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anti-Human Hepcidin Antibodies:
anti-Mouse (Murine) Hepcidin Antibodies:
anti-Rat (Rattus) Hepcidin Antibodies:
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Human Polyclonal Hepcidin Primary Antibody for ELISA - ABIN450057
Ward, Roberts, Brookes, Joy, Martin, Ismail, Spychal, Iqbal, Tselepis: Increased hepcidin expression in colorectal carcinogenesis. in World journal of gastroenterology 2008
Human Polyclonal Hepcidin Primary Antibody for WB - ABIN4892228
Bose, Megyesi, Shah, Hiatt, Hall, Karaduta, Swaminathan: Evidence Suggesting a Role of Iron in a Mouse Model of Nephrogenic Systemic Fibrosis. in PLoS ONE 2015
Human Polyclonal Hepcidin Primary Antibody for WB - ABIN2776909
Theurl, Theurl, Seifert, Mair, Nairz, Rumpold, Zoller, Bellmann-Weiler, Niederegger, Talasz, Weiss: Autocrine formation of hepcidin induces iron retention in human monocytes. in Blood 2008
Postexercise supplementation with protein and carbohydrate and vitamins D3 and K2 did not blunt the postexercise hepcidin response in highly trained athletes.
A decrease in serum FGF23 (show FGF23 Antibodies) and hepcidin levels was observed in chronic hemodialysis patients treated with lanthanum carbonate.
These observations suggest correlations between serum hepcidin and progression of chronic HBV infection, and may shed a new light on the development of biomarkers for HBV-related disease surveillance.
Increasing adiposity in the pediatric population is associated with increasing IL-6 (show IL6 Antibodies), which stimulates hepcidin synthesis, leading to functional iron deficiency due to inhibition of iron absorption and mobilization from iron stores.
Suggest that pentoxifylline may be a clinically and biologically meaningful modulator of hepcidin-25 in dialysis of patients with ESA (show FLOT2 Antibodies)-hyporesponsive anaemia.
Among low birth weight infants, gestational age, IL-6 (show IL6 Antibodies), erythropoietin (show EPO Antibodies), and soluble transferrin receptor were associated with Hep25 levels. Therefore, prematurity, inflammation, hypoxia, and erythropoietic activity may be important perinatal factors that affect hepcidin levels.
genetic association studies in cohort of infants in Spain: Data suggest that serum hepcidin levels increase in infants during first year of life and are positively associated with iron status only in infants with wild-type HFE (show HFE Antibodies) gene (not in infants with genetic polymorphisms C282Y, H63D, and S65C). (HFE (show HFE Antibodies), homeostatic iron regulator)
Data suggest that circulating hepcidin levels (a biomarker for iron-deficiency anemia) may be regulated by dietary factors other than iron; here, one-time high-dose vitamin D3 reduces plasma hepcidin levels in adults one week post-dosing, without changes in plasma pro-inflammatory cytokine or ferritin (show FTL Antibodies) concentrations.
Hepcidin can very well be utilized as a potential prognostic marker to follow patients with breast cancer metastatic to bone.
Data suggest that hematologic parameters in children consuming lacto-ovo vegetarian diets are comparable with those of control children, but ferritin (show FTL Antibodies) levels are lower; inclusion of novel serum biomarkers, soluble transferrin receptor and hepcidin, in nutritional assessment can better detect subclinical iron deficiency in children following vegetarian diet. This study was conducted in Poland with children ages 4.5-9 years.
bone marrow transplantation between wild-type and TLR4 (show TLR4 Antibodies) knockout mice revealed that hepatic TLR4 (show TLR4 Antibodies)-dependent hepcidin expression was comparable to macrophage TLR4 (show TLR4 Antibodies)-dependent hepcidin expression induced by LPS (show TLR4 Antibodies)
Hepc decreases in Cyp1b1 (show CYP1B1 Antibodies)-/- and gestational vitamin A deficiency mice resulted in stellate activation and lipogenesis suppression.
data indicate that unlike with many other infections, hepcidin is decreased following M.tb infection, and show that hepcidin ablation does not influence M.tb growth in vivo
Hepcidin expression involves epigenetic regulation by histone deacetylase 3 (show HDAC3 Antibodies).
Serum hepcidin levels were measured by competitive ELISA in wild-type and Inhbb (show INHBB Antibodies)-/- mice at baseline and 4 hours after LPS (show TLR4 Antibodies) challenge. Although Smad1 (show SMAD1 Antibodies)/5/8 signaling is not activated by inflammatory stimuli in the absence of activin B (show Actbeta Antibodies), this has no impact on the induction of hepcidin expression.
Anti-hemojuvelin (show HFE2 Antibodies) antibody corrects anemia caused by inappropriately high hepcidin levels
hepcidin mRNA upregulation depends on M1 macrophage polarization
Our data provide evidence that the interplay of these two hormones could help improve the understanding of the pathogenesis of atherosclerosis and NAFLD (show TSC2 Antibodies).
downregulation of hepcidin by siRNA increased iron uptake in bone and liver, which aggravated unloading-induced bone loss.
These data suggest that, in Hjv (show HFE2 Antibodies)(-/-) females, Bmp6 (show BMP6 Antibodies) can provide a signal adequate to maintain hepcidin to a level sufficient to avoid extrahepatic iron loading.
This study shows that hepcidin knockdown in zebrafish using morpholinos leads to iron overload.
The data also show that the antibacterial activity of hepcidin-2 depends upon the disulfide bridges.
data support an alternative mechanism for hepcidin regulation during zebrafish embryonic development, which is independent of hemojuvelin (show HFE2 Antibodies).
Hepcidin expression is regulated by a transferrin-a (show Tf Antibodies)-dependent pathway in the zebrafish embryo.
this study demonstrates that urine hepcidin-25 concentrations strongly correlate with hepatic hepcidin mRNA abundance, plasma hepcidin-25 levels, iron transferrin (show Tf Antibodies) saturation and non-heme liver iron levels.
Data suugest that hepcidin might had antiinflammatory function and is a candidate regulator of the cross-talk between iron regulation and inflammation.
report the full-length cDNA sequences of porcine hepcidin and liver-expressed antimicrobial peptide-2 (LEAP-2 (show LEAP2 Antibodies))
HEP/FPN axis seems to have a central role in infections, with microorganisms within macrophages or that survive in the bloodstream or other cellular spaces. In addition, HEP may be responsible for iron flux regulation as a molecular bridge for iron trafficking and response to infection.
Hepcidin peptide is up-regulated by iron and bacterial components in the trout liver.
The product encoded by this gene is involved in the maintenance of iron homeostasis, and it is necessary for the regulation of iron storage in macrophages, and for intestinal iron absorption. The preproprotein is post-translationally cleaved into mature peptides of 20, 22 and 25 amino acids, and these active peptides are rich in cysteines, which form intramolecular bonds that stabilize their beta-sheet structures. These peptides exhibit antimicrobial activity. Mutations in this gene cause hemochromatosis type 2B, also known as juvenile hemochromatosis, a disease caused by severe iron overload that results in cardiomyopathy, cirrhosis, and endocrine failure.
, liver-expressed antimicrobial peptide 1
, putative liver tumor regressor
, hepcidin antimicrobial peptide 1
, hepcidin antimicrobial peptide
, antimicrobial peptide
, iron regulatory peptide
, preprohepcidin 1
, antimicrobial peptide hepcidin
, putative hepcidin antibacterial peptide