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HBx upregulated hepatic SATB1.
Invasion and metastasis of GIN in Chinese patients correlates with SATB1 overexpression in tumor tissues, most profoundly in gastric cancer.
High SATB1 expression is associated with Immunosuppressive Function of Regulatory T Cells in Chronic Hepatitis B.
SATB2 (show SATB2 Proteins) can induce dedifferentiation by inducing stemness and may have a role in pancreatic carcinogenesis
High SATB1 expression was associated with a better overall survival of patients with non-small cell lung carcinoma.ATB1 level correlates with Ki-67 (show MKI67 Proteins) expression in non-small cell lung carcinoma.
These data implied that SATB1 might regulate gene expression through its different oligomerization state.
High SATB1 expression is associated with glioblastoma.
These findings suggest SATB1 may play an important role in OSCC invasiveness and metastasis.
This paper provides evidence that SATB1 plays an oncogenic role in esophageal cancer by up-regulation of FN1 (show FN1 Proteins) and PDGFRB (show PDGFRB Proteins).
combination of the sequence-specific and nonspecific DNA-binding modes of SATB1 should be advantageous in a search for target loci during transcriptional regulation
Satb1 promotes osteoclastogenesis by recruiting CREB-binding protein (show CREBBP Proteins) to upregulate miR (show MLXIP Proteins)-223 expression in chronic kidney disease-mineral and bone disorder
SATB1 shapes the primary T cell pool by directing lineage-specific transcriptional programs in the thymus.
SATB1cKO mice, in which the SATB1 gene is specifically deleted from hematopoietic cells, develop Sjogren syndrome by 4 wk of age, soon after weaning.
RHS6 is a critical regulatory element for allergic airway inflammation and for coordinate regulation of Th2 cytokine genes by recruiting GATA3 (show GATA3 Proteins), SATB1, and IRF4 (show IRF4 Proteins).
these results identify a role for Satb1 in the lineage choice between pluripotency and differentiation and further our understanding of early embryonic lineage segregation.
This study demonstrating that Satb1-dependent Cntn5 (show CNTN5 Proteins) expression in ON-OFF direction-selective ganglion cells leads to branch-specific homophilic interactions with interneurons.
we show that special AT-rich binding protein 1 (SATB1), a T lineage-enriched chromatin organizer and regulator, is induced in response to TCR signaling during early thymocyte development
this study shows that Satb1-dependent Treg cell-specific super-enhancers activation is crucial for Treg cell lineage specification in the thymus
Tumor-driven, unremitting expression of Satb1 in activated Zbtb46 (show ZNF340 Proteins)+ inflammatory DCs that infiltrate ovarian tumors results in an immunosuppressive phenotype characterized by increased secretion of tumor-promoting Galectin-1 (show LGALS1 Proteins) and IL-6 (show IL6 Proteins).
These results suggest that SATB1 plays an essential role in establishment of immune tolerance.
This gene encodes a matrix protein which binds nuclear matrix and scaffold-associating DNAs through a unique nuclear architecture. The protein recruits chromatin-remodeling factors in order to regulate chromatin structure and gene expression. Multiple transcript variants encoding different isoforms have been found for this gene.
SATB homeobox 1
, special AT-rich sequence binding protein 1
, AT-rich binding protein-1
, DNA-binding protein SATB1-like
, DNA-binding protein SATB1
, special AT-rich sequence binding protein 1 (binds to nuclear matrix/scaffold-associating DNA)
, special AT-rich sequence-binding protein 1