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Natural killer (NK) cells are lymphocytes that can mediate lysis of certain tumor cells and virus-infected cells without previous activation. Additionally we are shipping KLRG1 Proteins (9) and many more products for this protein.
Showing 10 out of 125 products:
Human Monoclonal KLRG1 Primary Antibody for FACS - ABIN2658355
Huntington, Tabarias, Fairfax, Brady, Hayakawa, Degli-Esposti, Smyth, Tarlinton, Nutt: NK cell maturation and peripheral homeostasis is associated with KLRG1 up-regulation. in Journal of immunology (Baltimore, Md. : 1950) 2007
Show all 8 references for ABIN2658355
Human Monoclonal KLRG1 Primary Antibody for ICFC - ABIN2661956
Gründemann, Bauer, Schweier, von Oppen, Lässing, Saudan, Becker, Karp, Hanke, Bachmann, Pircher: Cutting edge: identification of E-cadherin as a ligand for the murine killer cell lectin-like receptor G1. in Journal of immunology (Baltimore, Md. : 1950) 2006
Show all 6 references for ABIN2661956
Human Monoclonal KLRG1 Primary Antibody for FACS - ABIN2659381
Blaser, Kaufmann, Pircher: Virus-activated CD8 T cells and lymphokine-activated NK cells express the mast cell function-associated antigen, an inhibitory C-type lectin. in Journal of immunology (Baltimore, Md. : 1950) 1999
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Human Monoclonal KLRG1 Primary Antibody for FACS - ABIN2660217
Robbins, Nguyen, Takahashi, Mikayama, Biron, Brossay: Cutting edge: inhibitory functions of the killer cell lectin-like receptor G1 molecule during the activation of mouse NK cells. in Journal of immunology (Baltimore, Md. : 1950) 2002
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Human Monoclonal KLRG1 Primary Antibody for FACS - ABIN2662779
McMahon, Zajac, Jamieson, Corral, Hammer, Ahmed, Raulet: Viral and bacterial infections induce expression of multiple NK cell receptors in responding CD8(+) T cells. in Journal of immunology (Baltimore, Md. : 1950) 2002
Show all 6 references for ABIN2662779
Human Monoclonal KLRG1 Primary Antibody for FACS - ABIN2660863
Robbins, Terrizzi, Sydora, Mikayama, Brossay: Differential regulation of killer cell lectin-like receptor G1 expression on T cells. in Journal of immunology (Baltimore, Md. : 1950) 2003
Show all 6 references for ABIN2660863
Mouse (Murine) Monoclonal KLRG1 Primary Antibody for FACS - ABIN371102
Corral, Hanke, Vance, Cado, Raulet: NK cell expression of the killer cell lectin-like receptor G1 (KLRG1), the mouse homolog of MAFA, is modulated by MHC class I molecules. in European journal of immunology 2000
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Human Polyclonal KLRG1 Primary Antibody for ELISA, WB - ABIN564422
Yoon, Lim, Park, Lee: Cell-to-cell contact with hepatitis C virus-infected cells reduces functional capacity of natural killer cells. in Journal of virology 2011
This finding provides a rationale for the reciprocal expression of KLRG1 and CD103 (show ITGAE Antibodies) in different CD8 (show CD8A Antibodies)(+) T-cell subsets.
Cytotoxic KLRG1-expressed CD8 (show CD8A Antibodies) effector cells and defective T regulatory cells cause murine autoimmune cholangitis.
KLRG1(+) iNKT cells coexpressing CD49d and granzyme A (show GZMA Antibodies) persisted for several months and displayed a potent secondary response to cognate antigen.
high levels of TfRs such as those found on activated lymphocytes were found to be associated with decreased KLRG1 inhibitory function, indicating that TfRs may sequester KLRG1 from interacting with cadherins.
data further demonstrate that the inhibitory activity of KLRG1 is decreased in T cells expressing high levels of TfR (show TFRC Antibodies), indicating that association of KLRG1 with TfR (show TFRC Antibodies) hinders KLRG1-mediated silencing.
Data indicate that CD103 (show ITGAE Antibodies)(+)CD8 (show CD8A Antibodies)(+) T(RM) cells developed in the skin from epithelium-infiltrating precursor cells that lacked expression of the effector-cell marker KLRG1.
Data indicate that increments of CD8 (show CD8A Antibodies) + effector memory T cells in human and mouse chronic lymphocytic leukemia (CLL)(Emu-TCL1 (show TCL1A Antibodies) model) were due to an expansion of the inhibitory killer cell lectin-like receptor (show Klra6 Antibodies) G1 (KLRG1) expressing cellular subset.
Despite their differences, KLRG1+ and KLRG1- Treg cells proved similarly potent in suppressing experimental autoimmune encephalomyelitis.
KLRG1(-/-) mice had a significant survival extension after Mycobacterium tuberculosis infection compared to wild-type controls, and maintained a significantly lower level of pulmonary bacterial load throughout chronic infection.
CD8 (show CD8A Antibodies)+ T cells lacking Id2 do not generate a robust terminally differentiated killer cell lectin-like receptor (show Klra6 Antibodies) G1 (KLRG1hi) effector population, but display a cell-surface phenotype and cytokine profile consistent with memory precursors.
More T cells were KLRG1+ in the synovial fluid of patients with spondylarthritis/rheumatoid arthritis than crystal arthritis or controls. Those T cells were more functionally active, and migrated towards the synovial fluid of SpA (show FASL Antibodies)/RA patients.
KLRG1 was overexpressed on CD4 (show CD4 Antibodies)(+) T cells.
These results indicate that KLRG1 negatively regulates natural killer cell numbers and functions via the Akt (show AKT1 Antibodies) pathway, thus providing a novel marker and therapeutic target for hepatitis C virus infection.
cytomegalovirus positive had lower proportions of NK-cells expressing inhibitory receptors (KLRG1 and CD158a (show KIR2DL1 Antibodies))
trastuzumab-mediated antibody-dependent cellular cytotoxicity was markedly enhanced by KLRG1-negative peripheral blood mononuclear cells
Results demonstrated a substantial reduction in both CD94 (show KLRD1 Antibodies)- and KLRG1- expressing CD3 (show CD3 Antibodies)(-)CD56 (show NCAM1 Antibodies)(+) NK cells with increased age.
Expression of MafA analyzed in conditional knockout mice during embryonic development is the first specific marker of a subpopulation of "early c-ret (show RET Antibodies)" positive sensory neurons.
Natural killer (NK) cells are lymphocytes that can mediate lysis of certain tumor cells and virus-infected cells without previous activation. They can also regulate specific humoral and cell-mediated immunity. The protein encoded by this gene belongs to the killer cell lectin-like receptor (KLR) family, which is a group of transmembrane proteins preferentially expressed in NK cells. Studies in mice suggested that the expression of this gene may be regulated by MHC class I molecules. Alternatively spliced transcript variants have been reported, but their full-length natures have not yet been determined.
killer cell lectin-like receptor subfamily G, member 1
, killer cell lectin-like receptor subfamily G member 1
, mast cell function-associated antigen 2F1
, C-type lectin domain family 15 member A
, C-type lectin domain family 15, member A
, ITIM-containing receptor MAFA-L
, MAFA-like receptor
, mast cell function-associated antigen (ITIM-containing)
, mast cell function-associated antigen