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Methionine is an essential amino acid required for protein synthesis and one-carbon metabolism. Additionally we are shipping MTRR Antibodies (10) and MTRR Kits (4) and many more products for this protein.
Showing 6 out of 12 products:
study widens the clinical, molecular, metabolic, and cytological knowledge of deficiency MTRR enzyme.
The present study suggests that the G allele of MTR (show MTR Proteins) A2756G polymorphism is associated with an increased risk of autism.
MTHFR (show MTHFR Proteins) C667T/A1298C and MTRR A66G genotypes are not associated with premature ovarian failure development, but they affect the patients' serum homocysteine concentrations.
MTRR genetic polymorphisms are risk factor for predicting cardiovascular manifestations in Marfan syndrome.
We aimed to explore the correlation between unexplained recurrent spontaneous abortion and polymorphisms in the methylene tetrahydrofolate reductase (MTHFR (show MTHFR Proteins)) and methionine synthase reductase (MTRR) genes.
an association between MTRR 66 and SHMT1 (show SHMT1 Proteins) 1420 polymorphisms and spaceflight-induced vision changes
Results identified an allelic variant in the first intron of MTRR that is associated with increased risk of anencephaly in neural tube defects cases.
polymorphisms of the MTHFR (show MTHFR Proteins), MTRR, and MTR (show MTR Proteins) enzymes are well documented as folate deficiency-related disorders. The aim of this study was to compare the genotypic distribution of these gene polymorphisms between patients with acromegaly and controls.
Variation in MTHFR (show MTHFR Proteins), MTR (show MTR Proteins), and MTRR were significantly associated with percent LINE-1 methylation.DNA methylation of LINE-1 elements in histologically normal breast tissues is influenced by polymorphisms in genes in the one-carbon metabolism pathway
interactions among homocysteine metabolism gene polymorphisms in MTHFR (show MTHFR Proteins), MTR (show MTR Proteins) and MTRR lead to dramatic elevations in the folate deficiency risk
The Mtrr genotype of either maternal grandparent dictates the developmental potential of their wild-type grandprogeny. These effects are associated with altered DNA methylation (show HELLS Proteins) patterns and two distinct phenotypes: intrauterine growth defects and congenital malformations that are separable through embryo transfer experiments.
Mtrr deficiency adversely impacts reproductive outcomes and cardiac development in mice.
Methionine is an essential amino acid required for protein synthesis and one-carbon metabolism. Its synthesis is catalyzed by the enzyme methionine synthase. Methionine synthase eventually becomes inactive due to the oxidation of its cob(I)alamin cofactor. The protein encoded by this gene regenerates a functional methionine synthase via reductive methylation. It is a member of the ferredoxin-NADP(+) reductase (FNR) family of electron transferases. Patients of the cbl-E complementation group of disorders of folate/cobalamin metabolism are defective in reductive activation of methionine synthase. Alternative splicing of this gene results in multiple transcript variants encoding distinct isoforms.
, 5-methyltetrahydrofolate-homocysteine methyltransferase reductase
, 5-methyltetrahydrofolate-homocysteine methyltransferase
, methionine synthase reductase
, 5-methyltetrahydrofolate--homocysteine methyltransferase
, [methionine synthase]-cobalamin methyltransferase (cob(II)alamin reducing)
, methionine synthase reductase, mitochondrial