. Total RNA was isolated, and the RT-PCR-RFLP technique was used to observe expression of the MAOA gene.
MAOA was seriously demethylated or showed aberrant methylation patterns in four aborted clones.
cloning and characterization of monoamine oxidase A and B genes in pig
Social behavior was facilitated by chronic fluoxetine treatment in juvenile rhesus monkeys interacting with a familiar peer. The type of social behavior affected depended on the MAOA genotype of the monkey and its partner.
Results provide novel evidence that blood MAOA expression predicts alcohol consumption and that heavy alcohol use is linked to low MAOA expression in both the blood and NAc (show NLRP1 ELISA Kits) core.
study found the ability to consolidate sleep during the dark cycle was disrupted by prenatal iron deprivation, specifically in monkeys with the low-MAOA genotype
The effects of iron deficiency are dependent on MAOA genotype in terms of both direction and size of the effect.
rhMAOA-LPR (show FAS ELISA Kits) allele modifies the effects of maternal temperament on offspring's personality development.
Zebrafish MAO (zMAO (show MAOB ELISA Kits)) exhibits functional properties similar to human hMAO A as well as exhibits its own unique behavior.
expression, purification & characterization of zebrafish monoamine oxidase (zMAO (show MAOB ELISA Kits)) using Pichia pastoris expression system is described; zMAO (show MAOB ELISA Kits) possesses little differential sensitivity to acetylenic inhibitors than exhibited by either human MAO A & MAO B (show MAOB ELISA Kits)
this study confirm the presence of functionally active MAO (show MAOB ELISA Kits) in zebrafish brain and other tissues and characterize the neural systems that express MAO (show MAOB ELISA Kits) and areas of intense activity in the brain.
This gene is one of two neighboring gene family members that encode mitochondrial enzymes which catalyze the oxidative deamination of amines, such as dopamine, norepinephrine, and serotonin. Mutation of this gene results in Brunner syndrome. This gene has also been associated with a variety of other psychiatric disorders, including antisocial behavior. Alternatively spliced transcript variants encoding multiple isoforms have been observed.
amine oxidase [flavin-containing] A
, monoamine oxidase type A
, monoamine oxidase A
, amine oxidase [flavin-containing] A-like
, amine oxidase
, amine oxidase [flavin-containing]
, putative monoamine oxidase A