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anti-Mouse (Murine) NFATC3 Antibodies:
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Human Polyclonal NFATC3 Primary Antibody for IF, WB - ABIN518311
Vihola, Sirito, Bachinski, Raheem, Screen, Suominen, Krahe, Udd: Altered expression and splicing of Ca(2+) metabolism genes in myotonic dystrophies DM1 and DM2. in Neuropathology and applied neurobiology 2013
MicroRNA-214 regulates immunity-related genes in bovine mammary epithelial cells by targeting NFATc3 and TRA
regulation of nuclear localization of NFAT (show NFATC1 Antibodies) is isoform-specific and dependent on nuclear export processes
FZD5 is a receptor for SFRP2 (show SFRP2 Antibodies) and mediates SFRP2 (show SFRP2 Antibodies)-induced angiogenesis via calcineurin/NFATc3 pathway in endothelial cells.
Redirected splicing of calcineurin A (show CAN Antibodies) to the fetal isoforms in adult muscle and in differentiated C2C12 slows the timing of muscle relaxation, promotes nuclear localization of calcineurin target Nfatc3, and/or affects expression of Nfatc (show NFATC1 Antibodies) transcription targets.
Our study demonstrates that Orai1 (show TMEM132A Antibodies)-Ca(2 (show CA2 Antibodies)+)-calcineurin-NFATc4 (show NFATC4 Antibodies) signaling is an essential inflammatory pathway required for TNFalpha (show TNF Antibodies)-induced endothelial cell activation and vascular inflammation. Therefore, Orai1 (show TMEM132A Antibodies) may be a potential therapeutic target for treatment of inflammatory diseases.
Both NFATc3 knock-out mice and ILK (show ILK Antibodies) conditional-knockdown mice (cKD-ILK (show ILK Antibodies)) display symptoms of NDI (show AVPR2 Antibodies) (polyuria and reduced AQP2 (show AQP2 Antibodies) expression).
we found that VIP (show Vip Antibodies) inhibits NFAT (show NFATC1 Antibodies) nuclear translocation in primary human pulmonary artery smooth muscle cells (PASMC). Early activation of NFATc3 in IPF patients may contribute to disease progression and the increase in VIP (show Vip Antibodies) expression could be a protective compensatory mechanism
the transcription factor NFATC3 binds to IRF7 (show IRF7 Antibodies) and functions synergistically to enhance IRF7 (show IRF7 Antibodies)-mediated IFN expression in Plasmacytoid dendritic cells.
Ca(2 (show CA2 Antibodies)+) influx through ASIC1 (show ACCN2 Antibodies) mediates chronic hypoxia and ET-1 (show EDN1 Antibodies)-induced NFATc3 nuclear import and 2) the scaffolding protein PICK1 (show PICK1 Antibodies) is necessary for NFATc3 nuclear import.
AKAP150-calcineurin signaling dyad is essential for the activation of the phosphatase and the subsequent down-regulation of Kv channel currents following myocardial infarction.
Study defines the NFAT4/ miR (show MLXIP Antibodies)-324-5p/Mtfr1 (show MTFR1 Antibodies) axis, which participates in the regulation of mitochondrial fission and cardiomyocyte apoptosis.
Disrupting the calcineurin-NFAT (show NFATC1 Antibodies) axis by either genetic or pharmacologic approaches confers resistance to the development of social stress-induced voiding and dysfunction.
The NFATc3 first induced the expression of its interaction partner FosB (show FOSB Antibodies) before forming the heterodimeric NFATc3-FosB (show FOSB Antibodies) transcription factor complex, which bound the proximal AP-1 (show FOSB Antibodies) site in the TF gene promoter and activated TF expression.
NFAT1 (show NFAT1 Antibodies) is stimulated by subplasmalemmal Ca2 (show CA2 Antibodies)+ microdomains, whereas NFAT4 additionally requires Ca2 (show CA2 Antibodies)+ mobilization from the inner nuclear envelope by nuclear InsP3 receptors.
Calcineurin together with its upstream molecule, calpain 2 (show CAPN2 Antibodies), and its downstream effector, NFAT (show NFATC1 Antibodies)-c3, might contribute to the development of atrial fibrillation in patients with heart valve disease and diabetes.
two NFAT isoforms (NFAT4 and NFAT1) have shifted band-pass windows for the same receptor in the GPCR signaling pathway
Data indicate that RNA interference of NFAT (show NFATC1 Antibodies) isoforms NFATc1 (show NFATC1 Antibodies), NFATc2 (show NFAT1 Antibodies), NFATc3 and NFATc4 (show NFATC4 Antibodies) regulate gene expression differentially in human retinal microvascular endothelial cells (HRMEC).
NFATc3 interacted in a SUMO-dependent manner with Trim17 (show TRIM17 Antibodies), an E3 ubiquitin ligase (show MUL1 Antibodies) necessary for neuronal apoptosis
Results show that two protein isoforms NFAT1 (show NFAT1 Antibodies) and NFAT4 are both cytosolic and stimulated by the same Ca2 (show CA2 Antibodies)+ messenger but require distinct subcellular Ca2 (show CA2 Antibodies)+ signals for activity.
NF-AT affects neural convergent extension.
transcriptional control of morphological and electrophysiological properties of neurons is mediated by distinct NFAT (show NFATC1 Antibodies) interactions
The product of this gene is a member of the nuclear factors of activated T cells DNA-binding transcription complex. This complex consists of at least two components: a preexisting cytosolic component that translocates to the nucleus upon T cell receptor (TCR) stimulation and an inducible nuclear component. Other members of this family participate to form this complex also. The product of this gene plays a role in the regulation of gene expression in T cells and immature thymocytes. Several transcript variants encoding distinct isoforms have been identified for this gene.
cytoplasmic nuclear factor of activated T-cells 3
, nuclear factor of activated T-cells, cytoplasmic, calcineurin-dependent 3
, nuclear factor of activated T-cells, cytoplasmic 3
, nuclear factor of activated T-cells, cytoplasmic 3-like
, T-cell transcription factor NFAT4
, T cell transcription factor NFAT4
, nuclear factor of activated T-cells c3 isoform IE-Xa
, nuclear factor of activated T-cell