MFAP1 antibody (AA 83-437)

Catalog No. ABIN7876941

The Rabbit Polyclonal anti-MFAP1 antibody is suitable to detect MFAP1 in samples from Human and Mouse. It has been validated for WB, ELISA, FACS and IF.

Quick Overview for MFAP1 antibody (AA 83-437) (ABIN7876941)

Target: MFAP1 (Microfibrillar Associated Protein 1 (MFAP1))

Reactivity: Human, Mouse

Host: Rabbit

Clonality: Polyclonal

Conjugate: This MFAP1 antibody is un-conjugated

Application: Western Blotting (WB), ELISA, Flow Cytometry (FACS), Immunofluorescence (IF)

Product Details anti-MFAP1 Antibody

Binding Specificity: AA 83-437

Purpose: MFAP1 Antibody / Microfibrillar-associated protein 1

Purification: Antigen affinity purified

Immunogen: An E.coli-derived human recombinant protein (D83-K437) was used as the immunogen for the MFAP1 antibody.

Isotype: IgG

Application Details

Application Notes: Optimal dilution of the MFAP1 antibody should be determined by the researcher.

Restrictions: For Research Use only

Handling

Format: Lyophilized

Buffer: 0.5 mg/mL if reconstituted with 0.2 mL sterile DI water

Storage: 4 °C,-20 °C

Storage Comment: After reconstitution, the MFAP1 antibody can be stored for up to one month at 4oC. For long-term, aliquot and store at -20oC. Avoid repeated freezing and thawing.

Target Details for MFAP1

Target: MFAP1 (Microfibrillar Associated Protein 1 (MFAP1))

Alternative Name: MFAP1

Background: Microfibrillar-associated protein 1 is a protein that in humans is encoded by the MFAP1 gene. Microfibrils are an important component of the extracellular matrix of many tissues and can either associate with or without elastin. Several microfibril associated proteins (MFAPs) have been cloned, including MFAP1, MFAP3 and MFAP4. The MFAP1 and MFAP3 genes are localized near the fibrillin genes FBN1 and FBN2, respectively. Mutations in FBN1 are linked to Marfan syndrome. Mutations in FBN2 have been linked to congenital contractural arachnodactyly. This suggests roles for MFAP1 and MFAP3 in heritable diseases affecting microfibrils. Deletion of MFAP4 was found in 30 of 31 patients with Smith-Magenis syndrome (SMS), a clinically recognizable multiple congenital anomaly/mental retardation syndrome

UniProt: P55081