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anti-Human Aquaporin 4 Antibodies:
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Human Polyclonal Aquaporin 4 Primary Antibody for IF (p), IHC (p) - ABIN671181
Duan, Hao, Fan, Wang, Liu, Hao, Xu, Liu, Zhang: The role of neuropeptide Y and aquaporin 4 in the pathogenesis of intestinal dysfunction caused by traumatic brain injury. in The Journal of surgical research 2013
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Human Monoclonal Aquaporin 4 Primary Antibody for IHC (fro), IHC (p) - ABIN2477485
Endert, Ritter, Schumann: [Isotope methods in the radiological diagnosis of venous disease. II. Indications and accuracy (author's transl)]. in RöFo : Fortschritte auf dem Gebiete der Röntgenstrahlen und der Nuklearmedizin 1980
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Human Polyclonal Aquaporin 4 Primary Antibody for ICC, IF - ABIN4281307
Lopez-Rodriguez, Acaz-Fonseca, Viveros, Garcia-Segura: Changes in cannabinoid receptors, aquaporin 4 and vimentin expression after traumatic brain injury in adolescent male mice. Association with edema and neurological deficit. in PLoS ONE 2015
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Human Monoclonal Aquaporin 4 Primary Antibody for EIA, IHC (fro) - ABIN119571
Esposito, Imitola, Lu, De Filippis, Scuderi, Ganesh, Folkerth, Hecht, Shin, Iuvone, Chesnut, Steardo, Sheen: Genomic and functional profiling of human Down syndrome neural progenitors implicates S100B and aquaporin 4 in cell injury. in Human molecular genetics 2008
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Human Monoclonal Aquaporin 4 Primary Antibody for EIA, IHC (fro) - ABIN119570
Bódis, Nagy, Németh, Mózsik: Active water selective channels in the stomach: investigation of aquaporins after ethanol and capsaicin treatment in rats. in Journal of physiology, Paris 2001
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Ordered disorder of the astrocytic dystrophin (show DMD Antibodies)-associated protein complex in the norm and pathology.
the AQP4 could play a role in the regulation of water balance and ion transport in the sensory cells of zebrafish, bringing new data for the utilizing of this experimental model in the biology of sensory system.
These results suggest that AQP4 is damaged first and decrease of EAAT-2 (show SLC1A2 Antibodies) may follow in pathogenesis of cortical degeneration. This is the first demonstration of decrease of AQP4 and its association with EAAT-2 (show SLC1A2 Antibodies) decrease in AIDS brain, suggesting a role in the pathogenesis of HIV-associated neurocognitive disorders.
Comparative molecular dynamics study of neuromyelitis optica-immunoglobulin G binding to AQP4 extracellular domains has been presented.
Osmotic water permeabilities of aquaporins AQP4, AQP5 (show AQP5 Antibodies), and GlpF from near-equilibrium simulations have been presented.
A clear correlation between AQP4 expression and ADCmax values in grade I meningioma was identified.
Peritumoral brain edema in patients with meningiomas may depend on AQP4 expression grades and not on tumor grade, tumor volume, Ki-67 (show MKI67 Antibodies) expression, and cell count.
AQP4 antibodies were higher in neuromyelitis optica Chinese Han patients with circulating auto-antibodies.
Retinal nerve fiber layer may be better preserved in MOG-IgG versus AQP4-IgG optic neuritis.
AQP4-autoantibody serostatus correlated with poor visual outcome in neuromyelitis optica (Meta-analysis)
Data show that aquaporin-4 (AQP4) and myelin-oligodendrocyte glycoprotein (MOG) autoantibodies double positive neuromyelitis optica spectrum disorder (NMOSD) patients had a multiphase disease course with a high annual relapse rate.
In this study, altered AQP4 expression was associated with aging brains.
Results suggest that pain is an important factor in the quality of life experienced by both AQP4-Ab positive and negative transverse myelitis patients and suggest an even greater association than disability
The results of this study that AQP4 and GLT1 do not have a strong physical interaction between them and are, instead, differentially regulated.
Study suggests that astaxanthin may exert neuroprotection following traumatic brain injury by ameliorating AQP4/NKCC1 (show SLC12A2 Antibodies)-mediated cerebral edema, and NKCC1 (show SLC12A2 Antibodies) inactivation inhibitions the upregulation of AQP4 after traumatic brain injury.
this study identified and fine mapped the major T-cell epitope of AQP4 and observed that in a transfer model, AQP4-specific T cells alone induced an encephalomyelitic syndrome, however, only in the additional presence of anti-AQP4 antibodies, central nervous system lesions are reminiscent of neuromyelitis optica
Study found that aquaporin-4 deficiency facilitated fear memory extinction and NMDA receptors-dependent long-term depression in the CA3 (show CA3 Antibodies)-CA1 (show CA1 Antibodies) hippocampal pathway.
Study demonstrated evidence of beta-dystroglycan cleavage by matrix metalloproteinase-2 (show MMP2 Antibodies)/-9 in permanent middle cerebral artery occlusion mouse brains; this cleavage was implicated in aquaporin-4 redistribution and brain edema in cerebral ischemia.
This study demonstrated that AQP4 dorsal hippocampal protein expression was significantly downregulated at 1day post epilepsy and was followed by a gradual return to baseline levels with a significant increase in ipsilateral protein levels by 30days post epilepsy.
The major findings in this study were (1) a higher AQP4 immunogold labeling density over parenchymal astrocytic membranes in human subjects than in mice, and (2) a lower degree of AQP4 immunogold polarization to perivascular astrocytic endfoot membranes in humans compared with mice. In the human individuals, endfoot AQP4 polarization did not differ between capillary and arteriolar vessel segments.
TRPV4 (show TRPV4 Antibodies)-AQP4 interactions 'turbocharge' astroglial sensitivity to small osmotic gradients
Elevation of AQP4 and selective cytokines in experimental autoimmune encephalitis mice provides some potential biomarkers in optic neuritis and demyelinating diseases.
This study provided the AQP4 modulates astrocyte-to-microglia communication in neuroinflammation.
AQP4 plays an important role in mediating brain edema in hypoxic-ischemic encephalopathy.
constitutive recycling of AQP2 (show AQP2 Antibodies) does not require phosphorylation at any C-terminal sites
In conclusion, HPO (show GFER Antibodies) can decrease AQP4 expression in brain tissue of rabbits with cerebral hemorrhage to suppress the progression of brain edema and promote repairing of injured tissue.
AQP4 may play an important role in brain edema after severe scald (show RDH11 Antibodies).
In the guinea pig AQP4 is localised to enteric glial cells.
This gene encodes a member of the aquaporin family of intrinsic membrane proteins that function as water-selective channels in the plasma membranes of many cells. The encoded protein is the predominant aquaporin found in brain. Two alternatively spliced transcript variants encoding distinct isoforms have been found for this gene.
, aquaporin 4
, aquaporin type4
, mercurial-insensitive water channel
, aquaporin 4.M23
, mercurial insensitive water channel
, water channel