Angiotensinogen (serpin Peptidase Inhibitor, Clade A, Member 8) (AGT) Peptide
AGT
Reactivity: Mammalian
Host: Synthetic
WB, BP, IHC
Catalog No. ABIN939371
Quick Overview for Angiotensinogen (serpin Peptidase Inhibitor, Clade A, Member 8) (AGT) Peptide (ABIN939371)
Target
AGT
(Angiotensinogen (serpin Peptidase Inhibitor, Clade A, Member 8) (AGT))
Origin
Mammalian
Source
Synthetic
Application
Western Blotting (WB), Blocking Peptide (BP), Immunohistochemistry (IHC)
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Peptide Type
- Synthetic
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Sequence
- IHPFHLVIHN ESTCEQLAKA NAGKPKDPTF IPAPIQAKTS PVDEKALQDQ
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Characteristics
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A synthetic peptide for use as a blocking control in assays to test for specificity of AGT antibody,
Alternative Names: AGT control peptide, AGT antibody Blocking Peptide, Anti-AGT Blocking Peptide, Angiotensinogen Blocking Peptide, Serpin Peptidase Inhibitor Clade A 8 Blocking Peptide, ANHU Blocking Peptide, SERPINA8 Blocking Peptide
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Angiotensinogen (serpin Peptidase Inhibitor, Clade A, Member 8) (AGT) peptide (Ovalbumin)
AGT Reactivity: Human, Mouse, Rat Host: Synthetic WB, Imm, SDS
Angiotensinogen (serpin Peptidase Inhibitor, Clade A, Member 8) (AGT) (AA 34-43) peptide (Ovalbumin)AGT Reactivity: Human Host: Synthetic WB, Imm, SDS
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Application Notes
- Optimal conditions should be determined by the investigator
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Restrictions
- For Research Use only
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Format
- Lyophilized
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Reconstitution
- Add 100 µL of distilled water for a final peptide concentration is 1 mg/mL.
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Buffer
- PBS
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Handling Advice
- Avoid repeated freeze/thaw cycles.
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Storage
- -20 °C
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Storage Comment
- Store at -20 °C long term.
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- AGT (Angiotensinogen (serpin Peptidase Inhibitor, Clade A, Member 8) (AGT))
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Background
- AGT, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
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Molecular Weight
- 53 kDa
Target
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