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IFI6 was first identified as one of the many genes induced by interferon. Additionally we are shipping IFI6 Antibodies (47) and IFI6 Kits (4) and many more products for this protein.
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In conclusion, over-expression of IFI6 promotes hepatitis C virus RNA replication and rescues the interferon alpha (show IFNA Proteins)-mediated anti-hepatitis C virus activity.
IFI6 inhibits HCV entry by impairing EGFR (show EGFR Proteins) mediated CD81 (show CD81 Proteins)/CLDN1 (show CLDN1 Proteins) interactions. This may be relevant to other virus entry processes employing EGFR (show EGFR Proteins).
Here we elucidate G1P3, a survival protein induced by interferons (IFNs), as a target of estrogen signaling and a contributor to poor outcomes in estrogen receptor (show ESR1 Proteins)-positive (ER(+)) breast cancer.
PRINS regulates G1P3, a gene with anti-apoptotic effects in keratinocytes. siRNA-mediated inhibition of PRINS gene resulted in altered cell morphology and gene expression alterations.
G1P3 protein may have function as a cell survival protein by inhibiting mitochondrial-mediated apoptosis
Respiratory syncytial virus upregulated the mRNA expression of chemokines CC and CXC and interfered with type alpha/beta interferon (show IFNA Proteins)-inducible gene expression by upregulation of MG11 (show SAMHD1 Proteins) and downregulation of G1P3.
IFN-alpha2b (show ADRA2B Proteins) induced the expression of G1P3 and antagonized the TRAIL induced apoptosis in myeloma suggesting that either the deregulated or the induced expression of G1P3 could lead to apoptosis resistance in tumor cells.
This gene was first identified as one of the many genes induced by interferon. The encoded protein may play a critical role in the regulation of apoptosis. A minisatellite that consists of 26 repeats of a 12 nucleotide repeating element resembling the mammalian splice donor consensus sequence begins near the end of the second exon. Alternatively spliced transcript variants that encode different isoforms by using the two downstream repeat units as splice donor sites have been described.
interferon alpha-inducible protein 6
, interferon, alpha-inducible protein clone IFI-6-16
, interferon-induced protein 6-16