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CXCL9 antibody

This Armenian Hamster Monoclonal antibody specifically detects CXCL9 in FACS. It exhibits reactivity toward Mouse.
Catalog No. ABIN2664906

Quick Overview for CXCL9 antibody (ABIN2664906)

Target

See all CXCL9 Antibodies
CXCL9 (gamma-Interferon-Induced Monokine (CXCL9))

Reactivity

  • 78
  • 23
  • 20
  • 16
  • 3
  • 2
  • 1
Mouse

Host

  • 59
  • 26
  • 2
  • 1
  • 1
Armenian Hamster

Clonality

  • 64
  • 25
Monoclonal

Conjugate

  • 43
  • 11
  • 8
  • 4
  • 4
  • 2
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
This CXCL9 antibody is un-conjugated

Application

  • 68
  • 37
  • 29
  • 15
  • 13
  • 13
  • 11
  • 9
  • 7
  • 5
  • 3
  • 3
  • 3
  • 3
  • 2
  • 2
  • 1
Flow Cytometry (FACS)

Clone

MIG-2F5-5
  • Purification

    The antibody was purified by affinity chromatography.

    Isotype

    IgG, IgG kappa
  • Application Notes

    Optimal working dilution should be determined by the investigator.

    Restrictions

    For Research Use only
  • Concentration

    0.5 mg/mL

    Buffer

    Phosphate-buffered solution, pH 7.2, containing 0.09 % sodium azide.

    Preservative

    Sodium azide

    Precaution of Use

    This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

    Storage

    4 °C

    Storage Comment

    The antibody solution should be stored undiluted between 2°C and 8°C.
  • Target

    CXCL9 (gamma-Interferon-Induced Monokine (CXCL9))

    Alternative Name

    CXCL9

    Background

    MIG, also known as mig-1, CXCL9, is a member of the alpha subfamily of inflammatory chemokine. It is inducible in macrophages, hepatocytes, and endothelial cells by IFN-γ, but not by TNF-α or bacterial lipopolysacchrides (LPS). Mig functions as a chemotactic factor for resting memory and activated T cells, both CD4+ and CD8+, and natural killer cells. Furthermore, it was reported that Mig induced both calcium signals and chemotaxis in activated B cells and that B cell activation induced expression of mouse CXCR3. MIG and CXCR3 may be important not only to recruit T cells to peripheral inflammatory sites, but also in some cases to maximize interactions among activated T cells, B cells, and dendritic cells within lymphoid organs to provide optimal humoral responses to pathogens.
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