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MYOD1 encodes a nuclear protein that belongs to the basic helix-loop-helix family of transcription factors and the myogenic factors subfamily. Additionally we are shipping Myogenic Differentiation 1 Kits (23) and Myogenic Differentiation 1 Proteins (10) and many more products for this protein.
Showing 10 out of 169 products:
Chicken Monoclonal MYOD1 Primary Antibody for EIA, IHC (fro) - ABIN272324
Thulasi, Dias, Houghton, Houghton: Alpha 2a-interferon-induced differentiation of human alveolar rhabdomyosarcoma cells: correlation with down-regulation of the insulin-like growth factor type I receptor. in Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research 1997
Show all 7 references for ABIN272324
Chicken Monoclonal MYOD1 Primary Antibody for IHC (fro), WB - ABIN967403
Davis, Weintraub, Lassar: Expression of a single transfected cDNA converts fibroblasts to myoblasts. in Cell 1988
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Human Polyclonal MYOD1 Primary Antibody for IF, WB - ABIN953579
Xynos, Corbella, Belmonte, Zini, Manfredini, Ferrari: Bone marrow-derived hematopoietic cells undergo myogenic differentiation following a Pax-7 independent pathway. in Stem cells (Dayton, Ohio) 2010
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Human Monoclonal MYOD1 Primary Antibody for ELISA, WB - ABIN969305
Mal: Histone methyltransferase Suv39h1 represses MyoD-stimulated myogenic differentiation. in The EMBO journal 2006
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Human Polyclonal MYOD1 Primary Antibody for WB - ABIN196752
Reynaud, Leibovitch, Tintignac, Pelpel, Guillier, Leibovitch: Stabilization of MyoD by direct binding to p57(Kip2). in The Journal of biological chemistry 2000
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Human Polyclonal MYOD1 Primary Antibody for WB - ABIN197309
Tintignac, Sirri, Leibovitch, Lécluse, Castedo, Metivier, Kroemer, Leibovitch: Mutant MyoD lacking Cdc2 phosphorylation sites delays M-phase entry. in Molecular and cellular biology 2004
Show all 3 references for ABIN197309
Human Polyclonal MYOD1 Primary Antibody for IF (cc), IF (p) - ABIN740340
Gong, Zhao, Yang, Li, Chen, Chen, Zhou: The control of mesenchymal stem cell differentiation using dynamically tunable surface microgrooves. in Advanced healthcare materials 2014
Human Polyclonal MYOD1 Primary Antibody for WB - ABIN2780432
Delgado-Olguín, Hernández-Hernández, Salamanca, Recillas-Targa, Coral-Vázquez: Identification of two E-boxes that negatively modulate the activity of MyoD on the alpha-sarcoglycan core promoter. in Biochimica et biophysica acta 2008
SRF and its cofactor MYOCD (show MYOCD Antibodies) likely contribute to the hypertrophy of peripheral airway smooth muscle observed in equine asthmatic airways, while the remodeling of the central airways is more static or involves different transcription factors.
Equine primary fibroblasts were transformed by lentiviral transduction of equine myogenic differentiation 1 into fusion-competent myoblasts.
these results suggest that sarcoma metastasis can be partially controlled through Pax7 (show PAX7 Antibodies)/MyoD-dependent activation of miR (show MLXIP Antibodies)-182 and provide insight into the role that myogenic transcription factors have in sarcoma progression
These observations demonstrated the first time that Wnt3a (show WNT3A Antibodies) can directly activate MyoD expression through targeting cis (show CISH Antibodies)-elements in the DE and the L fragment.
Studies indicate that MyoD occupies multiple promoters that induce the transcription of genes vital for establishing the myogenic fate and is also implicated as a mediator of many chromatin modifying enzymes for their recruitment to myogenic enhancers.
MUNC is not a classic cis (show CISH Antibodies)-acting enhancer RNA (e-RNA) acting exclusively by stimulating the neighboring MyoD gene
MyoD acts to promote SC proliferation and transition of cells into differentiation, while myogenin (show MYOG Antibodies) is known to drive terminal differentiation
A recurring point mutation in MYOD1 is found in 10% of embryonal rhabdomyosarcomas with distinct clinical features and poor prognosis.
Spindle cell and sclerosing rhabdomyosarcoma show recurrent MYOD1 mutations, in keeping with a single pathologic entity, regardless of age at presentation.
MyoD was required for the induction of FGF21 (show FGF21 Antibodies) gene transcription by mitochondrial dysfunction.
analysis of a mutation in MYOD1 that may have a role in progression of embryonal rhabdomyosarcoma and may be associated with mutations altering PI3K (show PIK3CA Antibodies)-AKT (show AKT1 Antibodies) pathway components
MYOD1 homozygous mutations are frequent, recurrent and pathognomonic events in adult-type spindle cell Rhabdomyosarcoma
established a model in which FHL3 (show FHL3 Antibodies) promotes the expression of MyHC 2a (show MYH2 Antibodies) through CREB (show CREB1 Antibodies)-mediated transcription and inhibits the expression of MyHC (show MYH13 Antibodies) 1/slow by inhibiting MyoD transcription activity during myogenesis
Transcription of the skeletal muscle program is achieved by the expression of MyoD, which binds to the same sites as Myf5 (show MYF5 Antibodies), indicating that each factor regulates distinct steps in gene initiation and transcription at a shared set of binding sites
The large TBP (show TBP Antibodies) polyQ repeat decreases the association of MyoD with TBP (show TBP Antibodies) and DNA promoters and causes muscle degeneration in spinocerebellar ataxia (show USP14 Antibodies) 17 transgenic mice.
results reveal a role for Six4 (show SIX4 Antibodies) during adult muscle regeneration and suggest a widespread mechanism of cooperation between Six4 (show SIX4 Antibodies) and MyoD.
redirecting MyoD binding from MyoD private sites to NeuroD2 (show NEUROD2 Antibodies) private sites
Jmjd2C (show KDM4C Antibodies) increases MyoD transcriptional activity to facilitate skeletal muscle differentiation by increasing MyoD stability through inhibiting G9a (show EHMT2 Antibodies)-dependent MyoD degradation.
MyoD affects chromatin looping at CCCTC-binding factor (show CTCF Antibodies)-binding sites represents the first evidence that a differentiation factor regulates chromatin-loop dynamics
MyoD differentially promotes activated and repressed chromatin structures at myogenic genes early after the onset of skeletal muscle differentiation in the developing mouse embryo.
Duxbl overexpression in C2C12 cells inhibited myogenic differentiation by decreasing MyoD downstream gene expressions, including M-cadherin, MyoG (show MYOG Antibodies), p21 (show D4S234E Antibodies) and cyclin D3 (show CCND3 Antibodies) but not MyoD itself.
KAP1 (show TRIM28 Antibodies) as a previously unappreciated interpreter of cell signaling, which modulates the ability of MyoD to drive myogenesis.
Of the eight adult pig tissue types that were tested, the expression of Myf5 (show MYF5 Antibodies) and MyoD1 was highest in the muscle tissue.
Single nucleotide polymorphisms in the MYOD1 and GDF8 (show MSTN Antibodies) genes are associated with genetic transcription during myogenesis in pigs.
Therefore, this study demonstrated that the different regulatory adipogenic roles of MSTN (show MSTN Antibodies) in ADSCs and MSCs act by differentially regulating PPARgamma (show PPARG Antibodies) and MyoD expression.
Therefore, the g.489C>T and g.1264C>A SNPs in MYOD1 may be meaningful DNA markers that can be used for improving important porcine economic traits.
The total expression profile of MyoD and Pax7 (show PAX7 Antibodies) genes suggests that higher muscularity in Pietrain pigs is associated with the presence of a greater number of active satellite stem cells compared to other breeds.
Exons and promoters are amplified and sequenced in the 5'UTR (show UTS2R Antibodies) region of this gene.
Relative MYOD1 expression was not different, but MYOG (show MYOG Antibodies) expression was higher in the (ligated-tube)crowded group embryos.
MYOD1 intron 1 DdeI polymorphism was not significantly associated with any meat quality traits tested
Bos taurus MYF5 (show MYF5 Antibodies) activates MYF5 (show MYF5 Antibodies) and MYOD1 expression in cultured fibroblasts.
results suggest that MyoD and Myf5 (show MYF5 Antibodies) influence the MyHC (show MYH13 Antibodies) isoform expression, although the effects are not decisive in specifying the phenotypes of adult muscles
n conclusion, hypoxia stimulates the proliferation of satellite cells and promotes their myogenic differentiation with MyoD playing an important role
Irxl1/Mkx (show MKX Antibodies) can repress myoD expression through direct binding to its promoter and may thus play a negative regulatory role in muscle differentiation.
Myod in turn up-regulates cdkn1c (show CDKN1C Antibodies), thereby providing a positive feedback loop that switches myogenic cells to terminal differentiation
Myf5 (show MYF5 Antibodies) and Myod function independently during cranial myogenesis.
This gene encodes a nuclear protein that belongs to the basic helix-loop-helix family of transcription factors and the myogenic factors subfamily. It regulates muscle cell differentiation by inducing cell cycle arrest, a prerequisite for myogenic initiation. The protein is also involved in muscle regeneration. It activates its own transcription which may stabilize commitment to myogenesis.
myogenic factor 3
, myogenic differentiation 1
, myoblast determination protein 1
, MYOD protein
, myogenic factor MyoD1
, MYOD1 homolog
, myoblast determination protein 1 homolog
, myogenic factor 1
, class C basic helix-loop-helix protein 1
, myogenic regulatory factor
, myogenic differenciation 1, transcription activator
, myogenic differenciation 1
, myoblast determination 1