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The protein encoded by CSF3R is the receptor for colony stimulating factor 3, a cytokine that controls the production, differentiation, and function of granulocytes. Additionally we are shipping CSF3R Kits (30) and CSF3R Proteins (20) and many more products for this protein.
Showing 10 out of 182 products:
Human Monoclonal CSF3R Primary Antibody for FACS, WB - ABIN4899922
Shao, Xu, Jing, Tweardy: Unique structural determinants for Stat3 recruitment and activation by the granulocyte colony-stimulating factor receptor at phosphotyrosine ligands 704 and 744. in Journal of immunology (Baltimore, Md. : 1950) 2006
Show all 2 references for ABIN4899922
Human Monoclonal CSF3R Primary Antibody for EIA, WB - ABIN114568
Baars, De Ruijter, Smedts, Van Niekerk, Poels, Seldenrijk, Ramaekers: The applicability of a keratin 7 monoclonal antibody in routinely Papanicolaou-stained cytologic specimens for the differential diagnosis of carcinomas. in American journal of clinical pathology 1994
Show all 2 references for ABIN114568
Human Polyclonal CSF3R Primary Antibody for EIA, FACS - ABIN951155
Germeshausen, Kratz, Ballmaier, Welte: RAS and CSF3R mutations in severe congenital neutropenia. in Blood 2009
Show all 2 references for ABIN951155
Human Polyclonal CSF3R Primary Antibody for ELISA, WB - ABIN4313030
Meenhuis, Verwijmeren, Roovers, Touw: The deubiquitinating enzyme DUB2A enhances CSF3 signalling by attenuating lysosomal routing of the CSF3 receptor. in The Biochemical journal 2011
Human Polyclonal CSF3R Primary Antibody for IF (p), IHC (p) - ABIN742275
Tajima, Waki, Tsuchiya, Hoshi et al.: Granulocyte colony-stimulating factor-producing undifferentiated carcinoma of the colon mimicking a pulmonary giant cell carcinoma: a case showing overexpression of CD44 along with highly ... in International journal of clinical and experimental pathology 2014
Human Monoclonal CSF3R Primary Antibody for ELISA (Capture), FACS - ABIN4899923
deBruin, Lincoln, Hartley, Shehabeldin, Van, Szilvassy: Most purported antibodies to the human granulocyte colony-stimulating factor receptor are not specific. in Experimental hematology 2010
Human Monoclonal CSF3R Primary Antibody for FACS - ABIN4896903
Dror, Durie, Ginzberg, Herman, Banerjee, Champagne, Shannon, Malkin, Freedman: Clonal evolution in marrows of patients with Shwachman-Diamond syndrome: a prospective 5-year follow-up study. in Experimental hematology 2002
Human Monoclonal CSF3R Primary Antibody for FACS - ABIN319850
Nicholson, Oates, Harpur, Ziemiecki, Wilks, Layton: Tyrosine kinase JAK1 is associated with the granulocyte-colony-stimulating factor receptor and both become tyrosine-phosphorylated after receptor activation. in Proceedings of the National Academy of Sciences of the United States of America 1994
Human Monoclonal CSF3R Primary Antibody for FACS - ABIN4896902
Radomska, Alberich-Jordà, Will, Gonzalez, Delwel, Tenen: Targeting CDK1 promotes FLT3-activated acute myeloid leukemia differentiation through C/EBPα. in The Journal of clinical investigation 2012
This study proposes that acquisition of CSF3R mutations may represent a mechanism by which myeloid precursor cells carrying the ELANE (show ELANE Antibodies) mutations evade the proapoptotic activity of the Neutrophil Elastase (show ELANE Antibodies) mutants in SCN (show SRI Antibodies) patients.
CSF3R expression is significantly upregulated in human masticatory mucosa during wound healing
Results indicate that granulocyte-colony stimulating factor receptor, tissue factor, and vascular endothelial growth factor receptor bound vascular endothelial growth factor expression as well as their co-expression might influence breast cancer biology.
The Colony-Stimulating Factor (show CSF2 Antibodies) 3 Receptor T640N Mutation Is Oncogenic, Sensitive to JAK (show JAK3 Antibodies) Inhibition, and Mimics T618I
CSF3R mutations, mechanisms of mutations, and their contributions to the myeloid malignancies (Review)
In conclusion, rhCSF3 can promote melanocyte proliferation through CSF3R without affecting tyrosinase (show TYR Antibodies) activity
CSF3R mutations are associated with congenital neutropenia.
The leukemogenic potential of G-CSFRIV is associated with the Stat5 (show STAT5A Antibodies)-dependent dysregulation of miR (show MLXIP Antibodies)-155 and the target genes of this miRNA.
No CSF3R mutations were found in cases of MDS (show PAFAH1B1 Antibodies), JMML or ET. The only mutation found in the CALR (show CALR Antibodies) gene was a frameshift (p.L367 fs) in one ET patient.
The SETBP1 (show SETBP1 Antibodies) and ASXL1 (show ASXL1 Antibodies) mutations have pathogenetic roles in CSF3R-mutated chronic neutrophilic leukemia.
this study shows that dorsal root ganglion neurons cultured in G-CSF (show CSF3 Antibodies) failed to respond to G-CSF (show CSF3 Antibodies) in vitro, and Csf3r gene expression could not be detected in dorsal root ganglion neurons by single-cell RT-PCR
Thr (show TRH Antibodies)-615 and Thr (show TRH Antibodies)-618 sites of membrane-proximal mutations are part of an O-linked glycosylation cluster. Mutation at these sites prevents O-glycosylation of CSF3R and increases receptor dimerization.
Fbw7 (show FBXW7 Antibodies) together with GSK3beta (show GSK3b Antibodies) negatively regulates G-CSFR expression and its downstream signaling.
G-CSFR signaling interacts with retinoic acid receptors in the regulation of myeloid differentiation
Expression of truncated G-CSFR significantly shortens the latency of AML (show RUNX1 Antibodies) in a G-CSF (show CSF3 Antibodies)-dependent fashion and it is associated with a distinct AML (show RUNX1 Antibodies) presentation characterized by higher blast counts and more severe myelosuppression.
G-CSFR signals in bone marrow monocytic cells inhibit the production of trophic factors required for osteoblast lineage cell maintenance, ultimately leading to hematopoietic stem and progenitor cell mobilization.
Signaling mechanisms coupled to tyrosines in the granulocyte colony-stimulating factor receptor orchestrate G-CSF (show CSF3 Antibodies)-induced expansion of myeloid progenitor cells.
murine granulocyte colony-stimulating factor receptor binds to a low molecular weight ligand
Mice with truncated G-CSF (show CSF3 Antibodies) receptors have neutropenia, susceptibility to infection, and bone marrow maturation arrest similar to severe congenital neutropenic humans, suggesting a role of receptor truncation mutations in SCN (show SRI Antibodies) pathology.
COOH-terminal truncation mutants of G-CSF-R, found in severe congenital neutropenia, lack internalization motifs and are completely defective in both spontaneous and ligand-induced internalization.
GCSF (show CSF3 Antibodies)/GCSFR is a conserved signaling system for facilitating the production of multiple myeloid cell lineages, as well as for early myeloid cell migration.
The protein encoded by this gene is the receptor for colony stimulating factor 3, a cytokine that controls the production, differentiation, and function of granulocytes. The encoded protein, which is a member of the family of cytokine receptors, may also function in some cell surface adhesion or recognition processes. Alternatively spliced transcript variants have been described. Mutations in this gene are a cause of Kostmann syndrome, also known as severe congenital neutropenia.
granulocyte colony-stimulating factor receptor
, colony stimulating factor 3 receptor (granulocyte)
, granulocyte colony-stimulating factor receptor-like
, CD114 antigen
, G-CSF receptor
, granulocyte stimulating factor receptor