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Human Polyclonal NR2C2 Primary Antibody for IF (p), IHC (p) - ABIN701185
Wang, Zhao, Yu, Feng, Zhang, Kou, Chu, Cui, Li, Zhang, Shen, Min: Regulation of steroid hormones and energy status with cysteamine and its effect on spermatogenesis. in Toxicology and applied pharmacology 2016
Show all 5 Pubmed References
Human Polyclonal NR2C2 Primary Antibody for ELISA, WB - ABIN251479
Omori, Matsumoto, Sanjo, Sato, Akira, Smart, Ninomiya-Tsuji: TAK1 is a master regulator of epidermal homeostasis involving skin inflammation and apoptosis. in The Journal of biological chemistry 2006
Human Polyclonal NR2C2 Primary Antibody for IF (p), IHC (p) - ABIN802023
Su, Zhou, Wang, Yang, Li, Yu, Li: The PPARβ/δ agonist GW501516 attenuates peritonitis in peritoneal fibrosis via inhibition of TAK1-NFκB pathway in rats. in Inflammation 2014
Human Polyclonal NR2C2 Primary Antibody for IF (p), IHC (p) - ABIN746333
Dvashi, Goldberg, Adir, Shapira, Pollack: TGF-?1 induced transdifferentiation of rpe cells is mediated by TAK1. in PLoS ONE 2015
Human Polyclonal NR2C2 Primary Antibody for WB - ABIN4357771
Liepelt, Mossanen, Denecke, Heymann, De Santis, Tacke, Marx, Ostareck, Ostareck-Lederer: Translation control of TAK1 mRNA by hnRNP K modulates LPS-induced macrophage activation. in RNA (New York, N.Y.) 2014
blockage of RhoA (show RHOA Antibodies)/ROCK repressed the TAK1 (show MAP3K7 Antibodies)/NOD2 (show NOD2 Antibodies)-mediated NF-kappaB (show NFKB1 Antibodies) pathway in HaCaT cells exposed to UVB.
Study found that TR4 might be able to function through activation of the AKT3 (show AKT3 Antibodies) expression to drive the EMT (show ITK Antibodies) phenotype and enhance the seminoma cell proliferation and invasion.
Altering TR4-ATF3 (show ATF3 Antibodies) signaling increases the efficacy of cisplatin to suppress hepatocellular carcinoma growth/progression.
High TAK1 (show MAP3K7 Antibodies) expression is associated with the progression of hepatocellular carcinoma.
Here, we report that Pseudomonas aeruginosa ExoY inhibits proinflammatory cytokine production through suppressing the activation of TAK1 (show MAP3K7 Antibodies) as well as downstream NF-kappaB (show NFKB1 Antibodies) and mitogen-activated protein (MAP) kinases.
SIRT7 inhibits TR4 degradation by deacetylation of DDB1.
TR4 binds GR to play an important role in glucocorticoid-directed corticotroph tumor POMC (show POMC Antibodies) regulation in addition to modulating glucocorticoid actions on other GR targets.
this study shows that TAP1 (show TAP1 Antibodies) plays a novel role in the negative regulation of virus-triggered NF-kappaB (show NFKB1 Antibodies) signaling and the innate immune response by targeting the TAK1 (show MAP3K7 Antibodies) complex
TAK1 (show MAP3K7 Antibodies)/TAB1 (show TAB1 Antibodies) expression in non-small cell lung carcinoma tissue is significantly increased and closely associated with patient clinical prognosis.
miR (show MLXIP Antibodies)-203 represses NF-kappaB (show NFKB1 Antibodies) signaling via targeting TAK1 (show MAP3K7 Antibodies) and PI3KCA and miR (show MLXIP Antibodies)-203 overexpression may contribute to the COPD (show ARCN1 Antibodies) initiation.
TNFalpha (show TNF Antibodies)-induced phosphorylation of RIPK1 (show RIPK1 Antibodies) in the intermediate domain by TAK1 plays a key role in regulating the decision between three distinct mechanisms of cell death: necroptosis, RIPK1 (show RIPK1 Antibodies)-independent and dependent apoptosis.
The results suggest that an intron-free Renilla luciferase reporter may provide a satisfactory internal control for TR4 at certain dose range. Findings advocate caution on the use of Renilla luciferase as an internal control in TR4-directed studies to avoid misleading data interpretation.
We conclude that TR4 is required for the normal differentiation and proliferation of erythroid cells, in addition to its previously characterized roles in embryonic and fetal globin gene repression
CNS-specific Tak1 deletion prevented ER-stress-induced hypothalamic leptin (show LEP Antibodies) resistance and hyperphagic obesity under a high-fat diet (HFD). Thus, TAK1 is a crucial regulator of ER stress in vivo, which could be a target for alleviation of ER stress and its associated disease conditions.
this study shows that TAK1 negatively regulates lipopolysaccharide-induced cytokine secretion in myeloid cells by inhibiting MEKK3 (show MAP3K3 Antibodies) activities
The present study demonstrates that TIPE2 (show TNFAIP8L2 Antibodies) acts as a novel negative regulator of inflammatory and immune responses through TAK1 signaling.
this study shows that increased activity of TAK1 contributes to diabetic nephropathy
Salidroside (SDS (show SDS Antibodies)) downregulated protein expression of toll-like receptor 4 (TLR4 (show TLR4 Antibodies)) and CD14 (show CD14 Antibodies). SDS (show SDS Antibodies) inhibited LPS (show TLR4 Antibodies)-triggered phosphorylation of LPS (show TLR4 Antibodies)-activated kinase 1 (TAK1), p38 (show CRK Antibodies), c-Jun (show JUN Antibodies) terminal kinase (JNK (show MAPK8 Antibodies)), and extracellular signal-regulated kinase (ERK (show EPHB2 Antibodies)).
Members of the nuclear hormone receptor family, such as NR2C2, act as ligand-activated transcription factors. The proteins have an N-terminal transactivation domain, a central DNA-binding domain with 2 zinc fingers, and a ligand-binding domain at the C terminus. The activated receptor/ligand complex is translocated to the nucleus where it binds to hormone response elements of target genes (Yoshikawa et al., 1996
Nuclear hormone receptor TR4
, TR4 nuclear hormone receptor
, nuclear receptor subfamily 2 group C member 2
, orphan nuclear receptor TAK1
, orphan nuclear receptor TR4
, testicular nuclear receptor 4
, testicular receptor 4
, TR4 orphan receptor
, TR4-NS orphan receptor
, orphan receptor TR4
, nuclear receptor subfamily 2, group H, member 2
, orphan receptor, TR4