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anti-Human ARID5B Antibodies:
anti-Mouse (Murine) ARID5B Antibodies:
anti-Cow (Bovine) ARID5B Antibodies:
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Human Polyclonal ARID5B Primary Antibody for ICC, IF - ABIN4281577
Hata, Takashima, Amano, Ono, Nakanishi, Yoshida, Wakabayashi, Matsuda, Maeda, Suzuki, Sugano, Whitson, Nishimura, Yoneda: Arid5b facilitates chondrogenesis by recruiting the histone demethylase Phf2 to Sox9-regulated genes. in Nature communications 2013
Show all 2 Pubmed References
Cow (Bovine) Polyclonal ARID5B Primary Antibody for WB - ABIN610895
Aspberg, Miura, Bourdoulous, Shimonaka, Heinegârd, Schachner, Ruoslahti, Yamaguchi: The C-type lectin domains of lecticans, a family of aggregating chondroitin sulfate proteoglycans, bind tenascin-R by protein-protein interactions independent of carbohydrate moiety. in Proceedings of the National Academy of Sciences of the United States of America 1997
arid5b isoform1 and isoform2 are differentially expressed during development. Isoform1 is strongly expressed maternally, while isoform2 expression is essentially restricted to tailbud stages. arid5b is predominantly expressed in the developing pronephros.
Differential methylation and expression of ARID5B to be associated with human atherosclerosis.
The present results identified MRF2/ARID5B as a potential susceptibility gene for new-onset T2DM in a Northern Chinese population, while the rs7087507 SNP was associated with HDL-C levels. Further larger studies are required to validate these findings.
We systematically screened 6 potentially functional SNPs in ARID5B and IKZF1 genes.
ARID5B reinforces the oncogenic transcriptional program by positively regulating the TAL1-induced regulatory circuit and MYC in T-ALL, thereby contributing to T-cell leukemogenesis.
Results identified a novel hotspot in the terminal exon of ARID5B.The ARID5B indel mutations were seen in both mismatch repair (MMR)-deficient and MMR-normal tumors, suggesting biologic selection in endometrial neoplasm.
Our results confirmed the role of ARID5B in childhood ALL susceptibility among Hispanics; however, our assessment did not reveal any strong novel inherited genetic risks for acute lymphoblastic leukemia among this ethnic group.
Associations between ALL and rs10821936 and rs7089424 ARID5B SNPs were found
ARID5B variants are associated with acute lymphoblastic leukemia in Yemeni children with several gender biases of ARID5B single nucleotide polymorphisms reported.
variants within IKZF1, ARID5B, and CEBPE were associated with pediatric ALL risks.
Genotypic and allelic frequencies differed significantly between cases and controls at IKZF1-rs4132601 (p=0.039, p=0.015) and ARID5B-rs10821936 (p=0.028, p=0.026).
Single nucleotide polymorphisms rs10994982 and rs7089424 in ARID5B gene are associated with increased risk of childhood acute lymphoblastic leukemia.
This study showed that rs4948496 in ARID5B is associated with several subphenotypes of systemic lupus erythematosus and this gene may cause the complicacy of clinical features.
Non-coding variant rs10821936 in ARID5B was strongly associated with childhood B-cell acute lymphoblastic leukemia. No coding variants were associated with B-ALL susceptibility.
variants within IKZF1, ARID5B, and CEBPE were associated with increased acute lymphoblastic leukemia (ALL) risk, and the effects for ARID5B and CEBPE were most prominent in high-hyperdiploid ALL subtype in the California Hispanic population
Data indicate no significant associations of transcription factors rs4132601 (IKZF1), rs7089424 (ARID5B) and rs2239633 (CEBPE) with risk of pediatric non-Hodgkin lymphoma (NHL).
Interactions between rs10740055 in ARID5B or rs4132601 in IKZF1 and each of the suspected non-genetic factors were tested.
These results evidence that interaction of genetic variants in ARID5B and IKZF1 and environmental exposures may further alter risk of childhood acute lymphoblastic leukemia.
ARID5B polymorphism confers an increased risk to acquire specific MLL rearrangements in early childhood leukemia.
Our findings confirm the association of novel genetic variations in folate-related and ARID5B genes with the serum MTX levels and acute toxicity.
We found that rs7073837 in ARID5B correlated with a risk for childhood B lineage ALL.
This gene encodes a member of the AT-rich interaction domain (ARID) family of DNA binding proteins. The encoded protein forms a histone H3K9Me2 demethylase complex with PHD finger protein 2 and regulates the transcription of target genes involved in adipogenesis and liver development. This gene also plays a role in cell growth and differentiation of B-lymphocyte progenitors, and single nucleotide polymorphisms in this gene are associated with acute lymphoblastic leukemia. Alternatively spliced transcript variants encoding multiple isoforms have been observed for this gene.
AT rich interactive domain 5B (MRF1-like)
, AT-rich interactive domain-containing protein 5B
, AT-rich interactive domain-containing protein 5B-like
, ARID domain-containing protein 5B
, MRF1-like protein
, modulator recognition factor 2 (MRF2)
, ARID class DNA binding protein
, AT rich interactive domain 5B (Mrf1 like)
, developmentally and sexually retarded with transient immune abnormalities protein
, modulator recognition factor 2
, modulator recognition factor protein 2