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Apolipoprotein H has been implicated in a variety of physiologic pathways including lipoprotein metabolism, coagulation, and the production of antiphospholipid autoantibodies. Additionally we are shipping APOH Antibodies (151) and APOH Proteins (35) and many more products for this protein.
Showing 10 out of 79 products:
Human APOH ELISA Kit for Sandwich ELISA - ABIN414966
Lackner, Beck, Eichmeir, Gemeiner, Hummel, Pullirsch, Razzazi-Fazeli, Seifner, Miller: Contamination of therapeutic human immunoglobulin preparations with apolipoprotein H (?2-glycoprotein I). in Electrophoresis 2014
Human APOH ELISA Kit for Sandwich ELISA - ABIN2683481
Kim, Park, Heo, Moon, Park: Plasma apolipoprotein H levels are different between aspirin induced respiratory diseases and aspirin tolerant asthma. in Pulmonary pharmacology & therapeutics 2014
These results suggested that CRP (show CRP ELISA Kits)/oxidized low density lipoprotein/beta 2-Glycoprotein I complexes may induce lipid accumulation and inflammation in macrophages via the p38/MAPK (show MAPK14 ELISA Kits) and NFkappaB signaling pathways.
TLR2 (show TLR2 ELISA Kits) increase the stimulating effect of beta2GP1/anti-beta2GP1 complex on the expression of TNF-alpha (show TNF ELISA Kits) in mouse peritoneal macrophages
The Fifth Domain of Beta 2 Glycoprotein I Protects from Natural IgM Mediated Cardiac Ischaemia Reperfusion Injury.
These results indicate that both NF-kappaB (show NFKB1 ELISA Kits) and c-Jun/AP-1 (show JUN ELISA Kits) are involved in regulating anti-beta2GPI-induced expression of prothrombotic and proinflammatory molecules in vivo.
Phospholipid scramblase 1 regulates phosphatidylserine exposure in response to oxygen stress, leading to beta2-glycoprotein I and IgM binding and lipid-mediated, inflammatory responses.
beta2-GPI-D-I-tDCs may offer a novel approach for developing therapy for APS (show SH2B2 ELISA Kits) patients
Deletion of beta2 GPI accelerates and potentiates the autoimmune phenotype in male BXSB.Yaa mice
Interaction of beta2-glycoprotein I with lipopolysaccharide leads to Toll-like receptor 4 (TLR4 (show TLR4 ELISA Kits))-dependent activation of macrophages
Data are consistent with pathogenic mechanisms in antiphospholipid syndrome involving both neutralization of beta(2)-glycoprotein I (beta(2)GPI) function and beta(2)GPI-immunoglobulin complex formation.
beta-2-glycoprotein 1 plays an important physiologic role in the recognition of dying cells
we have demonstrated a pathogenic role for aPL (show FASL ELISA Kits) containing samples, mediated via aPL (show FASL ELISA Kits)-2bGPI interactions, resulting in activation of the pro-apoptotic p38 MAPK (show MAPK14 ELISA Kits) pathway.
Pretransplant IgA-aB2GP1 was the main risk factor for graft thrombosis and early graft loss after renal transplantation.
These results suggest that antibodies directed against domain 1 of beta-2-glycoprotein 1 may contribute to antiphospholipid syndrome risk assessment, due to their association with the diagnosis of systemic autoimmunity and with more aggressive triple antiphospholipid antibody profiles, such as triple positivity and lupus anticoagulant activity.
Circulating Immune Complexes of IgA Bound to Beta 2 Glycoprotein are Strongly Associated with the Occurrence of Acute Thrombotic Events.
Genome-wide significant results in or near the APOH gene on chromosome 17 have been found for plasma apolipoprotein H levels in middle-aged and older adults.
Prevalence and pathogenicity of IgA aB2GPI antibodies may vary from a population of ESRD patients to another.
The association of IL28B (show IL28B ELISA Kits) and APOH single nucleotide polymorphisms (SNPs) with sustained virological response and of ITPA (show ITPA ELISA Kits) SNPs with anemia related phenotypes.
We identified APOH as an obesity-resistance gene that interacts with FTO (show FTO ELISA Kits) rs9939609 to more than double the occurrence of thinness.
Trp316Ser variant (rs1801690) near the apolipoprotein H (beta2-glycoprotein-I) gene was associated with serum lipid parameters in Chinese volunteers. Suggest that there may be have racial/ethnic-and/or gender-specificity.
Plasma apolipoprotein H limits HCV replication and associates with response to NS3 protease inhibitor-based therapy.
Apolipoprotein H has been implicated in a variety of physiologic pathways including lipoprotein metabolism, coagulation, and the production of antiphospholipid autoantibodies. APOH may be a required cofactor for anionic phospholipid binding by the antiphospholipid autoantibodies found in sera of many patients with lupus and primary antiphospholipid syndrome, but it does not seem to be required for the reactivity of antiphospholipid autoantibodies associated with infections.
, apolipoprotein H (beta-2-glycoprotein I)
, APC inhibitor
, activated protein C-binding protein
, beta-2-glycoprotein 1
, beta-2-glycoprotein I
, anticardiolipin cofactor