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Human Monoclonal TNFRSF1A Primary Antibody for FACS - ABIN4898906
Rebelo, Bainbridge, Amel-Kashipaz, Radford, Powell, Todd, Tighe: Modeling of tumor necrosis factor receptor superfamily 1A mutants associated with tumor necrosis factor receptor-associated periodic syndrome indicates misfolding consistent with abnormal function. in Arthritis and rheumatism 2006
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Human Monoclonal TNFRSF1A Primary Antibody for CyTOF, FACS - ABIN4900865
Dong, Kleinberg, Silins, Flørenes, Tropé, Risberg, Nesland, Davidson: Death receptor expression is associated with poor response to chemotherapy and shorter survival in metastatic ovarian carcinoma. in Cancer 2007
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Mouse (Murine) Polyclonal TNFRSF1A Primary Antibody for Agon, CyTOF - ABIN5013006
LaBue, Colburn, Green: Thyrocytes isolated from autoimmune-diseased thyroids secrete soluble tumor necrosis factor-R1 that is related to their elevated protein kinase C activity. in Thyroid : official journal of the American Thyroid Association 2004
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Human Monoclonal TNFRSF1A Primary Antibody for ELISA - ABIN2688922
Aggarwal, Gollapudi, Gupta: Increased TNF-alpha-induced apoptosis in lymphocytes from aged humans: changes in TNF-alpha receptor expression and activation of caspases. in Journal of immunology (Baltimore, Md. : 1950) 1999
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Human Monoclonal TNFRSF1A Primary Antibody for FACS, ELISA - ABIN2688925
Brockhaus, Schoenfeld, Schlaeger, Hunziker, Lesslauer, Loetscher: Identification of two types of tumor necrosis factor receptors on human cell lines by monoclonal antibodies. in Proceedings of the National Academy of Sciences of the United States of America 1990
Show all 8 Pubmed References
Human Polyclonal TNFRSF1A Primary Antibody for Agon, FACS - ABIN5013005
Hadjadj, Cloutier, Geoffroy, Tanguay: Stent implantation in coronary porcine arteries is associated with early activation of TNFalpha and TNFalpha receptor II expression. in Atherosclerosis 2007
Show all 5 Pubmed References
Human Monoclonal TNFRSF1A Primary Antibody for FACS - ABIN2688923
Browning, Dougas, Ngam-ek, Bourdon, Ehrenfels, Miatkowski, Zafari, Yampaglia, Lawton, Meier: Characterization of surface lymphotoxin forms. Use of specific monoclonal antibodies and soluble receptors. in Journal of immunology (Baltimore, Md. : 1950) 1995
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Mouse (Murine) Polyclonal TNFRSF1A Primary Antibody for Func, IA - ABIN2192161
Lucas, Garcia, Donati, Hribar, Mandriota, Giroud, Buurman, Fransen, Suter, Nunez, Pepper, Grau: Both TNF receptors are required for direct TNF-mediated cytotoxicity in microvascular endothelial cells. in European journal of immunology 1998
Show all 3 Pubmed References
Human Monoclonal TNFRSF1A Primary Antibody for Func, IHC (f) - ABIN2749013
Kohrgruber, Halanek, Gröger, Winter, Rappersberger, Schmitt-Egenolf, Stingl, Maurer: Survival, maturation, and function of CD11c- and CD11c+ peripheral blood dendritic cells are differentially regulated by cytokines. in Journal of immunology (Baltimore, Md. : 1950) 1999
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Human Monoclonal TNFRSF1A Primary Antibody for FACS - ABIN4898903
Hijdra, Vorselaars, Grutters, Claessen, Rijkers: Differential expression of TNFR1 (CD120a) and TNFR2 (CD120b) on subpopulations of human monocytes. in Journal of inflammation (London, England) 2013
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The TNFRSF1A c.625+10 G allele was associated with late response to anti-TNFalpha (show TNF Antibodies) therapy but TNFRSF1A gene SNPs is not associated with spondyloarthritis.
TNFR1 is associated longitudinally with kidney function decline but not with myocardial infarct, heart failure, or mortality risk after adjustment
One third of our childhood MS patients had a heterozygous mutation in the TNFRSF1A and/or MEFV (show MEFV Antibodies) gene. This proportion by far exceeds the number of mutations expected and was higher than in adult MS patients, suggesting that these mutations might contribute to the pathogenesis of childhood MS.
Studied the association of NLR family pyrin domain containing 3 (NLRP3) and tumor necrosis factor receptor superfamily member 1A (TNFRSF1A) polymorphisms and haplotypes in patients with ankylosing spondylitis (AS) and treatment response to etanercept.
Investigations of underlying molecular mechanisms of TNFR1 signaling showed that PDF affects TNFR1 signaling at the proapoptotic signaling pathway by upregulation of IkappaBalpha (show NFKBIA Antibodies) and downregulation of cFLIPL.
Case Report: tumor necrosis factor (show TNF Antibodies) receptor-associated periodic syndrome due to the R92Q TNFRSF1A variant associated with recurrent pericarditis and cardiac tamponade.
Serum TNFR1 is a biomarker for patients with chronic kidney disease.
This study demonstrated that TNFR1 expression levels are related to major depressive disorder and conjunctly mediate the effect of childhood maltreatment history on the risk of developing major depressive disorder.
Data suggest that Fas (show FAS Antibodies) and TNFR1 are involved in glaucoma mechanisms in cornea; pro-apoptotic effect of anti-glaucoma medication clonidine on corneal epithelial cells triggers Fas (show FAS Antibodies)/TNFR1-mediated, mitochondria-dependent signaling pathway. (Fas (show FAS Antibodies) = Fas (show FAS Antibodies) cell surface death receptor ; TNFR1 = TNF (show TNF Antibodies) receptor superfamily member 1A)
These results indicate that TNFRI-Fc and hHO-1 overexpression may apparently induce free iron in the liver and exert oxidative stress by enhancing reactive oxygen species production and block normal postneonatal liver metabolism.
Results provide experimental data on the existence of a TNFRp55-mediated anti-inflammatory circuit in dendritic cells which might contribute to the protection against reactive arthritis during the resolution of Yersinia entercolitica infection.
TNFR1 contributes to ethanol-induced hypertension and oxidative stress in the vasculature.
TNFR2 (show TNFRSF1B Antibodies) is important for the proliferative expansion of pathogenic Teff cells
NEMO (show IKBKG Antibodies) deficiency hampered activation of IKK (show CHUK Antibodies) complex in osteoclast precursors, causing arrest of osteoclastogenesis and apoptosis. Interestingly, inhibiting apoptosis by genetic ablation of TNFr1 significantly increased cell survival, but failed to rescue osteoclastogenesis or reverse osteopetrosis (show CSF1 Antibodies).
Data indicate that IL-1RI and TNF (show TNF Antibodies)-1R contribute to regulation of stress-induced, negatively reinforced drinking perhaps through overlapping signaling events downstream of these receptors, while leaving rewarding properties of alcohol largely unaffected.
Myeloid-derived suppressor cells from tumor necrosis factor (show TNF Antibodies) receptor (TNFR) 2(-/-) mice, but not from TNFR1(-/-) mice, failed to promote B-cell responses.
TNFR2 (show TNFRSF1B Antibodies) is required for inflammation-inducible M cells.
These findings indicate that TNFR1 is structurally positioned to modulate postsynaptic signaling in the PVN, suggesting a mechanism whereby TNFR1 activation contributes to cardiovascular and other autonomic functions.
Increased TNFR1 expression and signaling in injured peripheral nerves of mice with reduced BACE1 (show BACE Antibodies) activity
Results show that interleukin 6 (IL6 (show IL6 Antibodies)) promotes oval cell proliferation and liver regeneration, while tumor necrosis factor alpha (TNFalpha (show TNF Antibodies)) and TNF (show TNF Antibodies) receptor-1(TNFR1) do not affect this process.
Retinal ischemia results in increased expression of TNF-alpha (show TNF Antibodies) and its receptors (TNF-R1 and TNF-R2 (show TNFRSF1B Antibodies)).
Targeted gene knockdown of TNFRSF1B (show TNFRSF1B Antibodies) in zebrafish embryos results in the induction of a caspase-8 (show CASP8 Antibodies), caspase-2 (show CASP2 Antibodies) and P53 (show TP53 Antibodies)-dependent apoptotic program in endothelial cells that bypasses caspase-3 (show CASP3 Antibodies).
These results suggest that TNF-alpha (show TNF Antibodies) sources include immune cells, as well as large and small luteal cells, and that TNF-RI and TNF-RII (show TNFRSF1B Antibodies) are present in the luteal cells of the bovine corpus luteum.
The expression and cellular localization of tumor necrosis factor-alpha (TNF (show TNF Antibodies)) and its receptors (TNFRI and TNFRII (show TNFRSF1B Antibodies)) mRNAs and proteins, were determined.
The upregulation of TNFRI mRNA expression by IFNG (show IFNG Antibodies) suggests that TNF (show TNF Antibodies) and IFNG (show IFNG Antibodies) synergistically affect the death of luteal endothelial cells resulting in acute luteolysis
TNF (show TNF Antibodies) binding induces release of AIP1 (DAB2IP (show DAB2IP Antibodies)) from TNFR1, resulting in cytoplasmic translocation and concomitant formation of an intracellular signaling complex comprised of TRADD (show TRADD Antibodies), RIP1 (show RALBP1 Antibodies), TRAF2 (show TRAF2 Antibodies), and AIPl.
The protein encoded by this gene is a member of the TNF-receptor superfamily. This protein is one of the major receptors for the tumor necrosis factor-alpha. This receptor can activate NF-kappaB, mediate apoptosis, and function as a regulator of inflammation. Antiapoptotic protein BCL2-associated athanogene 4 (BAG4/SODD) and adaptor proteins TRADD and TRAF2 have been shown to interact with this receptor, and thus play regulatory roles in the signal transduction mediated by the receptor. Germline mutations of the extracellular domains of this receptor were found to be associated with the autosomal dominant periodic fever syndrome. The impaired receptor clearance is thought to be a mechanism of the disease.
, tumor necrosis factor binding protein 1
, tumor necrosis factor receptor 1A isoform beta
, tumor necrosis factor receptor superfamily member 1A
, tumor necrosis factor receptor type 1
, tumor necrosis factor-alpha receptor
, TNF receptor alpha chain
, tumor necrosis factor receptor 1
, tumor necrosis factor receptor type I
, p55 TNF receptor
, tumor necrosis factor receptor p60
, TNF Receptor 1 (TR1)
, tumor necrosis factor type I
, tumor necrosis factor receptor superfamily, member 1A
, tumor necrosis factor receptor superfamily member 1A-like
, tumor necrosis factor (TNF superfamily, member 2)
, tumor necrosis factor alpha
, tumor necrosis factor ligand superfamily member 2
, tumour necrosis factor