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These data identify suppression of CXCL2 (show CXCL2 ELISA Kits) and CXCL5 (show CXCL5 ELISA Kits) chemoattractant expression by 11beta-HSD1 (show HSD11B1 ELISA Kits) as a novel mechanism with potential for regulation of neutrophil recruitment to the injured myocardium, and cardiac fibroblasts as a key site for intracellular glucocorticoid regeneration during acute inflammation following myocardial injury.
IL-17RA (show IL17RA ELISA Kits) regulates CXL-1 and 5 production in the lungs during the adaptive response.
STAT3 (show STAT3 ELISA Kits) is required for maximal OSM (show OSM ELISA Kits)-induced CXCL5 (show CXCL5 ELISA Kits) expression.
CXCL5 (show CXCL5 ELISA Kits) has a role in neutrophil recruitment in TH17-mediated glomerulonephritis
Since adaptive villus growth occurs despite impaired CXCL5 (show CXCL5 ELISA Kits) expression and enhanced angiogenesis, this suggests that the growth of new blood vessels is not needed for resection-induced mucosal surface area expansion following massive SBR (show NXF1 ELISA Kits).
CXCL5 (show CXCL5 ELISA Kits) regulates pulmonary responses to infection and plays a central role in conferring clock control of inflammation.
findings demonstrated that CXCL1 (show CXCL1 ELISA Kits) and CXCL5 (show CXCL5 ELISA Kits) are increased in circulation with onset of T2D, are produced by islets under stress, and synergistically affect islet function, suggesting that these chemokines participate in pathogenesis of T2D.
TLR2 (show TLR2 ELISA Kits)-induced epithelial-derived CXCL5 (show CXCL5 ELISA Kits) is critical for polymorphonuclear leukocyte-driven destructive inflammation in pulmonary tuberculosis.
Our data suggest that the differential regulation of the chemokine CXCL5 between osteoblasts and endothelial cells upon FGF2 treatment is involved in Hematopoietic stem cell mobilization from the osteoblast niche or bone marrow to peripheral blood.
CXCL5 (show CXCL5 ELISA Kits) modulated macrophage activation, increased expression of the cholesterol efflux regulatory protein ABCA1 (show ABCA1 ELISA Kits), and enhanced cholesterol efflux activity in macrophages.
CXCL6 level is high in the serum of chronic hepatitis B patients.CXCL6 promotes human hepatocyte proliferation through the CXCR1-NFkappaB pathway and inhibits collagen I secretion by hepatic stellate cells.
The role of PITX2 in glaucoma may be mediated partly by regulating the expression of CXCL6 and BBS5 and thus affecting immune functions and intraocular pressure.
This is the first study to note that elevated systemic CCL5 (show CCL5 ELISA Kits) and CXCL6 were associated with moderate/severe lumbar disc degeneration. These chemokines may be systemic biomarkers for the diagnosis and monitoring of disc degeneration.
The neutrophil-recruiting chemokine (show CCL1 ELISA Kits) GCP-2/CXCL6 is expressed in cystic fibrosis (show S100A8 ELISA Kits) airways and retains its functional properties after binding to extracellular DNA.
HIF-1alpha (show HIF1A ELISA Kits) promotes HCC (show FAM126A ELISA Kits) progression and metastasis by upregulating CXCL6 transcription in HCC (show FAM126A ELISA Kits) cells.
Expression of the CXCL8 (show IL8 ELISA Kits), CXCL6 and CXCL1 (show CXCL1 ELISA Kits) genes are under the primary control of 1,25-dihydroxyvitamin D3 and its receptor.
Data suggest that expression of CXCL6 in trophoblasts is up-regulated during pregnancy development/placentation; CXCL6 expression inhibits trophoblast cell migration and invasion by down-regulating activity of MMP2 (matrix metalloproteinase 2 (show MMP2 ELISA Kits)).
Overexpression of GCP-2 (show FOLH1 ELISA Kits) in mesenchymal stem cells has the potential to enhance their angiogenic and survival properties.
Studies show that functional blocking of GCP-2 (show FOLH1 ELISA Kits) inhibits tumor growth and metastases.
The aim of this study was to investigate the role of the transcription factors, AP-1 (show FOSB ELISA Kits) and NF-kappaB (show NFKB1 ELISA Kits), in IL-6 (show IL6 ELISA Kits) and CXCL8 (show IL8 ELISA Kits) regulation in Jurkat T-cells.
mouse homolog is a member of the CXC chemokine family, is a neutrophil chemoattractant and is rapidly induced in response to muscle injury
C-X-C motif chemokine 5
, CXC chemokine LIX
, chemokine (C-X-C motif) ligand 6 (granulocyte chemotactic protein 2)
, cytokine LIX
, small-inducible cytokine B5
, granulocyte chemotactic protein-2
, small inducible cytokine B subfamily, member 5
, small inducible cytokine subfamily B, member 15
, C-X-C motif chemokine 6
, Small inducible cytokine subfamily B (Cys-X-Cys), member b
, chemokine alpha 3
, granulocyte chemotactic protein 2
, small inducible cytokine subfamily B (Cys-X-Cys), member 6 (granulocyte chemotactic protein 2)
, small-inducible cytokine B6
, chemokine (C-X-C motif) ligand 5
, inducible cytokine subfamily B (Cys-X-Cys), member 6