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Human Polyclonal SLC1A3 Primary Antibody for ICC, FACS - ABIN151381
Kobayashi, Millhorn: Hypoxia regulates glutamate metabolism and membrane transport in rat PC12 cells. in Journal of neurochemistry 2001
Show all 11 Pubmed References
Human Monoclonal SLC1A3 Primary Antibody for ELISA, WB - ABIN520226
Yamada, Urano-Tashiro, Tanaka, Akiyama, Tashiro: Involvement of crosstalk between Oct4 and Meis1a in neural cell fate decision. in PLoS ONE 2013
Human Polyclonal SLC1A3 Primary Antibody for WB - ABIN520225
Xiao, Li, Jones-Brando, Yolken: Abnormalities of neurotransmitter and neuropeptide systems in human neuroepithelioma cells infected by three Toxoplasma strains. in Journal of neural transmission (Vienna, Austria : 1996) 2013
Human Polyclonal SLC1A3 Primary Antibody for IF (p), IHC (p) - ABIN730783
Xiao, Xiao, Meng, Du, Hu, Gao, Yu, Wang, Ban: Effect of subarachnoid nerve block anesthesia on glutamate transporter GLAST and GLT-1 expressions in rabbits. in Asian Pacific journal of tropical medicine 2015
Rat (Rattus) Polyclonal SLC1A3 Primary Antibody for ELISA, ICC - ABIN346976
Rothstein, Dykes-Hoberg, Pardo, Bristol, Jin, Kuncl, Kanai, Hediger, Wang, Schielke, Welty: Knockout of glutamate transporters reveals a major role for astroglial transport in excitotoxicity and clearance of glutamate. in Neuron 1996
Show all 2 Pubmed References
Human Polyclonal SLC1A3 Primary Antibody for ELISA, WB - ABIN4306802
Vandenberg, Handford, Campbell, Ryan, Yool: Water and urea permeation pathways of the human excitatory amino acid transporter EAAT1. in The Biochemical journal 2011
Eaat1 expression is promoted by Fringe-mediated neuron-glial communication during development and suggest that Eaat1 plays an essential role in regulating central nervous system neural circuits that control locomotion in Drosophila
dEAAT1 is present at the adult, but surprisingly not at embryonic and larval NMJ, suggesting a physiological maturation of the junction during metamorphosis
Episodic ataxias 6 is caused by heterozygous mutations in SLC1A3, which encodes a subunit of a glial excitatory amino acid transporter, EAAT1.
a novel missense mutation, c.383T>G (p.Met128Arg) in SLC1A3, in an episodic ataxia patient by whole-exome sequencing.
This is the first study to link SLC1A3 and EPHB2 (show EPHB2 Antibodies) to clinically relevant vertebral osteoporosis phenotypes.
crystal structures of a thermostabilized human SLC1 (show MCHR1 Antibodies) transporter, the excitatory amino acid transporter 1 (EAAT1), with and without allosteric and competitive inhibitors bound
This study demonstrated that the cytopathology and episodic paralysis in our Drosophila EA6 model stem from a gain-of-function chloride channelopathy of glial cells..
Starvation of Muller cells increased the glutamate (show GRIN1 Antibodies) uptake capacity as well as the expression of the most abundant glutamate transporter (show SLC1A1 Antibodies), EAAT1.
A heterozygous SLC1A3 c.1177G4A mutation has been detected in a patient with late-onset episodic ataxia. Same heterozygous mutation was identified in one clinically affected family member and two asymptomatic members.
that Abeta1-42 oligomers could cause disturbances in insulin (show INS Antibodies)/Akt (show AKT1 Antibodies)/EAAT (show SLC1A2 Antibodies) signaling in astrocytes
We consider an association between SLC1A3 and the behavioural problems which can also be considered a contributing factor to behavioural problems in larger duplications overlapping the 5p13 microduplication syndrome region.
In combination with other nearby residues, Arg-388 coordinates anion channel gating and forms part of the missing structural link between the anion conducting and substrate transport states in EAAT1.
Study shows that high-fat feeding induces metabolic disorders and disrupts lactate metabolism in the hippocampus. Glial glutamate transporters GLAST and GLT-1 may contribute to the high-fat diet induced abnormalities of the hippocampal lactate metabolism.
glutamate transporter (show SLC1A1 Antibodies) function by GLAST on Bergmann glia plays important roles in development and maintenance of proper synaptic wiring and wrapping in Purkinje cells
Loss of EAAT4 (show SLC1A6 Antibodies) accounts for the initial hyper-excitability of Purkinje cells lacking b-III spectrin and that loss of GLAST appears to work synergistically to worsen motor deficits. When levels of both EAAT4 (show SLC1A6 Antibodies) and GLAST are compromised in b-III(-/-) mice, the proximal dendrites of Purkinje cells within the posterior cerebellum are the most vulnerable to degeneration.
The results of this study concluded that EAAT (show SLC1A2 Antibodies) anion channels play an important and unexpected role in adjusting glial intracellular anion concentration during maturation and in response to cerebellar activity.
SLC1A3 maintains a constant import of acidic amino acids independently of nutritional status in adipocytes.
glutamate/aspartate transporter (GLAST) was elevated in 8- to 10- and/or 20- to 22-month GHR (show GHR Antibodies)-KO mice when comparing genotypes
This study findings demonstrate that Ascl1 (show ASCL1 Antibodies)(CreERT2) and Glast(CreERT2) mouse lines enable simple and reliable labeling of adult-born GC lineages within restricted time windows.
arundic acid treatment prevented RGC death by upregulating GLAST in heterozygous (GLAST(+/-)) mice
Results show that expression of GLAST is decreased in cerebellar astrocytes in a mouse model of SCA1 (show ATXN1 Antibodies); decrease occurs in non-cell autonomous manner late in disease and correlates well with the loss of Purkinje neurons
we have demonstrated for the first time that DOR receptor activation induces astrocytic expression of EAAT1 and EAAT2 (show SLC1A2 Antibodies)
This gene encodes a member of a member of a high affinity glutamate transporter family. Mutations in this gene are associated with episodic ataxia, Type 6. Alternative splicing results in multiple transcript variants.
, drosophila glutamate transporter-1
, excitatory amino acid transporter 1
, glutamate transporter
, sodium-dependent excitatory amino acid transporter 1
, sodium-dependent glutamate transporter
, excitatory amino acid transporter SLC1A3a
, solute carrier family 1 (glial high affinity glutamate transporter), member 3
, Na+-dependent glutamate/aspartate transporter
, Sodium-dependent glutamate/aspartate transporter 1
, excitatory amino acid transporter 1-like
, glial glutamate transporter
, glutamate/aspartate transporter
, sodium-dependent glutamate/aspartate transporter 1
, solute carrier family 1, member 3
, solute carrier family 1 member 3
, glial high affinity glutamate transporter
, high-affinity neuronal glutamate transporter