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transcriptional repressor and an immediate-early gene [RGD, Feb 2006].. Additionally we are shipping NAB1 Antibodies (41) and NAB1 Proteins (6) and many more products for this protein.
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These data suggest that type I IFN stimulation induces a rapid recruitment of a repressive Egr3 (show EGR3 ELISA Kits)/Nab1 complex that silences transcription from the ifngr1 (show IFNGR1 ELISA Kits) promoter.
Nab1 is a potent inhibitor of cardiac growth in response to pathological stimuli in vivo.
Nab1 and Nab2 proteins are necessary for Schwann cells to exit the cell cycle, downregulate suppressed cAMP-inducible protein (SCIP) expression and upregulate expression of critical myelination genes.
Egr2 (show EGR2 ELISA Kits) directly represses genes during myelination in conjunction with NAB corepressors
Data show that the suppressive effect of valproic acid on chondrocytes is not due to reduced expression or recruitment of Egr-1 (show EGR1 ELISA Kits) to the mPGES-1 (show PTGES ELISA Kits) promoter and involves upregulation of NAB1.
Methylation pattern of the corresponding NGFI-A (show EGR1 ELISA Kits) binding site in the human glucocorticoid receptor (show NR3C1 ELISA Kits) exon 1-F specific promoter in post mortem hippocampal tissue.
NAB1 associates with RNA transcripts through a mechanism involving its CSD (show CSAD ELISA Kits) that is conserved with mechanisms of sequence-specific nucleic acid recognition employed by ancestrally related bacterial cold-shock proteins (CSPs).
All examined cysteine single and double mutants exhibited a reduced antenna at photosystem II caused by a perturbed NAB1 deactivation mechanism.
transcriptional repressor and an immediate-early gene
NGFI-A binding protein 1 (EGR1 binding protein 1)
, NGFI-A-binding protein 1-like
, nucleic acid binding protein
, EGR-1-binding protein 1
, NGFI-A-binding protein 1
, EGR1 binding protein 1
, transcriptional regulatory protein p54
, NGF1A-binding protein
, transcriptional repressor TA16