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anti-Human CCL24 Antibodies:
anti-Mouse (Murine) CCL24 Antibodies:
anti-Rat (Rattus) CCL24 Antibodies:
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Human Polyclonal CCL24 Primary Antibody for IF (p), IHC (p) - ABIN740925
Kanai, Park, Yoshida, Tsunoda, Yoshie: IL-35 Suppresses Lipopolysaccharide-Induced Airway Eosinophilia in EBI3-Deficient Mice. in Journal of immunology (Baltimore, Md. : 1950) 2016
Human Polyclonal CCL24 Primary Antibody for Func, ELISA - ABIN2473460
Patel, Kreider, Li, Li, Leung, Salcedo, Nardelli, Pippalla, Gentz, Thotakura, Parmelee, Gentz, Garotta: Molecular and functional characterization of two novel human C-C chemokines as inhibitors of two distinct classes of myeloid progenitors. in The Journal of experimental medicine 1997
Show all 2 Pubmed References
Mean eotaxin 2 concentrations in nasal fluid in patients with perennial allergic rhinitis and nonallergic and allergic chronic rhinosinusitis with nasal polyps patients were significantly higher in comparison to control subjects.
Review: eotaxins (CCL11 (show CCL11 Antibodies), CCL24, and CCL26 (show CCL26 Antibodies)) play key role(s) during symptomatic inflammatory responses raised in response to allergic crisis of allergic asthma and atopic dermatitis
HMGB1 (show HMGB1 Antibodies) did not elicit chemotaxis of human eosinophils alone and had no effect in combination with the eosinophil chemotactic agent, eotaxin-2 (CCL24).
these data suggest that CCL26 (show CCL26 Antibodies) and CCL24 are likely involved in the pathogenesis of chronic nasal hypereosinophilia, with a complex cooperation and different involvement of the various members of eotaxin (show CCL11 Antibodies) family.
CCL11 (show CCL11 Antibodies), CCL24, and CCL26 (show CCL26 Antibodies) are increased in TB patients; hence, it seems that TB suppresses Th1 (show TH1L Antibodies) and the classic function of macrophages subsequently by inducing the chemokines' expression
CCL11 (show CCL11 Antibodies), CCL24, and CCL26 (show CCL26 Antibodies) has a role in the recruitment of extravillous trophoblast into decidual tissue and vessels.
Pregnancy associated environments increased local CCL24/CCR3 (show CCR3 Antibodies), supporting the process of decidualization in human early pregnancy.
Phosphodiesterase 4 inhibitors, rolipram and RO-20-1724 have no effect on CCL24 expression in human primary bronchial epithelial cells.
The level of eotaxin (show CCL11 Antibodies) expression and inflammatory cell count were measured in the material from nasal brushing in healthy controls and in patients with allergic rhinitis, asthma, and chronic obstructive pulmonary disease.
Study shows that CCL24 levels were significantly increased in age-related macular degeneration (AMD (show AMD1 Antibodies)) patients despite Avastin treatment as compared with normal controls and those without Avastin.
the increased airway eosinophils and mucous cell metaplasia in house dust mite-challenged Cd163 (show CD163 Antibodies)-/- mice are mediated by augmented CCL24 production
TPL-2 (show MAP3K8 Antibodies)-regulated Ccl24 in CD11c (show ITGAX Antibodies)+CD11b (show ITGAM Antibodies)+ cells prevents accelerated type-2 mediated immunity to H. polygyrus.
this study shows that TPL-2 (show MAP3K8 Antibodies) inhibits Ccl24 expression in lung dendritic cells, and blockade of Ccl24 prevents the exaggerated airway eosinophilia and lung inflammation in mice given house dust mites-pulsed Map3k8 (show MAP3K8 Antibodies)-/- dendritic cells
production in M-CSF (show CSF1R Antibodies)-induced bone marrow-derived macrophages is synergistically induced by IL-4 (show IL4 Antibodies) and IL-10 (show IL10 Antibodies)
Autologous transfer of peritoneal macrophages in to the airways of asthmatic mice reduces eotaxin (show CCL11 Antibodies) production.
Data indicate that eotaxin 2 (CCL-24), which is known to influence eosinophil migration, was highly up-regulated in broncho-alveolar macrophages (BAMs).
The oesophageal production of CCL24 upon IL-13 (show IL13 Antibodies) stimulation is sufficient to promote eosinophil migration.
Results demonstrate a cooperative mechanism between interleukin-13 (show IL13 Antibodies) and eotaxin-2, where IL-13 (show IL13 Antibodies) mediates allergen-induced eotaxin-2 expression, and eotaxin-2 mediates IL-13 (show IL13 Antibodies)-induced airway eosinophilia.
Distinct acidic and basic residues within CCR3 (show CCR3 Antibodies) determine both receptor expression and activation by the eotaxins.
The eotaxin-2 pathway plays a fundamental role in eosinophil recruitment during ovalbumin (show OVA Antibodies)-induced experimental asthma.
This gene belongs to the subfamily of small cytokine CC genes. Cytokines are a family of secreted proteins involved in immunoregulatory and inflammatory processes. The CC cytokines are proteins characterized by two adjacent cysteines. The cytokine encoded by this gene displays chemotactic activity on resting T lymphocytes, a minimal activity on neutrophils, and is negative on monocytes and activated T lymphocytes. The protein is also a strong suppressor of colony formation by a multipotential hematopoietic progenitor cell line.
C-C motif chemokine 24
, eosinophil chemotactic protein 2
, myeloid progenitor inhibitory factor 2
, small inducible cytokine subfamily A (Cys-Cys), member 24
, small-inducible cytokine A24
, CC chemokine CCL24
, small inducible cytokine A24
, eotaxin-2-like protein
, eosinophil chemotactic protein-2
, small chemokine ligand 24