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Human Monoclonal NR4A3 Primary Antibody for ELISA, WB - ABIN969434
Tsai, Chow, Chen, Huang, Hong, Jan, Kuo, Tsao, Chen, Cheng: Cef1p is a component of the Prp19p-associated complex and essential for pre-mRNA splicing. in The Journal of biological chemistry 1999
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Human Polyclonal NR4A3 Primary Antibody for IHC (p), ELISA - ABIN4369826
Nore, Mattsson, Antonsson, Bäckesjö, Westlund, Lennartsson, Hansson, Löw, Rönnstrand, Smith: Identification of phosphorylation sites within the SH3 domains of Tec family tyrosine kinases. in Biochimica et biophysica acta 2003
Show all 3 Pubmed References
Human Monoclonal NR4A3 Primary Antibody for IF, ELISA - ABIN521726
Rodríguez-Calvo, Guadall, Calvayrac, Navarro, Alonso, Ferrán, de Diego, Muniesa, Osada, Rodríguez, Martínez-González: Over-expression of neuron-derived orphan receptor-1 (NOR-1) exacerbates neointimal hyperplasia after vascular injury. in Human molecular genetics 2013
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Bat Polyclonal NR4A3 Primary Antibody for IHC (p) - ABIN270817
Nomiyama, Nakamachi, Gizard, Heywood, Jones, Ohkura, Kawamori, Conneely, Bruemmer: The NR4A orphan nuclear receptor NOR1 is induced by platelet-derived growth factor and mediates vascular smooth muscle cell proliferation. in The Journal of biological chemistry 2006
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Human Monoclonal NR4A3 Primary Antibody for IF, WB - ABIN393947
Medunjanin, Daniel, Weinert, Dutzmann, Burgbacher, Brecht, Bruemmer, Kähne, Naumann, Sedding, Zuschratter, Braun-Dullaeus: DNA-dependent protein kinase (DNA-PK) permits vascular smooth muscle cell proliferation through phosphorylation of the orphan nuclear receptor NOR1. in Cardiovascular research 2015
Human Monoclonal NR4A3 Primary Antibody for ELISA, WB - ABIN967165
van Dijk, van Den Akker, Amelsvoort, Mano, Löwenberg, von Lindern: Stem cell factor induces phosphatidylinositol 3'-kinase-dependent Lyn/Tec/Dok-1 complex formation in hematopoietic cells. in Blood 2000
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Human Polyclonal NR4A3 Primary Antibody for WB - ABIN4235536
Cramer, Shao, Wang, Yuan: miR-190 is upregulated in Epstein-Barr Virus type I latency and modulates cellular mRNAs involved in cell survival and viral reactivation. in Virology 2014
NR4A sub-family of nuclear orphan receptors (Nor-1, Nurr-1 (show NR4A2 Antibodies) and Nur-77 (show NR4A1 Antibodies)) may have a role in trophoblastic cell differentiation.
VTN (show VTN Antibodies) levels were increased in cell supernatants from vascular smooth muscle cells that overexpress NOR-1
NR4A2 (show NR4A2 Antibodies) and NR4A3 are components of a downstream transcriptional response to PKA activation in the neutrophil, and that they positively regulate neutrophil survival and homeostasis.
NOR1 suppresses cancer stem-like cell properties in nasopharyngeal carcinoma cells by inhibiting the AKT (show AKT1 Antibodies)-GSK-3beta (show GSK3b Antibodies)-Wnt (show WNT2 Antibodies)/beta-catenin (show CTNNB1 Antibodies)-ALDH1A1 (show ALDH1A1 Antibodies) signaling circuit.
Transcript analysis of four different aggressive lymphoma cell lines overexpressing either NR4A3 or NR4A1 (show NR4A1 Antibodies) revealed that apoptosis was driven similarly by induction of BAK (show BAK1 Antibodies), Puma (show BBC3 Antibodies), BIK (show BIK Antibodies), BIM (show BCL2L11 Antibodies), BID (show BID Antibodies), and Trail. Overall, our results showed that NR4A3 possesses robust tumor suppressor functions of similar impact to NR4A1 (show NR4A1 Antibodies) in aggressive lymphomas
Study found a marked down-regulated gene expression of the NR4A subfamily (NR4A1 (show NR4A1 Antibodies), NR4A2 (show NR4A2 Antibodies), and NR4A3) obtained from Parkinson's disease patients, but only a NR4A1 (show NR4A1 Antibodies) decrease in Alzheimer's disease patients compared to healthy controls. This study reports that the entire NR4A subfamily and not only NR4A2 (show NR4A2 Antibodies) could be systemically involved in Parkinson's disease.
the role of intragenic DNA hypermethylation in reducing the expression of NR4A3 in AML (show RUNX1 Antibodies)
Overexpression of NR4A3 in adipocytes produces a complex phenotype characterized by impaired glucose metabolism and low serum catecholamines due to enhanced degradation by adipose tissue.
NOR1 promotes the progression of hepatocellular carcinoma cells by activating Notch (show NOTCH1 Antibodies) pathway.
accumulation of NR4A3 is specific to alpha-synucleinopathy.
Nor-1 expression drives multiple physiological changes/pathways that are critical to the beneficial responses of muscle to exercise.
these results show that NR4A3 is involved in TLR-mediated activation and gene expression of dendritic cells
NR4A3 plays an important role in the regulation of CD103 (show ITGAE Antibodies)+ mDCs by regulating CCR7 (show CCR7 Antibodies)-dependent cell migration.
Study shows that NOR-1 can transduce survival signals in neuronal cells responsible for hypoxiainduced apoptotic insults through activation of a CREB (show CREB1 Antibodies)/cIAP2 (show BIRC3 Antibodies)-dependent mechanism
Promoting Liver X receptor-alpha (show NR1H3 Antibodies) expression inhibits lipopolysaccharide induced inflammation in Kupffer cells through elevating NOR-1 expression.
Identified Nur77 (show NR4A1 Antibodies)/Nor1 as novel regulators of thrombomodulin (show THBD Antibodies) expression and function in vascular endothelial cells.
Nr4a3 factor plays crucial role regulatory T cells, mediating the two defining characteristics of regulatory T cells: stability of cell lineage and immunosuppressive functions.
Studies demonstrate that NOR1 deletion in hematopoietic stem cells accelerates atherosclerosis formation by promoting myelopoiesis in the stem cell compartment and by inducing local proatherogenic activities in the macrophage.
Our data support a role for NOR-1 as a negative modulator of the acute response elicited by pro-inflammatory stimuli in the vasculature.
Results indicate that DNA hypomethylation of the CpG island at Nr4a3 exon 3 is associated with low Nr4a3 expression, and correlates with poor prognosis of neuroblastoma (show ARHGEF16 Antibodies).
This gene encodes a member of the steroid-thyroid hormone-retinoid receptor superfamily. The encoded protein may act as a transcriptional activator. The protein can efficiently bind the NGFI-B Response Element (NBRE). Three different versions of extraskeletal myxoid chondrosarcomas (EMCs) are the result of reciprocal translocations between this gene and other genes. The translocation breakpoints are associated with Nuclear Receptor Subfamily 4, Group A, Member 3 (on chromosome 9) and either Ewing Sarcome Breakpoint Region 1 (on chromosome 22), RNA Polymerase II, TATA Box-Binding Protein-Associated Factor, 68-KD (on chromosome 17), or Transcription factor 12 (on chromosome 15). Multiple transcript variants encoding different isoforms have been found for this gene.
chondrosarcoma, extraskeletal myxoid, fused to EWS
, mitogen-induced nuclear orphan receptor
, neuron-derived orphan receptor 1
, nuclear hormone receptor NOR-1
, nuclear receptor subfamily 4 group A member 3
, translocated in extraskeletal chondrosarcoma
, neural orphan receptor 1
, orphan nuclear receptor TEC
, neuron-derived orphan receptor 1/2
, nuclear hormone receptor NOR-1/NOR-2
, nuclear receptor subfamily 4, group A, member 3
, neural orphan nuclear receptor NOR1