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Calcium Channel, Voltage-Dependent, T Type, alpha 1G Subunit Proteins (CACNA1G)

Voltage-sensitive calcium channels mediate the entry of calcium ions into excitable cells, and are also involved in a variety of calcium-dependent processes, including muscle contraction, hormone or neurotransmitter release, gene expression, cell motility, cell division, and cell death. Additionally we are shipping CACNA1G Antibodies (80) and many more products for this protein.

list all proteins Gene Name GeneID UniProt
CACNA1G 8913 O43497
Mouse CACNA1G CACNA1G 12291  
Rat CACNA1G CACNA1G 29717 O54898
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Top CACNA1G Proteins at antibodies-online.com

Showing 1 out of 4 products:

Catalog No. Origin Source Conjugate Images Quantity Supplier Delivery Price Details
HOST_Escherichia coli (E. coli) Human His tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 1 mg Log in to see 29 to 34 Days
$4,331.68
Details

CACNA1G Proteins by Origin and Source

Origin Expressed in Conjugate
Human

More Proteins for Calcium Channel, Voltage-Dependent, T Type, alpha 1G Subunit (CACNA1G) Interaction Partners

Human Calcium Channel, Voltage-Dependent, T Type, alpha 1G Subunit (CACNA1G) interaction partners

  1. In 2 families with autosomal dominant SCA, a CACNA1G mutation p.Arg1715His was found at S4 of repeat IV, the voltage sensor of the CaV3.1 which shifted it toward a positive potential.

  2. A Recurrent Mutation in CACNA1G Alters Cav3.1 T-Type Calcium-Channel Conduction and Causes Autosomal-Dominant Cerebellar Ataxia (show USP14 Proteins).

  3. CaV3.1 channel is required for the generation of a given set of thalamocortical rhythms during unconsciousness. Further, that thalamocortical resonant neuronal activity supported by this channel is important for the control of vigilance states

  4. CaV3.1 downregulation is a major initiating factor for the increased production of the toxic Ab peptide, then the CaV3.1 T-type calcium channel represents a novel target for preventative therapeutics in Alzheimer's disease.

  5. Ethanol affects CaV3.2 (show CACNA1H Proteins) but not CaV3.1 nor CaV3.3 (show CACNA1I Proteins) channel isoforms.

  6. Cd(2 (show CD2 Proteins)) carried sizable inward currents through Ca(v)3.1 channels (210 +/- 20 pA at -60 mV with 2 mM Cd(2 (show CD2 Proteins))).

  7. Ca(V)3.1 channels represent a likely pathway for Fe(2) entry into cells.

  8. Augmentation of CaV3.1 currents by Ras-ERK (show EPHB2 Proteins) activation is associated with enhanced trafficking of channels to the plasma membrane.

  9. Cav3.1 channels may contribute to the repression of tumor proliferation and the promotion of apoptosis mediated via Cav3.1-specific calcium signals

  10. Data showed expression of L-type (Ca(v) 1.2 (show CACNA1C Proteins)), P/Q-type (Ca(v) 2.1 (show CACNA1A Proteins)), and T-type subtype (Ca(v) 3.1 and Ca(v) 3.2) voltage-gated calcium channels (Ca(v)s) in renal artery and dissected intrarenal blood vessels from nephrectomies.

Mouse (Murine) Calcium Channel, Voltage-Dependent, T Type, alpha 1G Subunit (CACNA1G) interaction partners

  1. GluA4 (show GRIA4 Proteins) subunits are required to produce an EPSC-triggerable postsynaptic action potentials after the presynaptic action potential, while Cav3.1 expression is needed to establish the driver function of L5B-POm synapses at hyperpolarized membrane potentials

  2. Mice deficient for CaV3.1 were resistant to the induction of experimental autoimmune encephalomyelitis and had reduced productions of the granulocyte-macrophage colony-stimulating factor (show CSF2 Proteins) by central nervous system-infiltrating Th1 (show HAND1 Proteins) and Th17 cells.

  3. Cross-frequency coupling between low-frequency and gamma rhythms was pronounced in wild-type but not in CaV3.1 knockouts, suggesting that the presence of CaV3.1 channels is a key element in the pathophysiology of trigeminal neuropathic pain.

  4. CaV3.1 and CaV3.2 (show CACNA1H Proteins) are substrates for EHD3 (show EHD3 Proteins)-dependent protein trafficking in heart

  5. This study reported on the cloning and characterization of a proximal promoter region and initiated the analysis of transcription factors that control CaV (show CA5A Proteins) 3.1 channel expression using the murine Cacna1g gene as a model.

  6. Ca(v)3.1-dependent synaptic depression at thalamocortical projections contributes to mechanisms of forward suppression in the auditory cortex

  7. Suggest that T-type Ca(2 (show CA2 Proteins)+) channels play an important role in infranodal escape automaticity.

  8. CaV (show CA5A Proteins) 3.1 channels are important for the myogenic tone at low arterial pressure, which is potentially relevant under resting conditions in vivo mice.

  9. Ca(v)3.1 is required for vascular smooth muscle proliferation during neointimal formation, and blocking of Ca(v)3.1 may be beneficial for preventing restenosis.

  10. Activation of Cav3.1 T-type channel subunit during wake-like states is a major determinant for single and multiple spike occurrence during tonic firing and for the robustness of the thalamocortical transfer of sensory inputs.

Cow (Bovine) Calcium Channel, Voltage-Dependent, T Type, alpha 1G Subunit (CACNA1G) interaction partners

  1. CaV3.1 structural analysis and comparison to CaV1.2 (show CACNA1C Proteins) channel

CACNA1G Protein Profile

Protein Summary

Voltage-sensitive calcium channels mediate the entry of calcium ions into excitable cells, and are also involved in a variety of calcium-dependent processes, including muscle contraction, hormone or neurotransmitter release, gene expression, cell motility, cell division, and cell death. This gene encodes a T-type, low-voltage activated calcium channel. The T-type channels generate currents that are both transient, owing to fast inactivation, and tiny, owing to small conductance. T-type channels are thought to be involved in pacemaker activity, low-threshold calcium spikes, neuronal oscillations and resonance, and rebound burst firing. Many alternatively spliced transcript variants encoding different isoforms have been described for this gene.

Gene names and symbols associated with CACNA1G

  • calcium channel, voltage-dependent, T type, alpha 1G subunit (CACNA1G)
  • calcium channel, voltage-dependent, T type, alpha 1G subunit (cacna1g)
  • calcium channel, voltage-dependent, T type, alpha 1G subunit (Cacna1g)
  • a1G protein
  • alpha-1G protein
  • Ca(v)3.1 protein
  • Ca(V)T.1 protein
  • CACNA1G protein
  • Cav3.1 protein
  • Cav3.1d protein
  • mKIAA1123 protein
  • NBR13 protein
  • [a]1G protein

Protein level used designations for CACNA1G

voltage-dependent calcium channel alpha 1G subunit , calcium channel, voltage-dependent, T type, alpha 1G subunit , calcium channel voltage-dependent T type Cav3.1, alpha 1g subunit , cav3.1c , voltage-dependent T-type calcium channel subunit alpha-1G , voltage-gated calcium channel subunit alpha Cav3.1 , calcium channel, voltage-dependent, alpha 1G subunit

GENE ID SPECIES
455128 Pan troglodytes
563904 Danio rerio
769385 Gallus gallus
100070000 Equus caballus
100346309 Oryctolagus cuniculus
100462914 Xenopus laevis
8913 Homo sapiens
12291 Mus musculus
29717 Rattus norvegicus
491083 Canis lupus familiaris
282411 Bos taurus
100513431 Sus scrofa
100728128 Cavia porcellus
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