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anti-Human Resistin Antibodies:
anti-Mouse (Murine) Resistin Antibodies:
anti-Rat (Rattus) Resistin Antibodies:
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Human Polyclonal Resistin Primary Antibody for IC, WB - ABIN223668
Plaisance, Lukasova, Offermanns, Zhang, Cao, Judd: Niacin stimulates adiponectin secretion through the GPR109A receptor. in American journal of physiology. Endocrinology and metabolism 2009
Show all 2 Pubmed References
Rat (Rattus) Polyclonal Resistin Primary Antibody for WB - ABIN809520
Chemaly, Kang, Zhang, McCollum, Chen, Bénard, Purushothaman, Hajjar, Lebeche: Differential patterns of replacement and reactive fibrosis in pressure and volume overload are related to the propensity for ischaemia and involve resistin. in The Journal of physiology 2013
Human Polyclonal Resistin Primary Antibody for IF (p), IHC (p) - ABIN678713
Sato, Muraoka, Kusunoki, Masuoka, Yamada, Ogasawara, Imai, Akasaka, Tochigi, Takahashi, Tsuchiya, Kawai, Nanki: Resistin upregulates chemokine production by fibroblast-like synoviocytes from patients with rheumatoid arthritis. in Arthritis research & therapy 2017
Human Polyclonal Resistin Primary Antibody for ELISA, WB - ABIN1169370
Cho, Youn, Chung, Kim, Lee, Yu, Park, Shin, Park: Common genetic polymorphisms in the promoter of resistin gene are major determinants of plasma resistin concentrations in humans. in Diabetologia 2004
The effects of exogenous human kisspeptin-10 (KP10) were studied on three important adipokines, namely, adiponectin, leptin, and resistin in a set of four chair-restraint habituated intact adult male rhesus monkeys.
Results provide evidence that Visfatin and resistin regulate the expression levels of some miRNA involved in osteoarthritis pathogenesis and exert catabolic functions in chondrocytes via the NF-kappaB pathway.
Higher circulating resistin correlated with node-negative patients and longer DFS independent of Body mass index and estrogen receptor status in women with Early-onset breast cancer
Resistin 420G > C and 62G > A gene polymorphisms play a role in Endometrial Cancer (EC) development. Multivariate regression analysis revealed that the presence of 420G + allele increased the EC risk 1.99 fold while the presence of 62A + allele was shown to decrease the risk of EC.
Resistin can be released by degranulating neutrophils and blunts neutrophil plaque infiltration by modulating their migration towards known atherosclerotic mediators.
This study is the first to examine the risk factors associated with RETN single nucleotide polymorphisms in Oral squamous cell carcinoma progression and development in Taiwan.
providing evidence for a causal role of resistin in shaping the risk of mortality in diabetic patients.
The data revealed no correlation between the Resistin rs3745367 polymorphism and cancer risk.
serum level higher among infants with severe sepsis and among those who needed mechanical ventilation
in patients with coronary artery disease and without acute myocardial infarction leptin may represent a potential mechanism of adverse cardiac remodeling. Resistin and TNF-alpha might not be involved in ventricular remodeling in these patients.
The results of this study indicated that resistin gene polymorphisms might affect the genetic predisposition of rheumatoid arthritis in Chinese population.
It was demonstrated that high resistin expression was predominantly observed in lung adenocarcinoma tissues. It is associated with a more malignant clinicopathological status and poorer survival. Resistin could promote A549 and H1975 tumor cell proliferation, migration and invasion while inhibit their apoptosis in vitro.
A multi-cytokine resistin pathway is operational in humans, and it plays a substantial role in cardiovascular events in high-risk individuals.
An increase in resistin concentration expressed also as resistin/BMI, and resistin/adiponectin ratios, observed in children with cystic fibrosis may suggests that this adipokine is involved in the inflammatory process underlying the disease and is related to worse spirometric parameters describing airways obstruction.
n T2 mice, the mRNA expression of Retn showed a moderate up-regulation (fold change=8.32; p=0.0019) in the adipose tissues. Iapp expression was also significantly up-regulated (fold change=9.78; p=0.012). Moreover, a 6.36-fold up-regulation for Drd5 was observed in the adipose tissues of T2 mice
RETN and CAP1 polymorphisms and gene expression may be potential biomarkers for breast cancer risk
resistin induced MUC5AC and MUC5B expression via activation of different signaling pathways in human airway epithelial cells.
Study provides clear evidence that resistin is a clinically relevant endogenous ligand for TLR4, which promotes tumor progression via TLR4/NF-kappaB/STAT3 signaling.
women with gestational diabetes showed significantly low adiponectin and high resistin levels when compared with control group.
increased levels of resistin in erosive and non-erosive hand osteoarthritis
Resistin promotes a shift from proliferation to apoptosis in vascular smooth muscle cells and macrophage co-culture systems with cellular composition similar to that found in vulnerable regions of plaques.
Resistin inhibited the activation of CREB, resulting in suppression of PGC-1alpha. Importantly, overexpressing PGC-1alpha can rescue the inhibitory effects of resistin on fatty acid beta oxidation.
Resistin-associated VSMC dysfunction and intimal hyperplasia are related to PKCepsilon-dependent Nox activation and ROS generation
Resistin may enhance inflammation by cross-talking with TLR4/NF-kappaB signaling during the development of coronary arteritis in mice
OLI is a physiological repressor of systemic resistin release whereas FFA upregulate resistin release in vitro from adipocytes.
Resistin possibly acts via an intracrine pathway as an intracellular sensor, regulating the adipocyte insulin sensitivity.
In 3T3-L1 adipocytes, catechin and quercetin attenuated TNF-alpha-induced elevated protein carbonyls, increased proinflammatory cytokine expression (MCP-1, resistin), and decreased adiponectin.
Mouse Resistin has regulative effects on murine bone marrow hematopoiesis.
murine resistin is increased in the lungs of wild-type mice following acute ozone exposure but does not promote ozone-induced lung pathology.
Inhibition of miR-696 restored the triglycerides content by up to 80%, which suggests that, in C2C12 cells, resistin at least partially increases the deposition of lipids through miR-696.
Antiresistin RNA oligo ameliorated metabolic abnormalities and histopathology of nonalcoholic fatty liver disease through attenuating proinflammatory cytokines
Resistin regulates PAI-1 expression in 3T3-L1 adipocytes via Akt phosphorylation.
Oxidized-LDL promotes the expression and secretion of visfatin and resistin through its activation of endoplasmic reticulum stress.
ET type-A receptor, ERK1/2, JNKs, AKT, and JAK2, but not ET type-B receptor or p38, are necessary for the ET-1 stimulation of resistin gene expression
resistin is a negative regulator of SIRT1 in both human hepatoma cell line HepG2 and mouse hepatocytes.
Both resistin gene expression and its secretion from the epididymal adipose tissue were decreased in db/db obese mice, while the insulin-sensitizing drug rosiglitazone increased resistin production.
lipid content was significantly decreased in 3T3-L1 cells transfected with anti-resistin small interfering RNA (siRNA) after adipocyte differentiation
The effect between resistin and adiponectin on the proliferation of vascular smooth muscle cells (VSMCs), was investigated.
The current study was initiated to determine whether resistin has additional roles in glucose-dependent insulinotropic polypeptide -regulated adipocyte functions.
significant changes in fasting-induced adipose factor (FIAF), resistin (Rstn), long-form leptin receptor (LepRb) and suppressor of cytokine-3 (SOCS3) gene expression following chronic SCI and with advanced age
results identify resistin as a potential inhibitory ligand for the receptor ROR1 and demonstrate, for the first time, that ROR1 plays an important role in adipogenesis and glucose homeostasis in 3T3-L1 cellsand glucose homeostasis in 3T3-L1 cells.
The data suggest that there is local cooperation between resistin and PPARgamma expression in the porcine ovary. Resistin significantly increased the expression of PPARgamma, whereas PPARgamma decreased resistin expression; thus, PPARgamma is a new key regulator of resistin expression and function.
The expression, immunolocalization and concentration of resistin in different sized ovarian follicles, was investigated.
This gene belongs to the family defined by the mouse resistin-like genes. The characteristic feature of this family is the C-terminal stretch of 10 cys residues with identical spacing. The mouse homolog of this protein is secreted by adipocytes, and may be the hormone potentially linking obesity to type II diabetes. Alternatively spliced transcript variants encoding the same protein have been found for this gene.
, C/EBP-epsilon regulated myeloid-specific secreted cysteine-rich protein precursor 1
, adipose tissue-specific secretory factor
, c/EBP-epsilon-regulated myeloid-specific secreted cysteine-rich protein
, cysteine-rich secreted protein A12-alpha-like 2
, cysteine-rich secreted protein FIZZ3
, found in inflammatory zone 3
, resistin delta2
, adipose-specific cysteine-rich secreted protein A12-alpha
, dominant inhibitory adipocyte-specific secretory factor
, adipocyte specific secreted hormone