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these data suggest that microRNA-143 suppresses IL-13 (show IL13 ELISA Kits) activity and inflammation through targeting of IL-13Ralpha1 in epidermal keratinocytes
required for induction of the alternative macrophage activation pathway by IL-13 (show IL13 ELISA Kits) but not by IL-4 (show IL4 ELISA Kits)
analysis of IL-4 (show IL4 ELISA Kits) and IL-13 (show IL13 ELISA Kits) receptors in cancer biology and discussion of pre-clinical and clinical studies pertaining to recombinant immunotoxins designed to target these receptors [review]
review of IL-4 (show IL4 ELISA Kits) and IL-13 (show IL13 ELISA Kits) receptor structure, receptor regulation, signaling and experimental therapeutics [review]
Results identified FAM120A (show FAM120A ELISA Kits) in the IL13 (show IL13 ELISA Kits)/IL13Ralpha2 signaling pathway as a key mediator of invasion and liver metastasis in colon cancer.
Data show that high expression of interleukin-4 receptor (show IL4R ELISA Kits) IL-4Ralpha correlated with increased recurrence, while interleukin-13 (show IL13 ELISA Kits) receptor IL-13Ralpha1 had an inverse relationship to recurrence and survival in oral cavity squamous cell carcinoma patients.
miR (show MLXIP ELISA Kits)-143 regulation of IL-13 (show IL13 ELISA Kits)-induced inflammatory cytokine and mucus production in nasal epithelial cells from allergic rhinitis patients probably partly depends on inhibition of IL13Ralpha1.
MiR (show MLXIP ELISA Kits)-143 may be associated with allergic reaction in human mast cells via downregulation of IL13RA1.
Expression of IL-4 (show IL4 ELISA Kits), IL-4R, and IL-13R (show IL13RA2 ELISA Kits) are involved in the process of local metastases in colorectal cancer, while IL-13 (show IL13 ELISA Kits) expression has an impact on survival.
In Korean asthmatic children, increased serum IgE level was a multi-locus interaction between IL-4Ralpha, IL-13 (show IL13 ELISA Kits), IL-13Ralpha1, CD14 (show NDUFA2 ELISA Kits), and CTLA4 (show CTLA4 ELISA Kits) polymorphisms. The combination of CTLA4 (show CTLA4 ELISA Kits)/IL-13 (show IL13 ELISA Kits) and IL-13 (show IL13 ELISA Kits)/IL-13Ralpha1 polymorphisms showed strong synergistic effects.
These results show that activation of IL-13Ralpha1 is critical for key aspects of the immune and functional responses to Hb2 infection that facilitate expulsion.
These data establish for the first time a molecular mechanism by which Mac-1 (show ITGAM ELISA Kits) regulates the signaling activity of IL-13 (show IL13 ELISA Kits) in macrophages.
CAR T cells killed IL13Ra1- and/or IL13Ra2 (show IL13RA2 ELISA Kits)-positive cells in contrast to IL13Ra1- and IL13Ra2 (show IL13RA2 ELISA Kits)-negative controls
in neonatal mice, IL-12 (show IL12A ELISA Kits) uses IL-12Rbeta2 to counter IL-13Ralpha1 expression in addition to promoting Th1 (show HAND1 ELISA Kits) differentiation
The extracellular and transmembrane domains of the gammaC and interleukin (IL)-13 (show IL13 ELISA Kits) receptor alpha1 chains, not their cytoplasmic domains, dictate the nature of signaling responses to IL-4 (show IL4 ELISA Kits) and IL-13 (show IL13 ELISA Kits)
IL-13Ralpha1 is the key receptor mediating airway hypersensitivity responses, mucus production, and transforming growth factor (TGF)-beta (show TGFB1 ELISA Kits) induction in response to aeroallergens.
a central role for the IL-13 (show IL13 ELISA Kits)/IL-13Ralpha1 pathway in the regulation of intestinal epithelial cell Cl(-) secretion via up-regulation of cystic fibrosis transmembrane conductance regulator (show CFTR ELISA Kits)
IL-13 (show IL13 ELISA Kits) and IL-4 (show IL4 ELISA Kits) via IL-13 (show IL13 ELISA Kits) receptor alpha1 and the type II IL-4 (show IL4 ELISA Kits) receptor have direct roles in asthma pathogenesis
The protein encoded by this gene is a subunit of the interleukin 13 receptor. This subunit forms a receptor complex with IL4 receptor alpha, a subunit shared by IL13 and IL4 receptors. This subunit serves as a primary IL13-binding subunit of the IL13 receptor, and may also be a component of IL4 receptors. This protein has been shown to bind tyrosine kinase TYK2, and thus may mediate the signaling processes that lead to the activation of JAK1, STAT3 and STAT6 induced by IL13 and IL4.
, IL-13 receptor subunit alpha-1
, IL-13R subunit alpha-1
, IL13 receptor alpha-1 chain
, bB128O4.2.1 (interleukin 13 receptor, alpha 1)
, cancer/testis antigen 19
, interleukin-13 receptor subunit alpha-1
, interleukin-13-binding protein
, novel cytokine receptor 4
, interleukin 13 receptor alpha 1
, interleukin 13 receptor alpha chain 1
, interleukin 13 receptor, alpha 1
, interleukin-13 receptor subunit alpha-1-like