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The transmembrane protein encoded by ANO10 is a member of a family of calcium-activated chloride channels. Additionally we are shipping Anoctamin 10 Antibodies (13) and many more products for this protein.
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results suggest that executive and attentional disorders are impaired in ANO10 mutation
ANO10 has a central role in innate immune defense against Borrelia infection.
The detection of mutations in ANO10 indicate that ANO10 defects cause secondary low coenzyme Q10 (show EIF2C2 Proteins).
An ANO10 mutation is responsible for autosomal recessive cerebellar ataxia (show USP14 Proteins) that is mainly characterized by cerebellar atrophy and lack of peripheral neuropathy.
Whole-exome and targeted sequencing have defined the genetic basis of dizziness including new genes causing ataxia (show USP14 Proteins): GBA2 (show GBA2 Proteins), TGM6 (show TGM5 Proteins), ANO10 and SYT14 (show SYT14 Proteins)
This study report Gypsy family with autosomal recessive ataxia (show USP14 Proteins) caused by the same truncating ANO10 defect.
New DNA sequencing technologies are enabling us to investigate the whole or large targeted proportions of the genome in a rapid, affordable, and comprehensive way. Exome and targeted sequencing ANO10 genes causing ataxia (show USP14 Proteins).
Ano10 is essential for Ca2 (show CA2 Proteins)+-dependent Cl- secretion in jejunum.
Data show the expression of Tmem16k during murine embryogenesis with an emphasis on the respiratory, digestive, skeletal, and integumentary systems.
The transmembrane protein encoded by this gene is a member of a family of calcium-activated chloride channels. Defects in this gene may be a cause of autosomal recessive spinocerebellar ataxia-10. Several transcript variants encoding different isoforms have been found for this gene.
, transmembrane protein 16K