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The protein encoded by BLVRA belongs to the biliverdin reductase family, members of which catalyze the conversion of biliverdin to bilirubin in the presence of NADPH or NADH. Additionally we are shipping Biliverdin Reductase Antibodies (95) and Biliverdin Reductase Proteins (14) and many more products for this protein.
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Suggest that Gata-1 (show GATA1 ELISA Kits) and Nrf2a (show NFE2L2 ELISA Kits) play differential roles in regulating the heme degradation enzymes hmox1a/bvra/bvrb (show BLVRB ELISA Kits) during an early developmental period of heightened cellular stress
BLVRA mRNA levels in the liver as well as in peripheral blood leukocytes are significantly higher in hepatocellular carcinoma patients most likely as a feedback mechanism to control increased oxidative stress associated with HCC (show FAM126A ELISA Kits) progression.
Data suggest that isoenzymes BVRA and BVRB (show BLVRB ELISA Kits) play different roles in energy metabolism and in pathogenesis of abdominal obesity and hypertriglyceridemia. [REVIEW]
Interactions of HO-2 (show HMOX2 ELISA Kits) with CPR (show POR ELISA Kits) and BVR, were evaluated.
Increased biliverdin reductase expression is associated with multidrug resistance in leukemia.
Our results suggest that patients with chronic HCV infection significantly upregulate BLVRA expression in PBL.
The current study reports increased levels of both HO-1 (show HMOX1 ELISA Kits) and BVR-A in plasma from probable Alzheimer disease patients, as a result of the increased oxidative environment.
rs699512 (Thr3Ala), the only common non-synonymous SNP within BLVRA, reduced the risk of essential hypertension in Kazaks.
A significant increase of nitrated BVRA was demonstrated only in Alzheimer's disease and mild cognitive impairment hippocampi
A homozygous BLVRA inactivating mutation is associated with the appearance of green jaundice accompanying cholestasis episodes.
data support the notion that BLVRA contributes significantly to modulation of the aging process by adjusting the cellular oxidative status.
BVR-A integrates both oxidative/nitrosative stress- and insulin (show INS ELISA Kits)-mediated signaling, both mechanisms dysregulated in the Alzheimer disease brain.
The biliverdin reductase A positive macrophage is a source of anti-inflammatory cytokine IL-10 (show IL10 ELISA Kits) in injured kidney.
Lack of p53 (show TP53 ELISA Kits) decreases basal oxidative stress levels in the brain through upregulation of thioredoxin-1 (show TXN ELISA Kits), biliverdin reductase-A, manganese superoxide dismutase (show SOD2 ELISA Kits), and nuclear factor kappa-B.
biliverdin reductase has a role in regulating the inflammatory response to endotoxin that requires eNOS (show NOS3 ELISA Kits)-derived NO and TLR4 (show TLR4 ELISA Kits) signaling in macrophages
biliverdin reductase mediates biliverdin-induced anti-inflammatory effects via phosphatidylinositol 3-kinase and Akt (show AKT1 ELISA Kits)
Inhibition of biliverdin reductase increases ANG II (show AGT ELISA Kits)-dependent superoxide levels in cultured renal tubular epithelial cells.
biliverdin reductase A (BLVRA) was identified as a partner of ELSPBP1 (show ELSPBP1 ELISA Kits) by immunoprecipitation followed by tandem mass spectrometry.
The protein encoded by this gene belongs to the biliverdin reductase family, members of which catalyze the conversion of biliverdin to bilirubin in the presence of NADPH or NADH. Mutations in this gene are associated with hyperbiliverdinemia. Alternatively spliced transcript variants have been found for this gene.
biliverdin reductase A
, biliverdin IX-beta reductase
, biliverdin reductase
, biliverdin reductase a
, BVR A
, biliverdin-IX alpha-reductase