×
For best experience we recommend to activate Javascript in your browser.
ZMYND10 antibody
ZMYND10
Reactivity: Human, Mouse, Rat
ELISA, IHC
Host: Rabbit
Polyclonal
unconjugated
Product Details anti-ZMYND10 Antibody
(hide)
Target
See all ZMYND10 Antibodies
ZMYND10
(Zinc Finger, MYND-Type Containing 10 (ZMYND10))
Reactivity
Human, Mouse, Rat
Host
All hosts for ZMYND10 antibodies
Rabbit
Clonality
All clonalities for ZMYND10 antibodies
Polyclonal
Conjugate
All conjugates for ZMYND10 antibodies
This ZMYND10 antibody is un-conjugated
Application
All applications for ZMYND10 antibodies
ELISA, Immunohistochemistry (IHC)
Characteristics
Polyclonal Antibody
Purification
Antigen affinity purification
Immunogen
Fusion protein of human ZMYND10
Isotype
IgG
Top Product
Discover our top product ZMYND10 Primary Antibody
Alternatives
(show)
Application Details
(hide)
Application Notes
IHC 1:30-1:150, ELISA 1:5000-1:10000
Restrictions
For Research Use only
Handling
(hide)
Format
Liquid
Concentration
0.5 mg/mL
Buffer
PBS with 0.05 % Sodium azide and 40 % Glycerol, pH 7.4
Preservative
Sodium azide
Precaution of Use
This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
Storage
-20 °C
Storage Comment
Store at -20°C. Avoid freeze / thaw cycles.
Target Details for ZMYND10
(hide)
Target
ZMYND10
(Zinc Finger, MYND-Type Containing 10 (ZMYND10))
Alternative Name
ZMYND10 (ZMYND10 Products )
Synonyms
MGC134474 antibody, MGC98297 antibody, Blu antibody, BLU antibody, FLU antibody, zinc finger, MYND-type containing 10 antibody, zinc finger MYND-type containing 10 antibody, zinc finger MYND-type containing 10 L homeolog antibody, zinc finger, MYND domain containing 10 antibody, Zmynd10 antibody, ZMYND10 antibody, zmynd10.L antibody
Background
This gene encodes a protein containing a MYND-type zinc finger domain that likely functions in assembly of the dynein motor. Mutations in this gene can cause primary ciliary dyskinesia. This gene is also considered a tumor suppressor gene and is often mutated, deleted, or hypermethylated and silenced in cancer cells. Alternative splicing results in multiple transcript variants.
UniProt
O75800
Recently viewed
(hide)