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anti-Human AGT Antibodies:
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Human Polyclonal AGT Primary Antibody for ICC, IF - ABIN447446
Dimitrijevic, Rissler, Luts, Edvinsson: Reduced expression of angiotensin II and angiotensin receptor type 1 and type 2 in resistance arteries from nasal lesions in granulomatosis with polyangiitis (Wegener's granulomatosis). in Scandinavian journal of rheumatology 2011
Show all 6 Pubmed References
Human Monoclonal AGT Primary Antibody for WB - ABIN1882204
Desong Liu, Fang Lu, Songhui Zhai, Liu Wei, Shi Ma, Xiuying Chen, Liqun Dong, Yannan Guo, Jin Wu, Zheng Wang: Renin-angiotensin system gene polymorphisms in children with Henoch-Schönlein purpura in West China. in Journal of the renin-angiotensin-aldosterone system : JRAAS 2010
Show all 3 Pubmed References
Human Monoclonal AGT Primary Antibody for WB - ABIN1882205
Kieć-Wilk, Olszanecka, Mikołajczyk, Kawecka-Jaszcz et al.: [Role of the M235T (c.704c>T) polymorphism of angiotensynogen gene as well as A724A (c.2171G>A) polymorphism of SERCA2a gene in ethiopathogenesis of left ventricular hypertrophy in essential... in Przegla̧d lekarski 2010
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Human Monoclonal AGT Primary Antibody for ELISA, WB - ABIN968948
Xu, Carretero, Lin, Cavasin, Shesely, Yang, Reudelhuber, Yang: Role of cardiac overexpression of ANG II in the regulation of cardiac function and remodeling postmyocardial infarction. in American journal of physiology. Heart and circulatory physiology 2007
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Human Monoclonal AGT Primary Antibody for ELISA, WB - ABIN559812
Jain, Li, Patil, Kumar: HNF-1alpha plays an important role in IL-6-induced expression of the human angiotensinogen gene. in American journal of physiology. Cell physiology 2007
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Human Polyclonal AGT Primary Antibody for IHC, IHC (p) - ABIN4352882
Bachmann, Burté, Pramana, Conte, Brown, Orimadegun, Ajetunmobi, Afolabi, Akinkunmi, Omokhodion, Akinbami, Shokunbi, Kampf, Pawitan, Uhlén, Sodeinde, Schwenk, Wahlgren, Fernandez-Reyes, Nilsson: Affinity proteomics reveals elevated muscle proteins in plasma of children with cerebral malaria. in PLoS pathogens 2014
Data, including data using network analysis, suggest that angiotensinogen (AGT), mitogen-activated protein kinase-14 (MAPK14 (show MAPK14 Antibodies)), and prothrombin (show F2 Antibodies) (F2) in placental villous tissues are core factors in early embryonic development; these studies involved proteomics and bioinformatics analysis of altered protein expression in placental villous tissue from early recurrent miscarriage patients in comparison to control tissues.
The AGT (show AGXT Antibodies) (M235T) gene polymorphism do not seem to have a significant effect on the development of clinical properties or cardiovascular comordities of acromegalic patients.
Angiotensin II has a role in increasing glomerular permeability by beta-arrestin mediated nephrin (show NPHS1 Antibodies) endocytosis
After donor nephrectomy, increasing uAGT (show DPAGT1 Antibodies) levels can be the result of activation of the intrarenal renin (show REN Antibodies)-angiotensin system affecting the compensatory changes in the remaining kidney.
M235T polymorphism of the AGT (show AGXT Antibodies) gene seems unrelated to the development or the clinical course of endometriosis.
AGT (show AGXT Antibodies) missense polymorphisms are not associated with diabetic nephropathy in our subset of Slovenian type 2 diabetes mellitus patients
Association of AGT (show AGXT Antibodies) single nucleotide polymorphism rs3789678 and gestational hypertension in Chinese population.
The T allele of AGT (show AGXT Antibodies) may play a role in the pathogenesis of preeclampsia in South African Black women.
These data indicate that Ang II-AT2R (show AGTR2 Antibodies) regulates human bone marrow MSC (show MSC Antibodies) migration by signaling through the FAK (show PTK2 Antibodies) and RhoA (show RHOA Antibodies)/Cdc42 (show CDC42 Antibodies) pathways.
Data suggest that up-regulaton of Ang-(1 (show ANGPT1 Antibodies)-7) levels in follicular fluid correlates with increases in number of mature oocytes retrieved upon ovarian stimulation in preparation for in vitro fertilization.
ANG II is up-regulated in serum and heart tissues of mice with EAM and that ANG II significantly drives monocyte/macrophage infiltration through the C-C chemokine receptor 2/5 (CCR2/5) axis.
results established that A20 (show TNFAIP3 Antibodies) is involved in the renoprotective effect by calcitriol via negatively modulating the NF-kappaB (show NFKB1 Antibodies) pathway and necroptotic pathway in AngII-induced renal injury.
NLRP3 (show NLRP3 Antibodies) gene deletion attenuates Ang II-induced NLRP3 (show NLRP3 Antibodies) inflammasome activation, phenotypic transformation from a contractile phenotype to a synthetic phenotype and proliferation in primary mice Vascular Smooth Muscle Cells.
adipocyte-derived Agt (show AGXT Antibodies) has essentially no contribution to the plasma concentration and no impact on blood pressure compared to liver-derived Agt (show AGXT Antibodies).
Lung ischemia-reperfusion injury causes a dysregulation of circulating Ang 2 (show ANGPT2 Antibodies) levels and plasma PREP (show PREP Antibodies) activity, although no direct link between both phenomena could be shown.
Inhibition of TLR4 (show TLR4 Antibodies) ameliorates AngII-impaired cavernosal relaxation, decreases TNF-alpha (show TNF Antibodies) levels, and restores Nitric Oxide bioavailability, demonstrating that TLR4 (show TLR4 Antibodies) partly mediates AngII-induced cavernosal dysfunction.
Our study is the first to show the important role of IL-6 (show IL6 Antibodies) in regulating cardiac pathogenesis via inflammation and apoptosis during AngII-induced hypertension. We also provide a novel link between IL-6 (show IL6 Antibodies)/STAT3 (show STAT3 Antibodies) and EndoG (show ENDOG Antibodies)/MEF2A (show MEF2A Antibodies) pathway that affects cardiac hypertrophy during AngII stimulation.
this study demonstrated that Ang II could increase TRPC6 (show TRPC6 Antibodies) induced Ca(2 (show CA2 Antibodies)+) influx and enhance autophagy through increasing reactive oxygen species levels in podocytes, and autophagy could protect Ang II-treated podocytes.
These results implied that AngII could effectively induce EpiCs to differentiate into vascular smooth muscle-like cells through the AT1 receptor (show AGTRAP Antibodies).
Results suggest the involvement of angiotensin II (Ang II), through its angiotensin type-1 receptor (AT1R (show AGTRAP Antibodies)) in the inflammation induced by Aah (show ASPH Antibodies) venom, in the heart and the aorta.
In conclusion, endothelial vWF (show VWF Antibodies) knockdown prevented angiotensin II-induced ET-1 (show EDN1 Antibodies) upregulation through attenuation of NOX-mediated O2- production.
Data suggest that intra-adrenal metabolism of Ang II to Ang III is required for zona glomerulosa cell-mediated relaxation of adrenal arterioles but not for aldosterone secretion.
NADPH oxidase (show NOX1 Antibodies) plays an important role in proMMP-2 expression and activation and MMP-2 (show MMP2 Antibodies) mediated SMC (show DYM Antibodies) proliferation occurs through the involvement of Spm (show NPC1 Antibodies)-Cer (show CBLN1 Antibodies)-S1P (show MBTPS1 Antibodies) signaling axis under ANG II stimulation of PASMCs
The metabolism of angiotensin II (Ang II) to angiotensin III (Ang III) and its role in the vasorelaxation response in adrenal arteries are reported.
The study identified the serine phosphorylation (p-Ser (show SIGLEC1 Antibodies)) sites induced by PKC-Beta (show PRKCB Antibodies) activation or AGT, which inhibits insulin (show INS Antibodies)-induced p-Tyr (show TYR Antibodies) sites on IRS2 (show IRS2 Antibodies) and its signals in endothelial cells.
Data suggest up-regulation of AGT in granulosa cells and of Ang II in follicular fluid during preovulatory period; Ang II appears to amplify stimulatory effects of luteinizing hormone on secretion of progesterone/prostaglandins by granulosa cells.
Data suggest that angiotensin II promotes uptake/accumulation of iron (non-transferrin (show Tf Antibodies) bound iron) into vascular endothelial cells; such iron accumulation appears to depend on activation of angiotensin type 1 receptor and promotes oxidative stress.
a critical role for H(2)O(2) in angiotensin-II signaling to the endothelial cytoskeleton in a novel pathway that is critically dependent on MARCKS, Rac1, and c-Abl.
The objective of this study was to characterize the profiles of Ang-(1-7), MAS receptor, ACE(2), NEP and PEP during the ovulatory process in cattle.
Fetal adrenal cells in primary culture respond to angiotensin-II by increasing aldosterone production and aldosterone synthase (show CYP11B2 Antibodies) [P450c18/CYP11B2 (show CYP11B2 Antibodies)] activity.
ANG II inhibits bTREK-1 K(+) channels by a Ca(2+)-dependent mechanism that does not require the depletion of membrane-associated PIP(2).
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8
, angiotensin ll
, angiotensinogen (PAT)
, zC8A9.1 (angiotensinogen )
, Serpin A8