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ARHGAPs, such as ARHGAP24, encode negative regulators of Rho GTPases (see ARHA\; MIM 165390), which are implicated in actin remodeling, cell polarity, and cell migration (Katoh and Katoh, 2004 [PubMed 15254788]).[supplied by OMIM, Mar 2008].. Additionally we are shipping ARHGAP24 Proteins (4) and many more products for this protein.
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Human Polyclonal ARHGAP24 Primary Antibody for WB - ABIN657747
Horinouchi, Yagi, Imanishi, Mori, Yanai, Hayakawa, Takeshima, Hijioka, Okamura, Sakaeda, Matsuo, Okumura, Nakamura: Association of genetic polymorphisms with hepatotoxicity in patients with childhood acute lymphoblastic leukemia or lymphoma. in Pediatric hematology and oncology 2010
Show all 4 references for ABIN657747
Src (show SRC Antibodies) family tyrosine kinase (show TXK Antibodies) signaling may regulate FilGAP through association with RBM10 (show RBM10 Antibodies)
FilGAP may contribute to change in cell motility of B-lymphocytes and in addition, its expression appears to be useful for predicting the behavior of B-cell lymphoma, in particular follicular lymphoma.
study suggests that Arf6 (show ARF6 Antibodies) and phosphorylation of FilGAP may regulate FilGAP, and phosphorylation of Ser (show SIGLEC1 Antibodies)-402 may play a role in the regulation of cell spreading on fibronectin (show FN1 Antibodies)
Polymorphism rs346473 in the ARHGAP24 gene might be a part of the genetic variants.
FilGAP may function as a mediator of the regulation of Rac (show AKT1 Antibodies) by Arf6 (show ARF6 Antibodies).
Data indicate that phosphorylation of FilGAP by ROCK appears to promote amoeboid morphology.
Consistent with structural predictions, strain increases beta-integrin binding to FLNA (show FLNA Antibodies), whereas it causes FilGAP to dissociate from FLNA (show FLNA Antibodies), providing a direct and specific molecular basis for cellular mechanotransduction
sequencing of the ARHGAP24 gene in patients with focal segmental glomerulosclerosis (FSGS (show ACTN4 Antibodies)) identified a mutation that impaired its Rac1-GAP activity and was associated with disease in a family with FSGS (show ACTN4 Antibodies)
p73 (show TP73 Antibodies), a vascular cell-specific GTPase-activating protein (show RASA1 Antibodies), is an important modulator of angiogenesis
propose that RC-GAP72 affects cellular morphology by targeting activated Cdc42 and Rac1 GTPases to specific subcellular sites, triggering local morphological changes
Data show that the Mdm4 (show MDM4 Antibodies)-p73 axis cannot override the dominant role of p53 (show TP53 Antibodies) in development and tumorigenesis and that Mdm4 (show MDM4 Antibodies) and p73 interaction during development and tumorigenesis suggests new insight into the role of p53 (show TP53 Antibodies) family members.
In vivo inhibition of both p63 (show CKAP4 Antibodies) and p73 in combination accelerates tumor regression and increases survival of p53 (show TP53 Antibodies)-deficient mice.
Results indicate that p73 regulates basal and starvation-induced fuel metabolism in the liver, a finding that is likely to be highly relevant for metabolism-associated disorders, such as diabetes and cancer.
MDM2 (show MDM2 Antibodies) mediates p73 ubiquitination
p73 is required for endothelial cell differentiation, migration and the formation of vascular networks regulating VEGF (show VEGFA Antibodies) and TGFbeta (show TGFB1 Antibodies) signaling
Although mouse TIGAR (show C12orf5 Antibodies) expression is clearly induced in the intestines of mice following DNA-damaging stress of ionizing radiation, that was not dependent on p53 (show TP53 Antibodies) or TAp73 (show TP73 Antibodies).
Ribosomal proteins L11 (show RPL11 Antibodies) and L5 activate TAp73 (show TP73 Antibodies) by overcoming MDM2 (show MDM2 Antibodies) inhibition.
the p73 status has to be considered when studying the regulatory role of p53 (show TP53 Antibodies) protein in gliomagenesis [review]
Suggest that TAp73 (show TP73 Antibodies) promotes anabolism to counteract cellular senescence rather than to support proliferation.
p63 (show CKAP4 Antibodies) antagonizes p53 (show TP53 Antibodies) to promote cellular survival, whereas p73 regulates self-renewal and p53 (show TP53 Antibodies)-mediated apoptosis versus senescence.
ARHGAPs, such as ARHGAP24, encode negative regulators of Rho GTPases (see ARHA\; MIM 165390), which are implicated in actin remodeling, cell polarity, and cell migration (Katoh and Katoh, 2004
RAC1- and CDC42-specific GTPase-activating protein of 72 kDa
, filamin-A-associated RhoGAP
, rho GTPase-activating protein 24
, rho-type GTPase-activating protein 24
, rhoGAP of 73 kDa
, sarcoma antigen NY-SAR-88
, Down-regulated in nephrectomized rat kidney 2
, p53-like transcription factor
, p53-related protein
, tumor protein p73
, transformation related protein 73