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FAAH encodes a protein that is responsible for the hydrolysis of a number of primary and secondary fatty acid amides, including the neuromodulatory compounds anandamide and oleamide. Additionally we are shipping FAAH Proteins (9) and FAAH Kits (7) and many more products for this protein.
Showing 10 out of 83 products:
Human Monoclonal FAAH Primary Antibody for WB - ABIN395692
de Luis, Gonzalez Sagrado, Aller, Izaola, Conde: Effects of C358A missense polymorphism of the endocannabinoid degrading enzyme fatty acid amide hydrolase on weight loss after a hypocaloric diet. in Metabolism: clinical and experimental 2011
Show all 5 references for ABIN395692
Human Monoclonal FAAH Primary Antibody for ELISA, WB - ABIN560832
McFarland, Bardell, Yates, Placzek, Barker: RNA interference-mediated knockdown of dynamin 2 reduces endocannabinoid uptake into neuronal dCAD cells. in Molecular pharmacology 2008
Show all 3 references for ABIN560832
Human Polyclonal FAAH Primary Antibody for ELISA, WB - ABIN1584431
Fu, Bottegoni, Sasso, Bertorelli, Rocchia, Masetti, Guijarro, Lodola, Armirotti, Garau, Bandiera, Reggiani, Mor, Cavalli, Piomelli: A catalytically silent FAAH-1 variant drives anandamide transport in neurons. in Nature neuroscience 2011
Show all 2 references for ABIN1584431
Human Polyclonal FAAH Primary Antibody for ICC, IF - ABIN4309894
Shubbar, Helou, Kovács, Nemes, Hajizadeh, Enerbäck, Einbeigi: High levels of γ-glutamyl hydrolase (GGH) are associated with poor prognosis and unfavorable clinical outcomes in invasive breast cancer. in BMC cancer 2013
genetic deletion of FAAH may predispose animals to increased sensitivity to certain types of pain.
Impaired neurogenesis by HIV-1-Gp120 (show ITIH4 Antibodies) is rescued by genetic deletion of fatty acid amide hydrolase enzyme
Study identified FAAH as a novel player in the pathogenesis of lupus
Basal concentrations of anandamide was greater, and the severity of cystitis was reduced FAAH KO mice. Cystitis-associated increased peripheral sensitivity and enhanced bladder activity were attenuated in FAAH KO mice.
in FAAH(-/-) animals the number of microglia and the ratio of activated microglia and IL-1beta (show IL1B Antibodies) level were already higher in young animals
Results demonstrate that the supra-spinally-located FAAH enzyme is necessary for the analgesic action of paracetamol.
Data suggest that multitarget FAAH/Cox (show CPOX Antibodies) blockade may provide a transformative approach to inflammatory bowel disease (IBD) and other pathologies in which fatty acid amide hydrolase/cyclooxygenases (FAAH, Cox-1 (show PTGS1 Antibodies), and Cox-2 (show COX2 Antibodies)) are overactive.
our results do not support a clear role of FAAH, CNR1 (show CNR1 Antibodies) and NAPE-PLD (show NAPEPLD Antibodies) in BD and lithium response.
FAAH inhibitors provide a new class of anti-spastic agents that may have utility in treating spasticity in multiple sclerosis.
FAAH is required for chronic stress to induce hyperactivity and structural amygdala remodeling. FAAH-mediated decreases in arachidonylethanolamine signaling after chronic stress. This loss is functionally relevant to the effects of chronic stress.
fatty acid amide hydrolase inhibition exerts cutaneous anti-inflammatory effects
In transgenic mice, a direct involvement of the human FAAH C385A SNP was associated with alcohol "binge" drinking.
Results suggest that carriers of FAAH A allele are at increased risk of Myocardial Infarction.
FAAH gene variation was shown to associate with cold pain sensitivity with P129T/rs324420 being the most likely causal variant as it is known to reduce the FAAH enzyme activity.
Data suggest that subjects who are carriers of minor allele (A) at missense mutation rs324420 in FAAH benefit from increased consumption of oleic and docosahexaenoic acids in dietary treatment of abdominal obesity.
Specific FAAH polymorphisms are associated with refractory postoperative nausea and vomiting , opioid-related respiratory depression, and prolonged PACU stay due to opioid adverse effects in white children undergoing tonsillectomy.
the FAAH 385C>A polymorphism may have a role in the endocannabinoid-related obesity phenotype
Cannabis-using FAAH C/C carriers and A carrying controls have reduced white matter integrity compared to control C/C carriers.
Studied the mechanism of FAAH for lipid selection to specifically hydrolyze anandamide, examining the role of the dynamic paddle formed at the boundaries of the FAAH catalytic site.
FAAH genetic variation enhances fronto-amygdala function regarding fear regulation in mouse and human.
AtFAAH is one, but not the only, modulator of endogenous N-Acylethanolamines (NAE) levels in plants, and that NAE depletion likely participates in the regulation of plant growth [fatty acid amide hydrolase] [AtFAAH]
Overexpression of AtFAAH inhibits innate immunity against Pseudomonas syringae in Arabidopsis, and plants had lower amounts of jasmonic acid, abscisic acid, and salicylic acid.
AtFAAH influences plant growth and interacts with ABA signaling and plant defense through distinctly different mechanisms
This gene encodes a protein that is responsible for the hydrolysis of a number of primary and secondary fatty acid amides, including the neuromodulatory compounds anandamide and oleamide.
fatty-acid amide hydrolase 1
, fatty acid amide hydrolase
, hypothetical protein LOC569067
, fatty-acid amide hydrolase
, fatty-acid amide hydrolase 1-like
, fatty acid amide hydrolase, gene 3
, oleamide hydrolase 1
, fatty acid alpha-hydroxylase
, fatty acid hydroxylase domain containing 1
, anandamide amidohydrolase 1
, amide hydrolase
, fatty acid amide hydrolase protein