Gastric Inhibitory Polypeptide (GIP) Peptide
Quick Overview for Gastric Inhibitory Polypeptide (GIP) Peptide (ABIN399441)
Target
-
-
Purity
- > 95 %
-
-
Gastric Inhibitory Polypeptide (GIP) peptide (BSA)
GIP Reactivity: Rat Host: Synthetic Imm, SDS, WB
Gastric Inhibitory Polypeptide (GIP) peptide (BSA)GIP Reactivity: Mouse Host: Synthetic Imm, SDS, WB
Gastric Inhibitory Polypeptide (GIP) peptide (Ovalbumin)GIP Reactivity: Rat Host: Synthetic Imm, SDS, WB
Gastric Inhibitory Polypeptide (GIP) peptide (Ovalbumin)GIP Reactivity: Mouse Host: Synthetic Imm, SDS, WB
Gastric Inhibitory Polypeptide (GIP) peptide (Ovalbumin)GIP Reactivity: Human Host: Synthetic Imm, SDS, WB
-
-
Restrictions
- For Research Use only
-
-
-
Storage
- -20 °C
-
-
- GIP (Gastric Inhibitory Polypeptide (GIP))
-
Background
- GIP, also known as gastric inhibitory polypeptide, or glucose-dependent insulinotropic polypeptide, is a 42-amino-acid peptide hormone synthesized in and secreted from K cells in the intestinal epithelium. There are two major GIP molecular forms in circulation, GIP (1-42) and GIP(3-42). Previous studies have demonstrated that GIP (3-42) is a degraded form of GIP (1-42) by the enzyme DPPIV. GIP secretion is primarily regulated by nutrients, especially fat. GIP exhibits potent incretin activity in rodent and human subjects. The primary action of GIP is the stimulation of glucose-dependent insulin secretion. GIP may also play a role in adipocyte biology.
-
CAS-No
- 100040-31-1
Target
-
