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The ALAD enzyme is composed of 8 identical subunits and catalyzes the condensation of 2 molecules of delta-aminolevulinate to form porphobilinogen (a precursor of heme, cytochromes and other hemoproteins). Additionally we are shipping Aminolevulinate Dehydratase Kits (33) and Aminolevulinate Dehydratase Proteins (9) and many more products for this protein.
Showing 10 out of 54 products:
Cow (Bovine) Polyclonal ALAD Primary Antibody for WB - ABIN2776921
Tang, Breinig, Stith, Mischel, Tannir, Kokona, Fairman, Jaffe: Single amino acid mutations alter the distribution of human porphobilinogen synthase quaternary structure isoforms (morpheeins). in The Journal of biological chemistry 2006
Human Polyclonal ALAD Primary Antibody for WB - ABIN513229
Anand, Wilson, Hasan, Maytin: Vitamin D3 enhances the apoptotic response of epithelial tumors to aminolevulinate-based photodynamic therapy. in Cancer research 2011
Human Polyclonal ALAD Primary Antibody for FACS, IHC (p) - ABIN390809
Guey, García-Closas, Murta-Nascimento, Lloreta, Palencia, Kogevinas, Rothman, Vellalta, Calle, Marenne, Tardón, Carrato, García-Closas, Serra, Silverman, Chanock, Real, Malats: Genetic susceptibility to distinct bladder cancer subphenotypes. in European urology 2010
Maternal ALAD gene polymorphism can affect early neonatal neurobehavioral development by influencing the blood lead level.
Genetic variation of ALAD is associated with blood lead level.
This study investigated the delta-aminolevulinate dehydratase (delta-ALA-D) activity in whole blood as well as the parameters of oxidative stress, in lung cancer.
Blood lead level affected ALAD but not GPx (show GPX1 Antibodies) activities, and these were not modulated by polymorphisms in ALAD and GPx (show GPX1 Antibodies) genes.
Study reveals relationship between ALAD genetic polymorphisms and basic lead toxicological parameters in occupationally exposed workers.
The ff carriers in VDR (show CYP27B1 Antibodies) FokI polymorphism were more susceptible to the effect of lead on the balance system, while other VDR (show CYP27B1 Antibodies) or ALAD genotypes did not significantly modify the effect.
Report the effect of ALAD polymorphism on hematopoietic, hepatic and renal toxicity from lead in occupational exposure workers.
Genetic variation in ALAD may modify associations between Pb and prostate cancer
The results lend support to the notion that ALAD polymorphism exerts no marked impact on lead body burden.
Workers with the ALAD 1-1 genotype were associated with higher blood lead levels than those with the ALAD 1-2 genotype.
Similar to its role at the beta-globin (show HBB Antibodies) promoter, KLF1 (show KLF1 Antibodies) induces factor recruitment and chromatin changes at the Alad1b promoter in a temporally-specific manner
The data supports the hypothesis that ALAD, an important enzyme for heme synthesis, is also important as a proteasome interacting protein.
The ALAD enzyme is composed of 8 identical subunits and catalyzes the condensation of 2 molecules of delta-aminolevulinate to form porphobilinogen (a precursor of heme, cytochromes and other hemoproteins). ALAD catalyzes the second step in the porphyrin and heme biosynthetic pathway\; zinc is essential for enzymatic activity. ALAD enzymatic activity is inhibited by lead and a defect in the ALAD structural gene can cause increased sensitivity to lead poisoning and acute hepatic porphyria.
, aminolevulinate, delta-, dehydratase
, delta-aminolevulinic acid dehydratase
, Delta-aminolevulinic acid dehydratase
, aminolevulinate dehydratase
, delta-aminolevulinic acid dehydratase-like
, delta - aminolevulinic acid dehydratase