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anti-Mouse (Murine) ATP5A1 Antibodies:
anti-Human ATP5A1 Antibodies:
anti-Rat (Rattus) ATP5A1 Antibodies:
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Upregulation of ATP5A1 increased ATP5A1 and ATP synthase activity, prevented mitochondrial ROS (show ROS1 Antibodies) generation and oxidative damage, and reduced cardiomyopathic changes in diabetic mice.
ATP5alpha1 is an interacting subunit of the ATP synthase complex localized to the mitochondria with aryl hydrocarbon receptor (show AHR Antibodies) in the absence of ligand.
Using systems biology techniques to study gene coexpression network, ATP5A1 was identified and found highly expressed in normal kidney than clear cell renal cell carcinoma (show MOK Antibodies) (ccRCC) tissues of each grade. Functional and pathway enrichment analysis demonstrated that ATP5A1 is overrepresented in pathway of oxidative phosphorylation, which associated with tumorigenesis and tumor progression.
This cohort study showed that the ATP5A1 gene was down regulation between patients with idiopathic Parkinson disease and controls
these data highlight a key role of the P2Y1 (show P2RY1 Antibodies)/PI3Kbeta axis in endothelial cell proliferation downstream of ecto (show TRIM33 Antibodies)-F1-ATPase (show DNAH8 Antibodies) activation by apoA-I (show APOA1 Antibodies). Pharmacological targeting of this pathway could represent a promising approach to enhance vascular endothelial protection.
High mRNA levels of ATP5A1 are associated with glioblastoma.
Hemoglobin (show HBB Antibodies) - a novel ligand of hepatocyte ectopic F1-ATPase (show DNAH8 Antibodies)
Here, we found that ATP synthase subunit alpha (ATP5A) was O-GlcNAcylated at Thr432 and ATP5A O-GlcNAcylation was decreased in the brains of AD patients and transgenic mouse model
Mitochondrial calpain-1 (show CAPN1 Antibodies) disrupts ATP synthase, leading to mitochondrial reactive oxygen species generation, which promotes proinflammatory response and myocardial dysfunction during endotoxemia.
Studies indicate that the F-ATP synthase can reversibly undergo a Ca(2 (show CA2 Antibodies)+)-dependent transition to form a channel that mediates the permeability transition.
A mutation in ATP5A1 causes a fatal neonatal mitochondrial encephalopathy.
Studies indicate that F1-ATPase (show DNAH8 Antibodies) (F1) is a rotary motor protein driven by ATP hydrolysis and the minimum complex of F1 for function as a rotary motor is the alpha3beta3gamma subcomplex.
F1-ATPase (show DNAH8 Antibodies) at the cell surface of colonic epithelial cells has a role in mediating cell proliferation
Nuclear magnetic resonance characterization of the interaction between OSCP (show ATP5O Antibodies)-N terminal and a peptide corresponding to residues 1-25 of the alpha-subunit (show POLG Antibodies) of bovine F(1)-ATPase is (show ATP11A Antibodies) presented.
This gene encodes a subunit of mitochondrial ATP synthase. Mitochondrial ATP synthase catalyzes ATP synthesis, using an electrochemical gradient of protons across the inner membrane during oxidative phosphorylation. ATP synthase is composed of two linked multi-subunit complexes: the soluble catalytic core, F1, and the membrane-spanning component, Fo, comprising the proton channel. The catalytic portion of mitochondrial ATP synthase consists of 5 different subunits (alpha, beta, gamma, delta, and epsilon) assembled with a stoichiometry of 3 alpha, 3 beta, and a single representative of the other 3. The proton channel consists of three main subunits (a, b, c). This gene encodes the alpha subunit of the catalytic core. Alternatively spliced transcript variants encoding the different isoforms have been identified. Pseudogenes of this gene are located on chromosomes 9, 2, and 16.
ATP synthase subunit alpha, mitochondrial
, mitochondrial H+ transporting ATP synthase F1
, ATP synthase, H+ transporting, mitochondrial F1 complex, alpha subunit 1, cardiac muscle
, ATP synthase H+ transporting mitochondrial F1 complex alpha subunit 1
, ATP synthase, H+ transporting, mitochondrial F1 complex, alpha subunit-like
, ATP synthase subunit alpha, mitochondrial-like
, ATP synthase, H+ transporting, mitochondrial F1 complex, alpha subunit
, modifier of Min 2
, ATP synthase alpha chain, mitochondrial
, ATP synthase, H+ transporting, mitochondrial F1 complex, alpha subunit, isoform 1, cardiac muscle
, ATP synthase, H+ transporting, mitochondrial F1 complex, alpha subunit, isoform 2, non-cardiac muscle-like 2
, ATP sythase (F1-ATPase) alpha subunit
, mitochondrial ATP synthetase, oligomycin-resistant
, mitochondrial H+-ATP synthase alpha subunit
, mitochondrial ATP synthase alpha subunit
, ATP synthase alpha subunit
, cardiac muscle ATP synthase H+ transporting mitochondrial F1 complex alpha subunit 1