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Solute Carrier Family 35, Member G5 (SLC35G5) (N-Term) antibody
Alternatives Western Blotting (WB), ELISA
|3 references available|
|Quantity||0.1 mg (0.25 mg/ml)|
|Price||280.50 $ Plus shipping costs $45.00|
|Availability||Will be delivered in 2 to 3 Business Days|
|Immunogen||This AMAC1L2 antibody is generated from rabbits immunized with a KLH conjugated synthetic peptide between 1-30 amino acids from the N-terminal region of human AMAC1L2.|
|Description||Other names: Protein AMAC1L2,AMAC1L2,AMAC|
|Characteristics||Peptide Affinity Purified Rabbit Polyclonal Antibody (Pab)|
|Specificity||This AMAC1L2 antibody is generated from rabbits immunized with a KLH conjugated synthetic peptide between 1-30 amino acids from the N-terminal region of human AMAC1L2.|
|Molecular Weight||35161 DA|
Background: This gene seems to be intronless. It has high sequence similarity to the gene encoding acyl-malonyl condensing enzyme on chromosome 17.
|Application Notes||The suggested dilution is: ELISA ~~ 1:1,000 Western blotting~~ 1:100~500|
|Buffer||Purified polyclonal antibody supplied in PBS with 0.09% (W/V) sodium azide. This antibody is purified through a protein A column, followed by peptide affinity purification.|
|Storage||Maintain refrigerated at 2-8 deg C for up to 6 months. For long term storage store at -20 deg C in small aliquots to prevent freeze-thaw cycles|
|Research Area||Cell Structure|
|Restrictions||For Research Use only|
|AMAC1L2 Antibody (N-term) (ABIN654847) western blot analysis in WiDr cell line lysates (35ug/lane). This demonstrates the AMAC1L2 antibody detected the AMAC1L2 protein (arrow).|
Appel, Filter, Reis et al.: "Physical and transcriptional map of the critical region for keratolytic winter erythema (KWE) on chromosome 8p22-p23 between D8S550 and D8S1759." in: European journal of human genetics : EJHG, Vol. 10, Issue 1, pp. 17-25, 2002 (PubMed).
Xing, Wang, Belancio et al.: "Emergence of primate genes by retrotransposon-mediated sequence transduction." in: Proceedings of the National Academy of Sciences of the United States of America, Vol. 103, Issue 47, pp. 17608-13, 2006 (PubMed).
Kathiresan, Willer, Peloso et al.: "Common variants at 30 loci contribute to polygenic dyslipidemia." in: Nature genetics, Vol. 41, Issue 1, pp. 56-65, 2008 (PubMed).