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Human Polyclonal SLC1A3 Primary Antibody for ICC, FACS - ABIN151381
Kobayashi, Millhorn: Hypoxia regulates glutamate metabolism and membrane transport in rat PC12 cells. in Journal of neurochemistry 2001
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Mouse (Murine) Polyclonal SLC1A3 Primary Antibody for IHC (fro), IHC (p) - ABIN3044001
Ding, Zhang, Liu, Zhang, Ma, Bruce, Zhang: Tumor necrosis factor-? promotes the expression of excitatory amino-acid transporter 2 in astrocytes: Optimal concentration and incubation time. in Experimental and therapeutic medicine 2014
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Rat (Rattus) Polyclonal SLC1A3 Primary Antibody for ELISA, ICC - ABIN346976
Rothstein, Dykes-Hoberg, Pardo, Bristol, Jin, Kuncl, Kanai, Hediger, Wang, Schielke, Welty: Knockout of glutamate transporters reveals a major role for astroglial transport in excitotoxicity and clearance of glutamate. in Neuron 1996
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Human Polyclonal SLC1A3 Primary Antibody for ELISA, WB - ABIN4306803
Vandenberg, Handford, Campbell, Ryan, Yool: Water and urea permeation pathways of the human excitatory amino acid transporter EAAT1. in The Biochemical journal 2011
Human Polyclonal SLC1A3 Primary Antibody for IF (p), IHC (p) - ABIN730783
Xiao, Xiao, Meng, Du, Hu, Gao, Yu, Wang, Ban: Effect of subarachnoid nerve block anesthesia on glutamate transporter GLAST and GLT-1 expressions in rabbits. in Asian Pacific journal of tropical medicine 2015
Human Polyclonal SLC1A3 Primary Antibody for WB - ABIN520225
Xiao, Li, Jones-Brando, Yolken: Abnormalities of neurotransmitter and neuropeptide systems in human neuroepithelioma cells infected by three Toxoplasma strains. in Journal of neural transmission (Vienna, Austria : 1996) 2013
Human Monoclonal SLC1A3 Primary Antibody for ELISA, WB - ABIN520226
Yamada, Urano-Tashiro, Tanaka, Akiyama, Tashiro: Involvement of crosstalk between Oct4 and Meis1a in neural cell fate decision. in PLoS ONE 2013
A Drosophila tripartite synapse model which exhibits morphological and functional properties similar to those of mammalian synapses.
Eaat1 expression is promoted by Fringe-mediated neuron-glial communication during development and suggest that Eaat1 plays an essential role in regulating central nervous system neural circuits that control locomotion in Drosophila
Terminal glial differentiation involves regulated expression of the excitatory amino acid transporters in the Drosophila embryonic CNS.
dEAAT1 is present at the adult, but surprisingly not at embryonic and larval NMJ, suggesting a physiological maturation of the junction during metamorphosis
EAAT1 rs2731880 SNP is associated with amygdala functional connectivity in bipolar disorder.
Episodic ataxias 6 is caused by heterozygous mutations in SLC1A3, which encodes a subunit of a glial excitatory amino acid transporter, EAAT1.
a novel missense mutation, c.383T>G (p.Met128Arg) in SLC1A3, in an episodic ataxia patient by whole-exome sequencing.
This is the first study to link SLC1A3 and EPHB2 to clinically relevant vertebral osteoporosis phenotypes.
crystal structures of a thermostabilized human SLC1 transporter, the excitatory amino acid transporter 1 (EAAT1), with and without allosteric and competitive inhibitors bound
This study demonstrated that the cytopathology and episodic paralysis in our Drosophila EA6 model stem from a gain-of-function chloride channelopathy of glial cells..
Starvation of Muller cells increased the glutamate uptake capacity as well as the expression of the most abundant glutamate transporter, EAAT1.
A heterozygous SLC1A3 c.1177G4A mutation has been detected in a patient with late-onset episodic ataxia. Same heterozygous mutation was identified in one clinically affected family member and two asymptomatic members.
that Abeta1-42 oligomers could cause disturbances in insulin/Akt/EAAT signaling in astrocytes
We consider an association between SLC1A3 and the behavioural problems which can also be considered a contributing factor to behavioural problems in larger duplications overlapping the 5p13 microduplication syndrome region.
In combination with other nearby residues, Arg-388 coordinates anion channel gating and forms part of the missing structural link between the anion conducting and substrate transport states in EAAT1.
SPAK and OSR1 are powerful negative regulators of the excitatory glutamate transporters EAAT1 and EAAT2.
There was no association between pyramidal cell EAAT1 splice variant expression and schizophrenia.
data provide additional insights into the mechanism by which substrates gate the anion conductance in EAATs and suggest that in EAAT1, Arg-388 is a critical element for the structural coupling between the substrate translocation and the gating mechanisms
discovering compounds that can enhance EAAT1 expression and activity may be a novel strategy for therapeutic treatment of glaucoma.
Results showed that EAAT2 levels were significantly decreased in the essential tremor cerebellar cortex, in contrast to similar levels of EAAT1 levels between essential tremor cases and controls
EAAT1 polymorphism which is involved in the Regulation of extracellular glutamate concentrations, influences Cognitive performances with a detrimental effect of T/T homozygosis.
the delivered miR-124 increased the expression of the glutamate transporters, EAAT1 in NPCs and EAAT2 in both NPCs and astrocytes.
Results describe EAAT1 and EAAT2 labeling in the prefrontal cortex in human postmortem control brains at the light and electron microscopic level
Its dysregulation may contribute to the pathology and possibly affect the onset of fragile X-associated tremor/ataxia syndrome.
This gene encodes a member of a member of a high affinity glutamate transporter family. Mutations in this gene are associated with episodic ataxia, Type 6. Alternative splicing results in multiple transcript variants.
, drosophila glutamate transporter-1
, excitatory amino acid transporter 1
, glutamate transporter
, sodium-dependent excitatory amino acid transporter 1
, sodium-dependent glutamate transporter
, excitatory amino acid transporter SLC1A3a
, solute carrier family 1 (glial high affinity glutamate transporter), member 3
, Na+-dependent glutamate/aspartate transporter
, Sodium-dependent glutamate/aspartate transporter 1
, excitatory amino acid transporter 1-like
, glial glutamate transporter
, glutamate/aspartate transporter
, sodium-dependent glutamate/aspartate transporter 1
, solute carrier family 1, member 3
, solute carrier family 1 member 3
, glial high affinity glutamate transporter
, high-affinity neuronal glutamate transporter