Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
In vertebrates, the genes encoding the class of transcription factors called homeobox genes are found in clusters named A, B, C, and D on four separate chromosomes. Additionally we are shipping HOXA9 Proteins (9) and many more products for this protein.
Showing 10 out of 112 products:
Human Polyclonal HOXA9 Primary Antibody for ICC, IF - ABIN4319699
Molino, Jabès, Bonnet, Gaudin, Bernard, Benech, Khrestchatisky: Gene expression comparison reveals distinct basal expression of HOX members and differential TNF-induced response between brain- and spinal cord-derived microvascular endothelial cells. in Journal of neuroinflammation 2016
Show all 2 Pubmed References
Mouse (Murine) Polyclonal HOXA9 Primary Antibody for ELISA, WB - ABIN4319694
Hu, Fong, Ferrell, Largman, Shen: HOXA9 modulates its oncogenic partner Meis1 to influence normal hematopoiesis. in Molecular and cellular biology 2009
Human Monoclonal HOXA9 Primary Antibody for IF, ELISA - ABIN561318
Storlie, Jackson, Hutchinson, Grose: Delayed biosynthesis of varicella-zoster virus glycoprotein C: upregulation by hexamethylene bisacetamide and retinoic acid treatment of infected cells. in Journal of virology 2006
Cow (Bovine) Polyclonal HOXA9 Primary Antibody for WB - ABIN2778526
Whelan, Ludwig, Bertrand: HoxA9 induces insulin-like growth factor-1 receptor expression in B-lineage acute lymphoblastic leukemia. in Leukemia 2008
Show all 2 Pubmed References
Consistent with these findings, a previously developed DHODH inhibitor, brequinar (BRQ), also induced differentiation of HOXA9-dependent and -independent cells and human AML (show RUNX1 Antibodies) cell lines. BRQ was well tolerated in mice, and in xenograft models of AML (show RUNX1 Antibodies), BRQ treatment slowed tumor growth, extended survival, and promoted cellular differentiation.
This is the first time the protein partners of either E2A (show TCF3 Antibodies)-PBX1 (show PBX1 Antibodies) or HOXA9 oncoproteins were identified using an unbiased biochemical approach. The identification of translation initiation factors associated with HOXA9 might indicate a novel function for HOX (show MSH2 Antibodies) proteins independent of their transcriptional activity.
The HOXA9 moiety of NUP98 (show NUP98 Antibodies)-HOXA9 is essential for binding to the Hoxa gene locus. MLL (show MLL Antibodies) is important for the recruitment of NUP98 (show NUP98 Antibodies)-HOXA9 to the HOXA locus and for NUP98 (show NUP98 Antibodies)-HOXA9-induced HOXA gene expression.
In the present study, we found in a translational stepwise approach that promoter DNA methylation (show HELLS Antibodies) of the homeobox (show Lbx1 Antibodies) (HOX (show MSH2 Antibodies)) gene HOXA9 could help predict resistance or response to cisplatin-based chemotherapy in patients with bladder cancer
DYNLT1 (show DYNLT1 Antibodies) interacts with nucleoporins and plays a role in the dysregulation of gene expression and induction of hematopoietic cell proliferation by the leukemogenic nucleoporin (show AGFG2 Antibodies) fusion, NUP98 (show NUP98 Antibodies)-HOXA9
Low HOXA9 expression is associated with Non Small Cell Lung Cancer.
This study identified HOXA9 as a target gene of miR (show MLXIP Antibodies)-196b and determined that the mechanism of miR (show MLXIP Antibodies)-196b-mediated epithelial-to-mesenchymal transition and invasion processes involves the regulation of HOXA9 expression in non-small cell lung cancer cells.
Advanced glycation end products could induce endothelial cell dysfunction through NF-kappaB (show NFKB1 Antibodies) dependent HoxA9 downregulation.
HOXA9 role in acute myeloid leukemia (show BCL11A Antibodies).
NUP98 (show NUP98 Antibodies)-HOXA9 ability to induce blood cell expansion is evolutionarily conserved.
The analysis points to a critical role for Hoxa9 and PU.1 in distal regulation of c-myb (show MYB Antibodies) expression in murine myeloid cells during iL-6 (show IL6 Antibodies)-induced cell differentiation.
the Hoxa9- and Meis1 (show MEIS1 Antibodies)-associated upregulation of Flt3 (show FLT3 Antibodies) is a passive event with regard to leukemia development in mice and with limited relevance to the AML (show RUNX1 Antibodies) pathology.
the cohesin complex regulates PRC2 targeting to silence Hoxa7 and Hoxa9 and negatively regulate self-renewal. Our studies identify a novel epigenetic mechanism underlying leukemogenesis in AML patients with cohesin mutations.
data delineate an altered epigenetic stress response in activated satellite cells from aged mice, which limits satellite cell function and muscle regeneration by Hoxa9-dependent activation of developmental pathways
Pbx3 contributes to Hoxa9 leukemogenesis through stabilization of the Meis1 (show MEIS1 Antibodies) protein.
IGF-1 (show IGF1 Antibodies) is a direct HOXA9 target important for hematopoietic transformation.
deficiency of tumor suppressor prep1 accelerates the onset of meis1 (show MEIS1 Antibodies)- hoxa9 leukemogenesis
Hoxa9 collaborates with E2A (show TCF3 Antibodies)-PBX1 (show PBX1 Antibodies) in mouse B cell leukemia in association with Flt3 (show FLT3 Antibodies) activation and decrease of B cell gene expression.
results suggest a previously unidentified role for C/EBPalpha (show CEBPA Antibodies) in maintaining the proliferation required for Hoxa9/Meis1 (show MEIS1 Antibodies)-mediated leukemogenesis
This is the first description of a molecular link between HoxA9 and the regulation of Bcl-2 (show BCL2 Antibodies) family members in acute myeloid leukemia (show BCL11A Antibodies).
In vertebrates, the genes encoding the class of transcription factors called homeobox genes are found in clusters named A, B, C, and D on four separate chromosomes. Expression of these proteins is spatially and temporally regulated during embryonic development. This gene is part of the A cluster on chromosome 7 and encodes a DNA-binding transcription factor which may regulate gene expression, morphogenesis, and differentiation. This gene is highly similar to the abdominal-B (Abd-B) gene of Drosophila. A specific translocation event which causes a fusion between this gene and the NUP98 gene has been associated with myeloid leukemogenesis. Read-through transcription exists between this gene and the upstream homeobox A10 (HOXA10) gene.
, homeobox protein Hox-A9
, XIHbox 6
, homeobox protein Hox-1G
, homeodomain protein HOXA9
, homeo box A9
, homeobox protein Hox-1.7
, Homeobox gene A7
, homeobox protein Hox-1.1
, homeobox protein Hox-A7
, homeobox protein R5
, homeobox protein A9