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CCL3 Protein

This Recombinant CCL3 protein is expressed in Yeast (Pichia pastoris).
Catalog No. ABIN7536055

Quick Overview for CCL3 Protein (ABIN7536055)

Target

See all CCL3 Proteins
CCL3 (Chemokine (C-C Motif) Ligand 3 (CCL3))

Protein Type

Recombinant

Origin

  • 17
  • 8
  • 7
  • 3
  • 1
  • 1
  • 1
Human

Source

  • 27
  • 4
  • 3
  • 1
  • 1
  • 1
Yeast (Pichia pastoris)

Purity

> 95 % by SDS-PAGE.
  • Purpose

    Recombinant Human CCL3/MIP-1 alpha Protein

    Sequence

    ASLAADTPTA CCFSYTSRQI PQNFIADYFE TSSQCSKPGV IFLTKRSRQV CADPSEEWVQ KYVSDLELSA

    Specificity

    Ala23-Ala92

    Sterility

    0.22 μm filtered

    Endotoxin Level

    601EU/μg
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  • Restrictions

    For Research Use only
  • Format

    Lyophilized

    Reconstitution

    Centrifuge the vial before opening. Reconstitute to a concentration of 0.1-0.5 mg/mL in sterile distilled water. Avoid votex or vigorously pipetting the protein. For long term storage, it is recommended to add a carrier protein or stablizer (e.g. 0.1 % BSA, 5 % HSA, 10 % FBS or 5 % Trehalose), and aliquot the reconstituted protein solution to minimize free-thaw cycles.

    Buffer

    Lyophilized from a 0.22 μm filtered solution of PBS, pH 7.4.

    Storage

    -20 °C,-80 °C

    Storage Comment

    Store the lyophilized protein at -20°C to -80°C for 12 months.|After reconstitution, the protein solution is stable at -20°C for 3 months, at 2-8°C for up to 1 week.
  • Target

    CCL3 (Chemokine (C-C Motif) Ligand 3 (CCL3))

    Alternative Name

    CCL3/MIP-1 alpha

    Background

    Description: Monokine with inflammatory and chemokinetic properties. Binds to CCR1, CCR4 and CCR5. One of the major HIV-suppressive factors produced by CD8+ T-cells. Recombinant MIP-1-alpha induces a dose-dependent inhibition of different strains of HIV-1, HIV-2, and simian immunodeficiency virus (SIV).

    Name: MIP1A, SCYA3, G0S19-1, LD78ALPHA, MIP-1-alpha,CCL3

    Gene ID

    6348

    UniProt

    P10147

    Pathways

    Cellular Response to Molecule of Bacterial Origin, Autophagy
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