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The nicotinic acetylcholine receptors (nAChRs) are members of a superfamily of ligand-gated ion channels that mediate fast signal transmission at synapses. Additionally we are shipping CHRNB3 Antibodies (38) and CHRNB3 Kits (4) and many more products for this protein.
Showing 9 out of 13 products:
More low frequency variants in the CHRNA6/CHRNB3 gene region were observed in cases compared to controls.
the CHRNB3-A6 locus contains multiple variants affecting risk for vulnerability to cocaine and nicotine dependence as well as bipolar disorder, suggesting that they have pleiotropic effects.
Polymorphism in CHRNB3 gene is associated with esophageal adenocarcinoma.
The common variant rs13273442 in the CHRNB3-CHNRA6 (show CHRNA6 Proteins) region is associated significantly with nicotine dependence in European Americans and African Americans.
CHRNB3 c.-57A>G alteration affects the promoter activity and is associated with Parkinson's disease (PD), and smoking in PD patients.
Level of cigarettes per day during adolescence and young adulthood is associated with CHRNB3A6, CHRNA5A3B4, and CHRNA2 (show CHRNA2 Proteins)
Rare missense variants in CHRNB3 and CHRNA3 are associated with risk of alcohol and cocaine dependence.
this study provides convincing evidence for a role of CHRNB3 in Nicotine Dependence.
these results demonstrate that the combined effect of rs6474412-C/T polymorphism in smoking-related CHRNB3-CHRNA6 region gene and smoking behavior may not confer risk to psoriasis vulgaris (PV), but may have impact on PV severity in Chinese Han population.
Results demonstrate the association between SNPs in CHRNB3 nicotinic acetylcholine receptor genes and the risk of smoking in ADHD
Chimeric nicotinic acetylcholine receptors reveal the roles for N-terminal extracellular domains of (nAChR (show CHRNA4 Proteins)) beta3 subunits in the receptor activity and function.
beta3 subunit coexpression promotes function of alpha6*-nAChR (show CHRNA6 Proteins)
Data show that in Prkce (show PRKCE Proteins)(-/-) mice, conditioned place preference and self-administer behaviors are associated with reduced levels of alpha(6) and beta(3) nicotinic receptor subunit mRNA in the ventral midbrain and striatum.
The nicotinic acetylcholine receptors (nAChRs) are members of a superfamily of ligand-gated ion channels that mediate fast signal transmission at synapses. The nAChRs are (hetero)pentamers composed of homologous subunits. The subunits that make up the muscle and neuronal forms of nAChRs are encoded by separate genes and have different primary structure. There are several subtypes of neuronal nAChRs that vary based on which homologous subunits are arranged around the central channel. They are classified as alpha-subunits if, like muscle alpha-1 (MIM 100690), they have a pair of adjacent cysteines as part of the presumed acetylcholine binding site. Subunits lacking these cysteine residues are classified as beta-subunits (Groot Kormelink and Luyten, 1997
acetylcholine receptor, neuronal nicotinic, beta-3 subunit
, acetylcholine receptor, nicotinic, beta 3 (neuronal)
, cholinergic receptor, nicotinic, beta polypeptide 3
, neuronal acetylcholine receptor subunit beta-3
, cholinergic receptor, nicotinic, beta 3
, neuronal acetylcholine receptor subunit beta-3-like
, cholinergic receptor neuronal nicotinic beta protein 3
, nicotinic acetylcholine receptor beta 3 subunit