Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Show all species
Show all synonyms
Select your species and application
anti-Human PRKACG Antibodies:
anti-Mouse (Murine) PRKACG Antibodies:
anti-Rat (Rattus) PRKACG Antibodies:
Go to our pre-filtered search.
Human Polyclonal PRKACG Primary Antibody for ELISA, WB - ABIN562372
Adachi, Kano, Saido, Murata: Visual screening and analysis for kinase-regulated membrane trafficking pathways that are involved in extensive beta-amyloid secretion. in Genes to cells : devoted to molecular & cellular mechanisms 2009
Human Monoclonal PRKACG Primary Antibody for FACS, IHC - ABIN969365
Louiset, Stratakis, Perraudin, Griffin, Libé, Cabrol, Fève, Young, Groussin, Bertherat, Lefebvre et al.: The paradoxical increase in cortisol secretion induced by dexamethasone in primary pigmented nodular adrenocortical disease involves a glucocorticoid receptor-mediated effect of dexamethasone on ... in The Journal of clinical endocrinology and metabolism 2009
The missense p.74Ile>Met PRKACG mutation is associated with a marked defect in proplatelet formation and a low level in filamin A (show FLNA Antibodies) in megakaryocytes.
Results suggest that PKA can negatively regulate ERalpha (show ESR1 Antibodies), at least in part, through FoxH1 (show FOXH1 Antibodies).
PKA-mediated phosphorylation of GPIbbeta at Ser (show SIGLEC1 Antibodies)(166) negatively regulates VWF (show VWF Antibodies) binding to GPIb-IX and is one of the mechanisms by which PKA mediates platelet inhibition
Results suggest that Cgamma and Calpha differ in structure and function in vitro, supporting the hypothesis that functionally different C-subunit isozymes could affect cAMP signal transduction downstream of protein kinase A activation.
In human primary pigmented nodular adrenocortical disease tissues, dexamethasone paradoxically stimulates cortisol release through a glucocorticoid receptor (show NR3C1 Antibodies)-mediated effect on PKA catalytic subunits.
Findings show that PKIB (show PKIB Antibodies) and PKA-C kinase can have critical functions of aggressive phenotype of PCs through Akt (show AKT1 Antibodies) phosphorylation and that they should be a promising molecular target for PC treatment.
Cyclic AMP-dependent protein kinase (PKA) consists of two catalytic subunits and a regulatory subunit dimer. This gene encodes the gamma form of its catalytic subunit. The gene is intronless and is thought to be a retrotransposon derived from the gene for the alpha form of the PKA catalytic subunit.
, cAMP-dependent protein kinase catalytic subunit gamma
, serine(threonine) protein kinase
, protein kinase, cAMP-dependent, catalytic, gamma