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CpG methylation is an epigenetic modification that is important for embryonic development, imprinting, and X-chromosome inactivation. Additionally we are shipping DNMT3B Proteins (5) and DNMT3B Kits (3) and many more products for this protein.
Showing 10 out of 120 products:
Human Monoclonal DNMT3B Primary Antibody for ChIP, FACS - ABIN252478
Santoro, Li, Grummt: The nucleolar remodeling complex NoRC mediates heterochromatin formation and silencing of ribosomal gene transcription. in Nature genetics 2002
Show all 94 references for ABIN252478
Human Polyclonal DNMT3B Primary Antibody for ICC, IF - ABIN151734
Robert, Morin, Beaulieu, Gauthier, Chute, Barsalou, MacLeod: DNMT1 is required to maintain CpG methylation and aberrant gene silencing in human cancer cells. in Nature genetics 2003
Show all 15 references for ABIN151734
Human Polyclonal DNMT3B Primary Antibody for IHC (p), WB - ABIN387884
Lu, Markowetz, Unwin, Leek, Airoldi, MacArthur, Lachmann, Rozov, Maayan, Boyer, Troyanskaya, Whetton, Lemischka: Systems-level dynamic analyses of fate change in murine embryonic stem cells. in Nature 2009
Show all 13 references for ABIN387884
Human Polyclonal DNMT3B Primary Antibody for ChIP, IHC - ABIN152675
Okano, Bell, Haber, Li: DNA methyltransferases Dnmt3a and Dnmt3b are essential for de novo methylation and mammalian development. in Cell 1999
Show all 6 references for ABIN152675
Human Monoclonal DNMT3B Primary Antibody for ChIP, WB - ABIN2668954
Castro, Breiling, Luetkenhaus, Ceteci, Hausmann, Kress, Lyko, Rudel, Rapp: MYC-induced epigenetic activation of GATA4 in lung adenocarcinoma. in Molecular cancer research : MCR 2013
Show all 2 references for ABIN2668954
Human Polyclonal DNMT3B Primary Antibody for IF (p), IHC (p) - ABIN727623
Zhao, Hou, Chen, Shao, Zhu, Bu, Gu, Li, Zhang, Du, Fu, Kong, Luo, Long, Li, Deng, Zhao, Cen: Prenatal cocaine exposure impairs cognitive function of progeny via insulin growth factor II epigenetic regulation. in Neurobiology of disease 2015
Human Polyclonal DNMT3B Primary Antibody for IF, IHC - ABIN1534163
Deloukas, Matthews, Ashurst, Burton, Gilbert, Jones, Stavrides, Almeida, Babbage, Bagguley, Bailey, Barlow, Bates, Beard, Beare, Beasley, Bird, Blakey, Bridgeman, Brown, Buck, Burrill, Butler, Carder et al.: The DNA sequence and comparative analysis of human chromosome 20. ... in Nature 2002
Chicken Polyclonal DNMT3B Primary Antibody for WB - ABIN2783648
Boland, Christman: Characterization of Dnmt3b:thymine-DNA glycosylase interaction and stimulation of thymine glycosylase-mediated repair by DNA methyltransferase(s) and RNA. in Journal of molecular biology 2008
Human Polyclonal DNMT3B Primary Antibody for EIA, IHC (p) - ABIN356569
Stipani, Cappello, Daddabbo, Natuzzi, Miniero, Stipani, Palmieri: The mitochondrial oxoglutarate carrier: cysteine-scanning mutagenesis of transmembrane domain IV and sensitivity of Cys mutants to sulfhydryl reagents. in Biochemistry 2001
dnmt7 specifically methylates no tail gene in the genome
Among 18 genotypes analyzed, we were unable to record any significant differences in 5-methyl-2'-deoxycytidine levels, which suggested that age-related changes in global DNA methylation (show HELLS Antibodies) content are rather a function of time, and not a genetic component.
miR (show MLXIP Antibodies)-29b was negatively correlated with DNMT3A (show DNMT3A Antibodies)/3B expression at the cellular/histological levels.
mRNA and protein expression levels of DNMT3b were upregulated in genotype 1b and 3a HCV-infected hepatocellular carcinoma patients as compared to control. DNMT3b mRNA levels did not change in genotypes 2a, 3, and 4, but were upregulated at the protein level by genotype 1b, 2a, and 3a. No differences were seen for genotypes 5 and 7.
Haplotype analysis reveals a strong association of the interaction of DNMT3B gene with APOEepsilon4 in a sample of Alzheimer disease patients.
Our meta-analysis suggested that DNMT1 (show DNMT1 Antibodies) rs16999593 and DNMT3A (show DNMT3A Antibodies) rs1550117 could contribute to GC and that DNMT3B rs1569686 might function as a protective factor against gastric carcinogenesis.
DNMT3B Gene Polymorphism is associated with Gastric and Colorectal Cancer.
We found that pre-miR (show MLXIP Antibodies)-29c or miR (show MLXIP Antibodies)-29c mimics significantly increases the expression level of miR (show MLXIP Antibodies)-34c and miR (show MLXIP Antibodies)-449a. We further found DNA methyltransferase 3a (show DNMT3A Antibodies) and 3b are the target gene of miR (show MLXIP Antibodies)-29c
Data show that microRNA miR (show MLXIP Antibodies)-221 contributes to breast cancer tumorigenicity by regulating stemness, at least in part through the control of DNA methyltransferase 3B (DNMT3b) expression.
Downregulation of DNMT3B, one of the targets identified using this method, radiosensitizes cancer cells by disturbing multiple DNA damage responses.
The HBVX gene upregulated the mRNA and protein expression levels of DNMT3A (show DNMT3A Antibodies)/3B, downregulated the expression of SOCS1 (show SOCS1 Antibodies) and increased SOCS1 (show SOCS1 Antibodies) gene promoter CpG island methylation.
Our results demonstrate DNMT3B4-del as a critical factor in developing aberrant DNA methylation (show HELLS Antibodies) patterns during lung tumorigenesis and suggest that DNMT3B4-del may be a target for lung cancer prevention.
The epiblast expressed epithelial markers, MUC1 (show MUC1 Antibodies) and E-CADHERIN (show CDH1 Antibodies), and the pluripotency markers, DNMT3B and CRIPTO (show TDGF1 Antibodies).
Developmental changes in expression of DNMT3B are indicative of a possible role in changes in methylation in cattle.
The expression levels of DNMT3a (show DNMT3A Antibodies) and DNMT3b were associated with several beef quality traits.
Loss of DNMT3B results in hypomethylation of the miR (show MLXIP Antibodies)-196b promoter and increased miR (show MLXIP Antibodies)-196b expression, which directly targets the mTORC2 (show CRTC2 Antibodies) component Rictor (show RICTOR Antibodies).
The findings define PRMT7 (show PRMT7 Antibodies) as a regulator of the DNMT3b/p21 (show D4S234E Antibodies) axis required to maintain muscle stem cell regenerative capacity.
These results demonstrate that Dnmt1 (show DNMT1 Antibodies) and Dnmt3b cooperate to maintain DNA methylation (show HELLS Antibodies) and genomic integrity in the intestinal epithelium.
High levels of Dnmt3b expression prolong leukemia latency.
The expression of DNMT1 (show DNMT1 Antibodies) and DNMT3b was decreased at 1 mT, and 50 Hz ELF (show SPTBN1 Antibodies)-EMF can increase the expression...the alterations of genome-wide methylation and DNMTs expression may play an important role in the biological effects of 50 Hz ELF (show SPTBN1 Antibodies)-EMF exposure
DKO cardiomyocytes showed virtual absence of targeted Dnmt3a (show DNMT3A Antibodies) and Dnmt3b mRNA transcripts.
An unexpected oncogenic role for Dnmt3a (show DNMT3A Antibodies) in MTCL through methylation-independent repression of Dnmt3b.
we report that cardiac specific deletion of Dnmt3b, the predominant DNA methyltransferase (show DNMT1 Antibodies) in adult mouse hearts, leads to an accelerated progression to severe systolic insufficiency and myocardial thinning without a preceding hypertrophic response.
These results indicated a role for the miR29b-Dnmt3a (show DNMT3A Antibodies)/3b-DNA methylation (show HELLS Antibodies) axis in mouse early embryonic development, and we provide evidence that miR29b is indispensable for mouse early embryonic development
CpG methylation is an epigenetic modification that is important for embryonic development, imprinting, and X-chromosome inactivation. Studies in mice have demonstrated that DNA methylation is required for mammalian development. This gene encodes a DNA methyltransferase which is thought to function in de novo methylation, rather than maintenance methylation. The protein localizes primarily to the nucleus and its expression is developmentally regulated. Mutations in this gene cause the immunodeficiency-centromeric instability-facial anomalies (ICF) syndrome. Eight alternatively spliced transcript variants have been described. The full length sequences of variants 4 and 5 have not been determined.
DNA (cytosine-5)-methyltransferase 3B
, DNA (cytosine-5-)-methyltransferase 3 beta
, DNA cytosine-5 methyltransferase 3 beta
, DNA (cytosine-5)-methyltransferase 3B-like
, DNA methyl transferase beta
, DNA methyltransferase 3B
, DNA MTase HsaIIIB
, DNA methyltransferase HsaIIIB
, DNA (cytosine-5-)-methyltransferase 3 beta, like
, DNA (cytosine-5-)-methyltransferase 7
, DNA MTase MmuIIIB
, DNA methyltransferase MmuIIIB