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Nonvoltage-gated, amiloride-sensitive, sodium channels control fluid and electrolyte transport across epithelia in many organs. Additionally we are shipping Sodium Channel, Nonvoltage-Gated 1, gamma Proteins (7) and Sodium Channel, Nonvoltage-Gated 1, gamma Kits (5) and many more products for this protein.
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Human Polyclonal SCNN1G Primary Antibody for ELISA - ABIN449556
Boyd, Náray-Fejes-Tóth: Steroid-mediated regulation of the epithelial sodium channel subunits in mammary epithelial cells. in Endocrinology 2007
Plasmin (show PLG Antibodies) in nephrotic urine activates the epithelial sodium channel
The derlin-1 (show DERL1 Antibodies) pathway therefore may represent a significant early checkpoint in the recognition and degradation of ENaC (show SCNN1A Antibodies) in mammalian cells.
hENaC incorporating the Liddle-mutated beta-subunit lacks one or more PKC phosphorylation sites, thereby significantly reducing the inhibitory effect of PKC on Na(+) channel activity, whereas hENaC incorporating Liddle-mutated gamma-subunits remains as susceptible to PKC as wild-type hENaC.
deltabetagamma-ENaC (show SCNN1A Antibodies) is inhibited by CFTR (show CFTR Antibodies) but activated by cyclic AMP (show APRT Antibodies).
One nonsynonymous amino acid variants in SCNN1G in nonwhite Cystic fibrosis (show S100A8 Antibodies) patients with non-diagnostic CFTR (show CFTR Antibodies) genotypes was identified.
Both vascular smooth muscle cell and bronchial gamma-ENaC downregulation may explain pulmonary fluid disturbances and participate in pulmonary edema pathogenesis.
The transcriptional expression of alpha, beta, and gamma subunits of ENaC (show SCNN1A Antibodies) was elevated in nasal polyp compared to nasal mucosa
Suggest that SCNN1G gene has important roles in blood pressure regulation in the Han Chinese population.
SCNN1A and SCNN1G SNPs may contribute to blood pressure changes over time in the Han Chinese population.
Data indicate that urokinase-type plasminogen activator (uPA (show PLAU Antibodies)) activates epithelial sodium channel, gamma subunit (ENaC gamma) through catalytic activity-dependent proteolytic modification.
High serum leptin (show LEP Antibodies) may reduce endometrial receptivity by activating the STAT3 (show STAT3 Antibodies) signal pathway and down-regulating gamma-ENaC expression in the endometrium.
ENaC (show SCNN1A Antibodies) and ROMK (show KCNJ1 Antibodies) channel activity in kidney tubules are inhibited in TgWnk4(pseudoaldosteronism type II) mice. Wnk4 (show WNK4 Antibodies)(PHAII)-induced inhibition of ENaC (show SCNN1A Antibodies) and ROMK (show KCNJ1 Antibodies) may contribute to the suppression of K(+) secretion in the tubules.
Mouse cells conditionally overexpressing gamma ENAC had enhanced apical Na(+) entry increased the basolateral cell surface expression of Na,K-ATPase (show ATP1A1 Antibodies) by inhibiting p38 (show CRK Antibodies) kinase-mediated endocytosis of Na,K-ATPase (show ATP1A1 Antibodies).
Data indicate that insulin receptor (InsR (show INSR Antibodies)) is important for epithelial sodium channel (ENaC (show SCNN1A Antibodies)) activity.
These data suggest that adenylyl cyclase VI mediates vasopressin (show AVP Antibodies)-stimulated ENaC (show SCNN1A Antibodies) activity in the kidney.
Cys (show DNAJC5 Antibodies) substitutions at sites in the wrist domains of the beta and gamma subunits alter the Na+ self-inhibition response.
ENaC (show SCNN1A Antibodies) is not a passive passenger in regulated epithelial vesicle trafficking, but plays a role in establishing and maintaining the pool of vesicles that respond to cAMP stimulation.
The reactive intermediates formed during and after exposure to chlorine activated ERK1/2 (show MAPK1/3 Antibodies) in alveolar epithelial cells, leading to decreased ENaC (show SCNN1A Antibodies) concentrations and activity.
TMPRSS4 (show TMPRSS4 Antibodies) primarily activates ENaC (show SCNN1A Antibodies) by cleaving basic residues within the tract gammaK173-K186 distal to the furin (show FURIN Antibodies) cleavage site, thereby releasing a previously defined key inhibitory tract encompassing gammaR158-F168 from the gamma-subunit.
estrogen regulates expression and function of epithelial sodium channel gamma in osteoblasts
Nonvoltage-gated, amiloride-sensitive, sodium channels control fluid and electrolyte transport across epithelia in many organs. These channels are heteromeric complexes consisting of 3 subunits: alpha, beta, and gamma. This gene encodes the gamma subunit, and mutations in this gene have been associated with Liddle syndrome.
sodium channel, nonvoltage-gated 1, gamma
, amiloride-sensitive sodium channel subunit gamma
, epithelium sodium channel gamma subunit
, amiloride sensitive epithelial sodium channel gamma subunit
, ENaC gamma subunit
, amiloride-sensitive epithelial sodium channel gamma subunit
, amiloride-sensitive sodium channel gamma-subunit
, epithelial Na(+) channel subunit gamma
, nonvoltage-gated sodium channel 1 subunit gamma
, amiloride-sensitive sodium channel gamma subunit
, epithelial sodium channel ENaC gamma subunit
, epithelial sodium channel, gamma subunit
, ENaC gamma
, Sodium channel nonvoltage-gated 1 gamma (epithelial)
, Sodium channel, nonvoltage-gated 1, gamma (epithelial)
, amiloride sensitive sodium channel gamma1 subunit
, epithelial sodium channel gamma subunit
, sodium channel, nonvoltage-gated, type I, gamma