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Hamster Monoclonal NFATC1 Primary Antibody for ChIP, GS - ABIN152773
Alpini, Franchitto, Demorrow, Onori, Gaudio, Wise, Francis, Venter, Kopriva, Mancinelli, Carpino, Stagnitti, Ueno, Han, Meng, Glaser: Activation of alpha(1) -adrenergic receptors stimulate the growth of small mouse cholangiocytes via calcium-dependent activation of nuclear factor of activated T cells 2 and specificity protein 1. in Hepatology (Baltimore, Md.) 2011
Human Polyclonal NFATC1 Primary Antibody for ICC, IF - ABIN4339291
Mack, Mosqueiro, Archer, Jones, Sunshine, Faas, Briot, Aragón, Su, Romay, McDonald, Kuo, Lizama, Lane, Zovein, Fang, Tarling, de Aguiar Vallim, Navab, Fogelman, Bouchard, Iruela-Arispe: NOTCH1 is a mechanosensor in adult arteries. in Nature communications 1970
Loss of NFATC1 is associated with Bone resorption.
Berberine hydrochloride targets TRAF6 (show TRAF6 Antibodies) and NFATc1.
RCANs play critical roles in bone homeostasis by regulating both osteoclastogenesis and osteoblastogenesis, and they serve as inhibitors for calcineurin-NFATc1 signaling both in vivo and in vitro.
APC (show APC Antibodies) defines Treg differentiation and anti-inflammatory function through microtubule-mediated NFAT localization.
Tusc2/Fus1 (show TUSC2 Antibodies) regulates osteoclast differentiation through NF-kappaB (show NFKB1 Antibodies) and NFATc1
these data demonstrated that wedelolactone facilitated osteoblastogenesis through Wnt (show WNT2 Antibodies)/GSK3beta/beta-catenin (show CTNNB1 Antibodies) signaling pathway and suppressed RANKL (show TNFSF11 Antibodies)-induced osteoclastogenesis through NF-kappaB (show NFKB1 Antibodies)/c-fos/NFATc1 pathway.
data demonstrate that NC suppressed osteoclastogenesis and prevented OVX-induced bone loss by inhibiting RANKL (show TNFSF11 Antibodies)-induced NF-kappaB (show NFKB1 Antibodies) and NFATc1 signalling pathways. NC may be a natural and novel treatment for osteoclast-related bone lytic diseases.
mTORC1 Inhibits NF-kappaB (show NFKB1 Antibodies)/NFATc1 Signaling and Prevents Osteoclast Precursor Differentiation, In Vitro and In Mice
CD137 (show TNFRSF9 Antibodies) signaling is a new regulator of angiogenesis by modulating the Smad1 (show SMAD1 Antibodies)/5-NFATc1 pathway.
These results suggest that the PDZ domain (show INADL Antibodies) of PICK1 (show PICK1 Antibodies) directly interacts with calcineurin B (show CAN Antibodies) in osteoclast progenitor cells and promotes osteoclast differentiation through activation of calcineurin-NFAT signaling.
NFATc1 knockdown strongly reduced the number and the surface area of myotubes, NFATc4 (show NFATC4 Antibodies) knockdown increased the surface area of myotubes and reduced the pool of reserve cells.
NFAT2 is an important regulator for the anergic phenotype of chronic lymphocytic leukaemia.
Exposure to UVB radiation induces nuclear translocation and stimulates binding between NFAT5 (show NFAT5 Antibodies) and NF-kappaB (show NFKB1 Antibodies) proteins in HLE (show ELANE Antibodies)-B3 cells. These interactions may form part of the biochemical mechanism of cataractogenesis in UVB-irradiated HLECs.
data identified a novel role of SFKs in preventing aberrant NFAT1 (show NFAT1 Antibodies) activation in resting T cells, and suggest that maintaining this pool of active SFKs in therapeutic T cells may increase the efficacy of T cell therapies
Pathway analysis of the genes associated with the 46 CpG sites revealed an enrichment of immune system process genes, including LYST (show LYST Antibodies) (cg16962115, FDR = 1.24E-04), CADM1 (show CADM1 Antibodies) (cg21933078, FDR = 1.22E-02) and NFATC1 (cg06784563, FDR = 1.46E-02)
there is an NFATc1/ABCA1 (show ABCA1 Antibodies)-dependent mechanism in which local TNF (show TNF Antibodies) is sufficient to cause free cholesterol-dependent podocyte injury irrespective of TNF (show TNF Antibodies), TNFR1 (show TNFRSF1A Antibodies), or TNFR2 (show TNFRSF1B Antibodies) serum levels
NFATC1 transcription factor (NFATc1) expression is detected in prostate cancer (PCa (show FLVCR1 Antibodies)) specimens and PCa (show FLVCR1 Antibodies) cells but is absent in non-neoplastic human prostates and non-tumorigenic prostatic cells
DYRK1A phosphorylation of NFATc1/alphaA at S261, S278, S403 and S409 interfered with NFATc1 ubiquitination and ubiquitin-proteasome degradation.
In studies of human and mouse pancreatic cells and tissue, we identified context-specific epigenetic regulation of NFATc1 activity as an important mechanism of pancreatic cell plasticity.
In calf pulmonary artery endothelial cells, extracellular application of vasoactive agonist ATP or calcium ionophore ionomycin generates a rise in intracellular calcium that induces nuclear localization of NFATc1.
regulation of nuclear localization of NFAT is isoform-specific and dependent on nuclear export processes
The product of this gene is a component of the nuclear factor of activated T cells DNA-binding transcription complex. This complex consists of at least two components: a preexisting cytosolic component that translocates to the nucleus upon T cell receptor (TCR) stimulation, and an inducible nuclear component. Proteins belonging to this family of transcription factors play a central role in inducible gene transcription during immune response. The product of this gene is an inducible nuclear component. It functions as a major molecular target for the immunosuppressive drugs such as cyclosporin A. Five transcript variants encoding distinct isoforms have been identified for this gene. Different isoforms of this protein may regulate inducible expression of different cytokine genes.
nuclear factor of activated T-cells, cytoplasmic, calcineurin-dependent 1
, nuclear factor of activated T-cells, cytosolic component 1
, nuclear factor of activated T-cells calcineurin-dependent 1
, nuclear factor of activated T-cells, cytoplasmic 1
, nuclear factor of activated T-cells, cytoplasmic 1-like
, NFAT transcription complex cytosolic component
, transcription factor NF-ATc
, transcription factor NFATmac