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anti-Human PI4KB Antibodies:
anti-Rat (Rattus) PI4KB Antibodies:
anti-Mouse (Murine) PI4KB Antibodies:
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PI4KIIIbeta interaction with the VHS domain of GGA2 (show GGA2 Antibodies) affected PI4KIIIbeta localization.
several disordered regions of PI4KB become protected from proteolytical degradation upon 14-3-3 (show YWHAQ Antibodies) binding.
esults show that Aichi virus 3A protein activates the lipid kinase activity of PI4KIIIb,which activation is sensitized by the protein ACBD3 (show Acbd3 Antibodies). The interfaces between PI4KIIIbeta-ACBD3 (show Acbd3 Antibodies) and ACBD3 (show Acbd3 Antibodies)-3A were mapped with hydrogen-deuterium exchange mass spectrometry.
The results showed that, in contrast to the enteroviruses and the cardioviruses, foot-and-mouth disease virus replication does not require PI4KIII (PI4KIIIalpha and PI4KIIIbeta), and phosphatidylinositol 4-phosphate levels do not increase in foot-and-mouth disease virus-infected cells and phosphatidylinositol 4-phosphate is not seen at replication organelles.
Data show that ACBD3 (show Acbd3 Antibodies) can recruit PI4KB to model membranes as well as redirect PI4KB to cellular membranes where it is not naturally found. Also, results show that ACBD3 (show Acbd3 Antibodies) regulates the enzymatic activity of PI4KB kinase through membrane recruitment rather than allostery.
Analysis reveals novel aspects of the PI4KIIIb-Rab11 complex and determines binding and catalytic sites of the kinase.
PI4KIIIbeta likely plays a role in breast oncogenesis and that cooperation between Rab11a (show RAB11A Antibodies) and PI4KIIIbeta represents a novel Akt (show AKT1 Antibodies) activation pathway.
Although human rhinovirus 3A protein was previously shown to interact with ACBD3 (show Acbd3 Antibodies), these data suggest that PI4KIIIbeta recruitment occurred independently of both GBF1 (show GBF1 Antibodies) and ACBD3 (show Acbd3 Antibodies).
Authors found that NS5A and PI4KB competed for association of acyl-coenzyme A (show SOAT2 Antibodies) binding domain containing protein 3 (show HSPB3 Antibodies) (ACBD3 (show Acbd3 Antibodies)), which inhibited hepatitis C virus replication.
These results suggest that poliovirus proteins modulate PI4KB activity and provide PI4P for recruitment of OSBP (show OSBP Antibodies) to accumulate unesterified cholesterol on virus-induced membrane structure for formation of a virus replication complex.
findings reveal a novel signalling pathway involved in development of the semicircular canal system, and suggest a previously unrecognized role for NCS-1 (show NCS1 Antibodies) in mitochondrial function via its association with several mitochondrial proteins.
Depolarization increases phosphatidylinositol (PI) 4,5-bisphosphate level and KCNQ currents through PI 4-kinase mechanisms
Phosphorylates phosphatidylinositol (PI) in the first committed step in the production of the second messenger inositol- 1,4,5,-trisphosphate (PIP) (By similarity).
phosphatidylinositol 4-kinase, catalytic, beta polypeptide
, phosphatidylinositol 4-kinase, catalytic, beta
, phosphatidylinositol 4-kinase beta
, PtdIns 4-kinase beta
, phosphatidylinositol 4-kinase beta-like
, phosphatidylinositol 4-kinase, wortmannin-sensitive
, type III phosphatidylinositol 4-kinase beta
, catalytic phosphatidylinositol 4-kinase beta
, ptdIns 4-kinase beta
, phosphatidylinositol 4-kinase III beta